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Stem Cell Research on Cardiology
| Stem Cell Research on Cardiology |
| Autore | David Robert |
| Pubbl/distr/stampa | Basel, Switzerland, : MDPI - Multidisciplinary Digital Publishing Institute, 2020 |
| Descrizione fisica | 1 electronic resource (356 p.) |
| Soggetto topico |
Research & information: general
Biology, life sciences |
| Soggetto non controllato |
Fabry disease
human embryonic stem cells CRISPR/Cas9 genomic editing Mass spectrometry proteomic analysis hypertrophic cardiomyopathy disease model physical exercise cardiac cellular regeneration microRNA (miR) Akt signaling cardiomyocyte proliferation cardiac hypertrophy cardioprotection myocarditis inflammation leukocytes cardiomyocytes multi-electrode-array micro-electrode-array MEA drug/toxicity screening field potential arrhythmia electrocardiography cardiac regeneration stem cells iPSC PSC ESC cardiovascular disease regeneration cardiac progenitor cells induced pluripotent stem cells transdifferentiation direct reprogramming genetic engineering cardiac tissue engineering biomaterials 18F-FDG PET cardiac induced cells cardiac function non-invasive imaging human pluripotent stem cell ventricular maturation bone marrow stem cells angiogenesis myocardial infarction human induced pluripotent stem cell-derived cardiomyocytes (hiPSC-CMs) iPS cells big conductance calcium activated potassium channel (BK) Maxi-K slo1 KCa1.1 iberiotoxin long QT syndrome mesenchymal stromal cells (MSC) mRNA miRNA cardiac reprogramming cardiac differentiation cardiovascular diseases adult stem cells myocardial infraction 3D printing 3D model bioprinting cardiovascular medicine heart myocardium heart valves vascular graft endothelialization tissue engineering decorin fibronectin electrospinning endothelial progenitor cells bioreactor biostable polyurethane MicroRNA Mir-133 coronary heart disease biomarker meta-analysis |
| Formato | Materiale a stampa  |
| Livello bibliografico | Monografia |
| Lingua di pubblicazione | eng |
| Record Nr. | UNINA-9910557701803321 |
David Robert
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| Basel, Switzerland, : MDPI - Multidisciplinary Digital Publishing Institute, 2020 | ||
| Materiale a stampa | ||
| Lo trovi qui: Univ. Federico II | ||
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Transcriptional Regulation of Cardiac Development and Disease
| Transcriptional Regulation of Cardiac Development and Disease |
| Autore | Wagner Nicole |
| Pubbl/distr/stampa | Basel, : MDPI - Multidisciplinary Digital Publishing Institute, 2022 |
| Descrizione fisica | 1 electronic resource (222 p.) |
| Soggetto topico |
Research & information: general
Biology, life sciences Biochemistry |
| Soggetto non controllato |
pluripotent stem cells
cardiomyocytes cardiac tissue engineering human heart tissue maturation Wilms' tumor suppressor 1 cardiomyocyte differentiation mouse embryonic stem cells myocardial infarction YAP TEAD1 Toll-like receptor heart TLR4 cardiomyocyte innate immune responses Wilms' tumor suppressor 1 (Wt1) cardiac development coronary vessel formation transcriptional regulation cardiac malformation epicardium epicardial derived cells (EPDCs) epithelial mesenchymal transition (EMT) cardiac cell fate regeneration arrhythmogenic cardiomyopathy (ACM) lamin A/C atomic force microscopy (AFM) cell-cell adhesion neonatal rat ventricular fibroblasts (NRVF) tunneling nanotubes (TNT) gene therapy cardiovascular diseases cardiac regeneration cardiac reprogramming therapeutic angiogenesis growth factors reactive oxygen species apoptosis Bmp signaling Hand1 Smad heart development SH3BGR cardiac hypertrophy SRF signaling Hippo signaling cardiomyogenic differentiation cardiac mesenchymal stromal cell hydrogels histone deacetylase inhibitors translational studies gene expression and regulation cardiomyopathy heart failure reverse remodelling left ventricular assist device |
| Formato | Materiale a stampa |
| Livello bibliografico | Monografia |
| Lingua di pubblicazione | eng |
| Record Nr. | UNINA-9910674017103321 |
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Wagner Nicole
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| Basel, : MDPI - Multidisciplinary Digital Publishing Institute, 2022 | ||
| Materiale a stampa | ||
| Lo trovi qui: Univ. Federico II | ||
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