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Binge Eating : A Transdiagnostic Psychopathology / / edited by Guido K.W. Frank, Laura A. Berner
Binge Eating : A Transdiagnostic Psychopathology / / edited by Guido K.W. Frank, Laura A. Berner
Edizione [1st ed. 2020.]
Pubbl/distr/stampa Cham : , : Springer International Publishing : , : Imprint : Springer, , 2020
Descrizione fisica 1 online resource (313 pages)
Disciplina 616.8526
Soggetto topico Neurosciences
Neuropsychology
Psychology, Pathological
Behavioral therapy
Pharmacology
Psychology
Psychopathology
Behavioral Therapy
Pharmacology/Toxicology
Psychology, general
Trastorns de la gana
Neurociències
Soggetto genere / forma Llibres electrònics
ISBN 3-030-43562-8
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Chapter 1. Epidemiology of Binge Eating -- Chapter 2. Binge Eating Assessment -- Chapter 3. Developmental and Cultural Aspects of Binge Eating -- Chapter 4. Psychosocial Correlates of Binge Eating -- Chapter 5. Weight Dysregulation, Positive Energy Balance and Binge Eating in Eating Disorders -- Chapter 6. Theoretical Development and Maintenance Models of Binge Eating -- Chapter 7. Preclinical Models of Stress and Environmental Influences on Binge Eating -- Chapter 8. Emerging Translational Treatments to Target the Neural Networks of Binge Eating -- Chapter 9. Neuroimaging to Study Brain Reward Processing and Reward-Based Learning in Binge Eating Pathology -- Chapter 10. The Neurobiological Basis of Executive Function Alterations in Binge Eating Populations -- Chapter 11. Influence of Genetics and Sex Hormones on Binge Eating -- Chapter 12. Neuroendocrine Correlates of Binge Eating -- Chapter 13. Body Image Disturbance and Binge Eating -- Chapter 14. Food Addiction, Binge Eating and the Role of Dietary Restraint: Converging Evidence from Animal and Human Studies -- Chapter 15. Prevention of Binge Eating -- Chapter 16. Medication for Binge Eating -- Chapter 17. Oxytocin: Potential New Treatment for Binge Eating -- Chapter 18. Psychotherapy for Binge Eating -- Chapter 19. Self Help Interventions for the Treatment of Binge Eating -- Chapter 20. New Avenues for the Treatment of Binge Eating based on Implicit Processes -- Chapter 21. Binge-Eating Disorder: Unanswered Questions -- Chapter 22. Overcoming Barriers to the Treatment of Binge Eating.
Record Nr. UNINA-9910409686103321
Cham : , : Springer International Publishing : , : Imprint : Springer, , 2020
Materiale a stampa
Lo trovi qui: Univ. Federico II
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Complex cases and comorbidity in eating disorders : assessment and management / / Riccardo Dalle Grave, Massimiliano Sartirana, Simona Calugi
Complex cases and comorbidity in eating disorders : assessment and management / / Riccardo Dalle Grave, Massimiliano Sartirana, Simona Calugi
Autore Grave Riccardo Dalle
Pubbl/distr/stampa Cham, Switzerland : , : Springer, , [2021]
Descrizione fisica 1 online resource (xv, 161 pages) : illustrations
Disciplina 616.8526
Soggetto topico Eating disorders
Trastorns de la gana
Comorbiditat
Complicacions (Medicina)
Soggetto genere / forma Llibres electrònics
ISBN 3-030-69341-4
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Intro -- Preface -- Acknowledgements -- Contents -- Abbreviations -- Part I: Eating-Disorder Psychopathology, Comorbidity, and Cognitive Behaviour Therapy -- Chapter 1: Eating Disorders: An Overview -- 1.1 Eating Problems and Eating Disorders -- 1.2 Anorexia Nervosa -- 1.3 Bulimia Nervosa -- 1.4 Binge-Eating Disorder -- 1.5 Other Eating Disorders -- 1.6 The Transdiagnostic Perspective -- References -- Chapter 2: Eating Disorder Psychopathology and Its Consequences -- 2.1 Eating Disorder Psychopathology -- 2.1.1 Eating Problem Check List: A Questionnaire for Assessing Eating Disorder Psychopathology -- 2.2 The Effects of Calorie Restriction and Low Weight -- 2.2.1 Behavioural Effects -- 2.2.2 Psychological Effects -- 2.2.3 Social Effects -- 2.2.4 Physical Effects -- 2.2.5 Comments from Minnesota Starvation Experiment Participants -- 2.2.6 Implications for Psychopathology Assessment -- 2.2.7 Starvation Symptom Inventory: A Questionnaire for Assessing Starvation Symptoms -- 2.3 The Psychosocial Consequences of Eating Disorder Psychopathology -- 2.4 The Physical Consequences of Eating Disorder Psychopathology -- References -- Chapter 3: Enhanced Cognitive Behaviour Therapy for Eating Disorders -- 3.1 Transdiagnostic Cognitive Behavioural Theory -- 3.2 Core Maintenance Processes -- 3.3 Additional Maintenance Processes -- 3.4 CBT-E: An Overview -- 3.4.1 Treatment Goals -- 3.4.2 General Strategies -- 3.4.3 Forms of CBT-E -- 3.4.4 Adaptations for Clinical Groups and Settings -- 3.4.4.1 Outpatient CBT-E -- 3.4.4.2 Intensive Outpatient CBT-E -- 3.4.4.3 Inpatient CBT-E -- 3.4.4.4 Post-Inpatient Outpatient CBT-E -- 3.4.4.5 Broad CBT-E -- 3.4.5 CBT-E Clinical Services -- 3.4.6 The Current Status of CBT-E -- 3.4.7 Training in CBT-E -- References -- Part II: Management of Complex Eating Disorder Cases.
Chapter 4: General Strategies for the Management of Comorbidity in Eating Disorders -- 4.1 Epidemiological Data on Comorbidity in Eating Disorders -- 4.2 Methodological Problems with Studies Assessing Comorbidity in Eating Disorders -- 4.3 Comorbidity or Complex Cases? -- 4.4 CBT-E's Pragmatic Approach to Complex Cases -- 4.5 Multidisciplinary Management of Complex Cases -- 4.5.1 The Non-Eclectic CBT-E Multidisciplinary Team -- References -- Chapter 5: Coexisting Psychological Problems -- 5.1 Clinical Perfectionism -- 5.1.1 Characteristics of Clinical Perfectionism -- 5.1.2 Assessing for the Presence of Clinical Perfectionism -- 5.1.3 Strategies for Addressing Clinical Perfectionism -- 5.2 Core Low Self-Esteem -- 5.2.1 Characteristics of Core Low Self-Esteem -- 5.2.2 Assessing for Core Low Self-Esteem -- 5.2.3 Strategies for Addressing Core Low Self-Esteem -- 5.3 Marked Interpersonal Difficulties -- 5.3.1 Characteristics of Marked Interpersonal Difficulties -- 5.3.2 Assessing for Marked Interpersonal Difficulties -- 5.3.3 Strategies for Addressing Marked Interpersonal Difficulties -- 5.4 Mood Intolerance -- 5.4.1 Characteristics of Mood Intolerance -- 5.4.2 Assessing for the Presence of Mood Intolerance -- 5.4.3 Strategies for Addressing Mood Intolerance -- References -- Chapter 6: Coexisting Mental Disorders -- 6.1 Clinical Depression -- 6.1.1 Other Mood Disorders -- 6.2 Anxiety Disorders -- 6.2.1 Eating-Disorder Psychopathology Is Not a Specific Form of Phobia -- 6.3 Obsessive-Compulsive Disorder -- 6.4 Post-Traumatic Stress Disorder -- 6.5 Substance Use Disorder -- 6.6 Personality Disorders -- 6.7 Other Psychiatric Disorders -- References -- Chapter 7: Physical Complications -- 7.1 Osteopenia and Osteoporosis -- 7.2 Amenorrhoea and Infertility -- 7.3 Fluid and Electrolyte Abnormalities -- 7.4 Cardiovascular Abnormalities.
7.5 Gastrointestinal Abnormalities -- 7.6 Clinical Issues Requiring Prompt Medical Intervention -- References -- Chapter 8: Coexisting General Medical Diseases -- 8.1 Obesity -- 8.1.1 Prevalence -- 8.1.2 Risk Factors -- 8.1.3 Interactions between Eating Disorders and Obesity -- 8.1.4 Clinical Consequences -- 8.1.5 Is Weight Loss Indicated? -- 8.2 Type 1 Diabetes -- 8.2.1 Diagnostic Issues -- 8.2.2 Prevalence -- 8.2.3 Risk Factors -- 8.2.4 Eating Disorder Psychopathology in Type 1 Diabetes -- 8.2.5 Interactions Between Eating Disorders and Type 1 Diabetes -- 8.2.6 Clinical Consequences -- 8.2.7 Treatment -- 8.3 Coeliac Disease -- 8.3.1 Two-Way Association between Anorexia Nervosa and Coeliac Disease -- 8.3.2 Mechanisms that Explain the Bidirectional Association -- 8.3.3 Attention to the Initial Diagnosis -- 8.3.4 Interaction Between Eating Disorders and Coeliac Disease -- 8.3.5 Treatment -- 8.4 Allergies -- 8.4.1 Alternative Food Intolerance Tests -- 8.4.2 False Eating Intolerances and Eating Disorders -- 8.4.3 Treatment -- 8.5 Other Coexisting Medical Problems -- References -- Chapter 9: Severe and Enduring Anorexia Nervosa -- 9.1 Labels and Definitions Used for Patients with Enduring Anorexia Nervosa -- 9.2 Which Treatment Is Indicated for Patients with Severe and Enduring Anorexia Nervosa? -- 9.2.1 Management Strategies Designed to Enhance the Quality of Life and Reduce Harm -- 9.2.2 Treatments Designed to Address the Eating Disorder Psychopathology and Weight Regain -- 9.3 A Pragmatic Clinical Approach to Managing Patients with Severe and Enduring Anorexia Nervosa -- References -- Appendix A: The Eating Problem Check List -- What Is It, and How Should It Be Used? -- Status of the EPCL -- Scoring the EPCL -- Copyright -- The Eating Problem Checklist (EPCL) 3.1 -- Appendix B: The Starvation Symptom Inventory -- What Is It, and How Should It Be Used?.
Status of the SSI -- Scoring the SSI -- Copyright -- The Starvation Symptom Inventory (SSI).
Record Nr. UNINA-9910484143603321
Grave Riccardo Dalle  
Cham, Switzerland : , : Springer, , [2021]
Materiale a stampa
Lo trovi qui: Univ. Federico II
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Food addiction, obesity, and disorders of overeating : an evidence-based assessment and clinical guide / / Claire E. Wilcox
Food addiction, obesity, and disorders of overeating : an evidence-based assessment and clinical guide / / Claire E. Wilcox
Autore Wilcox Claire E.
Pubbl/distr/stampa Cham, Switzerland : , : Springer, , [2021]
Descrizione fisica 1 online resource (224 pages)
Disciplina 616.8526
Soggetto topico Compulsive eating
Trastorns de la conducta alimentària
Trastorns de la gana
Obesitat
Soggetto genere / forma Llibres electrònics
ISBN 3-030-83078-0
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Intro -- Introduction: Obesity, Eating Disorders and Food Addiction: Towards a Synthesis -- References -- Contents -- Contributors -- Part I: Standard Approaches to Clinical Assessment and Treatment of Obesity and Binge Eating Disorder (BED) -- 1: Determinants of Body Weight: Metabolism and the Homeostatic System -- 1.1 Energy Balance -- 1.2 Metabolism -- 1.3 Why Weight Gain Is So Easy and Weight Loss So Hard -- 1.4 Homeostatic Feeding Mechanisms -- 1.4.1 The Hypothalamus -- 1.4.2 Key Neuropeptides -- 1.4.3 Other Factors -- 1.5 Neuropeptides, Obesity, and Disordered Eating -- 1.6 Conclusion -- References -- 2: Obesity -- 2.1 Definition of Obesity -- 2.2 Epidemiology -- 2.3 Assessment -- 2.4 Causes, Contributors, and Risk Factors -- 2.5 Genetics -- 2.6 Management -- 2.6.1 Overview of Treatment -- 2.6.2 Comprehensive Models and Behavioral Weight Loss Therapy -- 2.6.3 Dietary Component -- 2.6.4 Physical Activity Component -- 2.6.5 Behavioral Modification Component -- 2.6.6 Pharmacotherapy -- 2.6.7 Dietary Supplements and Procedures to Avoid -- 2.6.8 Bariatric Surgery -- 2.7 Conclusion -- References -- 3: Binge-Related Eating Disorders (Binge Eating Disorder and Bulimia Nervosa) -- 3.1 Epidemiology -- 3.2 Diagnosis and Assessment of BED -- 3.3 Epidemiology of BN -- 3.4 Diagnosis and Assessment of BN -- 3.5 Etiology and Mechanisms of BED and BN -- 3.6 Treatment of BED: General Considerations -- 3.7 Psychotherapies for BED -- 3.8 Pharmacotherapy for BED -- 3.9 Nutritional Recommendations for BED -- 3.10 Best Practices and Guidelines for BED Treatment -- 3.11 Obesity and BED Treatment -- 3.12 Treatment of BN -- 3.13 Treatments for both BED and BN -- 3.14 Conclusion -- References -- Part II: Can the Food Addiction Concept Improve Treatment? -- 4: Problems with Current Approaches to Treating Disorders of Overeating.
4.1 Weight Loss with Available Treatments Is Modest -- 4.2 Maintenance of Weight Loss Is Difficult -- 4.3 The Biology Behind the Difficulty of Weight Loss and Maintenance -- 4.4 Eating Disorder (ED) Treatment Success Rates -- 4.5 ED and Obesity Treatments Give Conflicting Messages -- 4.6 Side Effects of Diets -- 4.7 Limitations and Side Effects of Medications -- 4.8 Side Effects of Bariatric Surgery -- 4.9 Difficulty of Exercising When Obese -- 4.10 Conclusion -- References -- 5: The Food Addiction Concept: History, Controversy, Potential Pitfalls, and Promises -- 5.1 History -- 5.2 Is the FA Concept Valid? -- 5.2.1 Can Obesity Be Explained by FA? -- 5.2.2 Is FA Distinct from BED and Bulimia? -- 5.2.3 Do DSM Criteria for SUD Present in Relation to Food in Humans and Do Symptoms Cluster Together? -- 5.2.4 Is It Valid to Claim Certain Foods Are "Addictive," and Might It Be More Accurate to Consider FA a Behavioral Disorder? -- 5.3 Is the FA Model Useful, and Do Benefits Outweigh Harms? -- 5.3.1 Abstinence-Based Food Plans -- 5.3.2 Self-Efficacy -- 5.3.3 Public Health -- 5.3.4 Stigma -- 5.4 Potential Promises for the Future -- 5.5 Increasing Community Acceptance -- 5.6 Conclusion -- References -- Part III: Clinical Evidence for Food Addiction -- 6: Clinical Evidence for the Validity of Food Addiction -- 6.1 Shared DSM Criteria -- 6.2 Yale Food Addiction Scale Development -- 6.3 SUD and Disordered Eating Co-occur -- 6.4 Sweet Preference, Addiction Transfer, and Cross-Sensitization -- 6.5 Overlapping Neuropsychological, Emotional, and Personality Traits, Psychiatric Diagnoses, and Predisposing Conditions (Trauma and Stress) -- 6.6 Conclusion -- References -- Part IV: Basic Biology of Food Addiction, and Its Overlap with Substance Use Disorders -- 7: Neurobiology and Cognitive Neuroscience of Substance Use Disorders.
7.1 Overview of Substance Use Disorders -- 7.2 Core Brain Regions -- 7.3 Reward -- 7.4 Conditioning: Positive Reinforcement -- 7.5 Motivation: Positive Reinforcement -- 7.6 Tolerance: Downregulation of Dopamine and Opioid System -- 7.7 Withdrawal and Hyperkatifeia -- 7.8 Conditioning and Motivation: Negative Reinforcement -- 7.9 Impulsivity and Executive Function Deficits -- 7.10 Benefits of Understanding the Neurobiology -- 7.11 Conclusion -- References -- 8: Neurobiology and Cognitive Neuroscience of Hedonic Eating -- 8.1 Reward and Hedonic Liking -- 8.2 Conditioning: Positive Reinforcement -- 8.3 Motivation: Positive Reinforcement -- 8.4 Food Reward, Conditioning, and Reward Motivation: Additional Factors -- 8.5 Tolerance and Downregulation of DA and Opioid Systems -- 8.6 Withdrawal -- 8.7 Conditioning and Motivation: Negative Reinforcement -- 8.8 Impulse Control and Executive Function Deficits -- 8.9 Conclusion -- References -- 9: Additional Biological Mechanisms of Hedonic Eating -- 9.1 Interactions Between the Homeostatic System and Hedonic System -- 9.1.1 Anatomy -- 9.1.2 Appetite-Regulating Neuropeptides Modulate Hedonic Eating -- 9.1.3 Appetite-Regulating Neuropeptides Moderate Drug and Alcohol Use -- 9.2 Stress, Hedonic Eating, and the Reward System -- 9.2.1 The Anatomy of the Stress Response -- 9.2.2 Acute and Chronic Stress Promote Hedonic Eating -- 9.3 Genetics -- 9.4 In Utero Exposure -- 9.5 Neuroinflammation -- 9.6 Oxidative Stress -- 9.7 Gut Microbiome and Gut-Brain Axis -- 9.8 Adrenergic System -- 9.9 Sleep and Circadian Rhythm -- 9.10 Serotonin System -- 9.11 Endocannabinoid System -- 9.12 Functional Connectivity -- 9.13 Conclusions -- References -- 10: Treatment-Related Evidence that Food Addiction Is a Valid Construct -- 10.1 Pharmacotherapy-Related Evidence -- 10.1.1 Stimulants.
10.1.2 Opioid Antagonists -- 10.1.3 Topiramate and Zonisamide -- 10.1.4 GLP-1 Agonists -- 10.1.5 Other Medications to Note -- 10.2 Bariatric Surgery -- 10.3 Conclusion -- References -- 11: Highly Palatable Foods Are Addictive -- 11.1 Problematic and "Addictive" Foods -- 11.2 Association Between HP Food Intake and Addiction in Animal Models -- 11.3 Association Between HP Food Intake and Weight Gain/Disordered Eating in Humans -- 11.4 Why Are HP Foods More Associated with Addictive Eating Patterns? -- 11.4.1 Innate Preferences -- 11.4.2 Conditioning from Rapid Post-oral Glucose Rise -- 11.4.3 Stimulation of Reward System by HP Foods -- 11.4.4 Effects on Inflammatory Processes, Oxidative Stress and Gut Microbiome -- 11.4.5 Cessation Leads to Withdrawal -- 11.4.6 Adverse Effects on Mood and Anxiety -- 11.4.7 Reduction in Executive Function -- 11.4.8 Reduction in Satiety Due to Changes in Homeostatic Feeding -- 11.4.9 Individual Variability -- 11.4.10 Feeding Patterns Influence Food Addiction -- 11.5 State Effects of Hunger/Food Restriction on Reward Circuitry and Brain Function -- 11.6 Artificial Sweeteners and Sugar Substitutes -- 11.7 What Should Be Considered Addictive Food? -- 11.8 Conclusion -- References -- Part V: Assessment and Treatment of Food Addiction -- 12: Evaluation of Food Addiction: Importance, Epidemiology, Diagnosis, and Assessment -- 12.1 Importance of Assessing for Food Addiction -- 12.2 Epidemiology of FA -- 12.3 YFAS: Scoring and Interpretation -- 12.4 Other Important Assessment Considerations and Common Comorbidities -- 12.4.1 SUD -- 12.4.2 ED History -- 12.4.3 Obesity History and Related Health Concerns -- 12.4.4 Psychiatric Comorbidity -- 12.5 Conclusion -- References -- 13: How to Treat Food Addiction from a Nutritional Perspective: Consideration of Diet and Abstinence.
13.1 Nutritional Approaches and Consideration of Abstinence -- 13.2 Related Tips -- 13.2.1 Increase Satiety and Brain Health-Promoting Foods -- 13.2.2 Do Not Over-restrict Calorie Intake -- 13.2.3 Realize that Craving Will Diminish with Time in Recovery -- 13.2.4 Abstinence Is Not Absolute: Avoid All-or-Nothing Thinking -- 13.2.5 Is It Better to Start More Extreme or Use a Graded Approach During Initiation? -- 13.2.6 Track Progress -- 13.3 How to Incorporate FA Treatment into ED Treatment Programming -- 13.4 What to Do with "Normal Weight" FA Patients? -- 13.5 Conclusion -- References -- 14: Clinical Applications of the Food Addiction Concept -- 14.1 Treatment Overview -- 14.2 Supplemental Programmatic Elements Which Might Be Useful for Treatment of FA -- 14.2.1 Psychoeducation: FA Is a Brain-Based Disorder -- 14.2.2 Psychosocial Interventions -- 14.2.2.1 CBT -- 14.2.2.2 Behavioral Weight Management -- 14.2.2.3 Motivational Interviewing (MI) -- 14.2.2.4 Psychotherapy to Reduce Negative Affect States, Improve Emotion Regulation, and Address Alexithymia -- 14.2.2.5 Mindfulness-Based (MB) Approaches -- 14.2.2.6 Body Image Work -- 14.2.3 Importance of Sleep -- 14.2.4 Importance of Exercise -- 14.2.5 Importance of Getting Psychiatric and Psychological Care -- 14.2.6 Neuromodulation Techniques -- 14.2.7 12-Step Programs and Other Support -- 14.2.8 Medications -- 14.2.9 Bariatric Surgery -- 14.3 Subtyping and FA Treatment Matching -- 14.3.1 Within-FA Treatment Matching -- 14.3.2 Using FA as a Treatment Matching Variable for Patients with Obesity and BE -- 14.4 Conclusion -- References -- Part VI: Research Possibilities -- 15: Emerging Treatments and Areas for Future Research -- 15.1 Emerging Treatments for Disordered Eating -- 15.1.1 Neurostimulation -- 15.1.2 Real-Time fMRI (Rt-fMRI) Neurofeedback Training.
15.1.3 Cognitive Training.
Record Nr. UNINA-9910502621703321
Wilcox Claire E.  
Cham, Switzerland : , : Springer, , [2021]
Materiale a stampa
Lo trovi qui: Univ. Federico II
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