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The biology of human longevity [[electronic resource] ] : inflammation, nutrition, and aging in the evolution of lifespans / / Caleb E. Finch
The biology of human longevity [[electronic resource] ] : inflammation, nutrition, and aging in the evolution of lifespans / / Caleb E. Finch
Autore Finch Caleb Ellicott
Pubbl/distr/stampa Amsterdam ; ; Boston, : Academic Press, c2007
Descrizione fisica 1 online resource (640 p.)
Disciplina 612.6/8
Soggetto topico Longevity - Physiological aspects
Aging - Physiological aspects
Inflammation
Nutrition
Soggetto genere / forma Electronic books.
ISBN 1-281-03721-4
9786611037215
0-08-054594-7
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Front Cover; The Biology of Human Longevity; Copyright Page; Contents; Preface; Acknowledgments; Chapter 1: Inflammation and Oxidation in Aging and Chronic Diseases; Part I; 1.1. Overview; 1.2. Experimental Models for Aging; 1.2.1. Mortality Rate Accelerations; 1.2.2. Mammals; 1.2.3. Cultured Cell Models and Replicative Senescence; 1.2.4. Invertebrate Models; 1.2.5. Yeast; 1.2.6. The Biochemistry of Aging; 1.2.7. Biomarkers of Aging and Mortality Risk Markers; 1.2.8. Evolutionary Theories of Aging; 1.3. Outline of Inflammation; 1.3.1. Innate Defense Mechanisms
1.3.2. Genetic Variations of Inflammatory Responses1.3.3. Inflammation and Energy; 1.3.4. Amyloids and Inflammation; 1.4. Bystander Damage and Dependent Variables in Senescence; 1.4.1. Free Radical Bystander Damage (Type 1); 1.4.2. Glyco-oxidation (Type 2); 1.4.3. Chronic Proliferation (Type 3); 1.4.4. Mechanical Bystander Effects (Type 4); Part II; 1.5. Arterial Aging and Atherosclerosis; 1.5.1. Overview and Ontogeny; 1.5.2. Hazards of Hypertension; 1.5.3. Mechanisms; 1.5.3.1. Inflammation; 1.5.3.2. Hemodynamics; 1.5.3.3. Aging; 1.5.3.4. Endothelial Progenitor Cells
1.5.4. Blood Risk Factors for Vascular Disease and Overlap with Acute Phase Responses1.6. Alzheimer Disease and Vascular-related Dementias; 1.6.1. Neuropathology of Alzheimer Disease; 1.6.2. Inflammation in Alzheimer Disease; 1.6.3. Prodromal Stages of Alzheimer Disease; 1.6.4. Overlap of Alzheimer and Cerebrovascular Changes; 1.6.5. Insulin and IGF-1 in Vascular Disease and Alzheimer Disease; 1.6.6. Blood Inflammatory Proteins: Markers for Disease or Aging, or Both?; 1.7. Inflammation in Obesity; 1.8. Processes of Normal Aging in the Absence of Specific Diseases; 1.8.1. Brain
1.8.2. Generalized Inflammatory Changes in Normal Tissue Aging1.9. Summary; Chapter 2 :Infections, Inflammogens, and Drugs; 2.1. Introduction; 2.2. Vascular Disease; 2.2.1. Historical Associations of Infections and Vascular Mortality; 2.2.2. Modern Serologic Associations; 2.3. Infections from the Central Tube: Metchnikoff Revisited; 2.3.1. Humans: Leakage from Periodontal Disease and Possibly the Lower Intestine; 2.3.2. Worms and Flies as Models for Human Intestinal Microbial Intrusion; 2.4. Aerosols and Dietary Inflammogens; 2.4.1. Aerosols; 2.4.2. Food
2.5. Infections, Inflammation, and Life Span2.5.1. Historical Human Populations; 2.5.2. Longer Rodent Life Spans with Improved Husbandry; 2.6. Are Infections a Cause of Obesity?; 2.7. Inflammation, Dementia, and Cognitive Decline; 2.7.1. Alzheimer Disease; 2.7.2. HIV, Dementia, and Amyloid; 2.7.3. Peripheral Amyloids; 2.7.4. Inflammation and Cognitive Decline During 'Usual' Aging; 2.8. Immunosenescence and Stem Cells; 2.8.1. Immunosenescence and Cumulative Exposure; 2.8.2. Immunosenescence and Telomere Loss; 2.8.3. Inflammation and Stem Cells; 2.9. Cancer, Infection, and Inflammation
2.9.1. Helicobacter Pylori and Hepatitis B Virus
Record Nr. UNINA-9910458126103321
Finch Caleb Ellicott  
Amsterdam ; ; Boston, : Academic Press, c2007
Materiale a stampa
Lo trovi qui: Univ. Federico II
Opac: Controlla la disponibilità qui
The biology of human longevity [[electronic resource] ] : inflammation, nutrition, and aging in the evolution of lifespans / / Caleb E. Finch
The biology of human longevity [[electronic resource] ] : inflammation, nutrition, and aging in the evolution of lifespans / / Caleb E. Finch
Autore Finch Caleb Ellicott
Pubbl/distr/stampa Amsterdam ; ; Boston, : Academic Press, c2007
Descrizione fisica 1 online resource (640 p.)
Disciplina 612.6/8
Soggetto topico Longevity - Physiological aspects
Aging - Physiological aspects
Inflammation
Nutrition
ISBN 1-281-03721-4
9786611037215
0-08-054594-7
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Front Cover; The Biology of Human Longevity; Copyright Page; Contents; Preface; Acknowledgments; Chapter 1: Inflammation and Oxidation in Aging and Chronic Diseases; Part I; 1.1. Overview; 1.2. Experimental Models for Aging; 1.2.1. Mortality Rate Accelerations; 1.2.2. Mammals; 1.2.3. Cultured Cell Models and Replicative Senescence; 1.2.4. Invertebrate Models; 1.2.5. Yeast; 1.2.6. The Biochemistry of Aging; 1.2.7. Biomarkers of Aging and Mortality Risk Markers; 1.2.8. Evolutionary Theories of Aging; 1.3. Outline of Inflammation; 1.3.1. Innate Defense Mechanisms
1.3.2. Genetic Variations of Inflammatory Responses1.3.3. Inflammation and Energy; 1.3.4. Amyloids and Inflammation; 1.4. Bystander Damage and Dependent Variables in Senescence; 1.4.1. Free Radical Bystander Damage (Type 1); 1.4.2. Glyco-oxidation (Type 2); 1.4.3. Chronic Proliferation (Type 3); 1.4.4. Mechanical Bystander Effects (Type 4); Part II; 1.5. Arterial Aging and Atherosclerosis; 1.5.1. Overview and Ontogeny; 1.5.2. Hazards of Hypertension; 1.5.3. Mechanisms; 1.5.3.1. Inflammation; 1.5.3.2. Hemodynamics; 1.5.3.3. Aging; 1.5.3.4. Endothelial Progenitor Cells
1.5.4. Blood Risk Factors for Vascular Disease and Overlap with Acute Phase Responses1.6. Alzheimer Disease and Vascular-related Dementias; 1.6.1. Neuropathology of Alzheimer Disease; 1.6.2. Inflammation in Alzheimer Disease; 1.6.3. Prodromal Stages of Alzheimer Disease; 1.6.4. Overlap of Alzheimer and Cerebrovascular Changes; 1.6.5. Insulin and IGF-1 in Vascular Disease and Alzheimer Disease; 1.6.6. Blood Inflammatory Proteins: Markers for Disease or Aging, or Both?; 1.7. Inflammation in Obesity; 1.8. Processes of Normal Aging in the Absence of Specific Diseases; 1.8.1. Brain
1.8.2. Generalized Inflammatory Changes in Normal Tissue Aging1.9. Summary; Chapter 2 :Infections, Inflammogens, and Drugs; 2.1. Introduction; 2.2. Vascular Disease; 2.2.1. Historical Associations of Infections and Vascular Mortality; 2.2.2. Modern Serologic Associations; 2.3. Infections from the Central Tube: Metchnikoff Revisited; 2.3.1. Humans: Leakage from Periodontal Disease and Possibly the Lower Intestine; 2.3.2. Worms and Flies as Models for Human Intestinal Microbial Intrusion; 2.4. Aerosols and Dietary Inflammogens; 2.4.1. Aerosols; 2.4.2. Food
2.5. Infections, Inflammation, and Life Span2.5.1. Historical Human Populations; 2.5.2. Longer Rodent Life Spans with Improved Husbandry; 2.6. Are Infections a Cause of Obesity?; 2.7. Inflammation, Dementia, and Cognitive Decline; 2.7.1. Alzheimer Disease; 2.7.2. HIV, Dementia, and Amyloid; 2.7.3. Peripheral Amyloids; 2.7.4. Inflammation and Cognitive Decline During 'Usual' Aging; 2.8. Immunosenescence and Stem Cells; 2.8.1. Immunosenescence and Cumulative Exposure; 2.8.2. Immunosenescence and Telomere Loss; 2.8.3. Inflammation and Stem Cells; 2.9. Cancer, Infection, and Inflammation
2.9.1. Helicobacter Pylori and Hepatitis B Virus
Record Nr. UNINA-9910784658403321
Finch Caleb Ellicott  
Amsterdam ; ; Boston, : Academic Press, c2007
Materiale a stampa
Lo trovi qui: Univ. Federico II
Opac: Controlla la disponibilità qui
The biology of human longevity : inflammation, nutrition, and aging in the evolution of lifespans / / Caleb E. Finch
The biology of human longevity : inflammation, nutrition, and aging in the evolution of lifespans / / Caleb E. Finch
Autore Finch Caleb Ellicott
Edizione [1st ed.]
Pubbl/distr/stampa Amsterdam ; ; Boston, : Academic Press, c2007
Descrizione fisica 1 online resource (640 p.)
Disciplina 612.6/8
Soggetto topico Longevity - Physiological aspects
Aging - Physiological aspects
Inflammation
Nutrition
ISBN 1-281-03721-4
9786611037215
0-08-054594-7
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Front Cover; The Biology of Human Longevity; Copyright Page; Contents; Preface; Acknowledgments; Chapter 1: Inflammation and Oxidation in Aging and Chronic Diseases; Part I; 1.1. Overview; 1.2. Experimental Models for Aging; 1.2.1. Mortality Rate Accelerations; 1.2.2. Mammals; 1.2.3. Cultured Cell Models and Replicative Senescence; 1.2.4. Invertebrate Models; 1.2.5. Yeast; 1.2.6. The Biochemistry of Aging; 1.2.7. Biomarkers of Aging and Mortality Risk Markers; 1.2.8. Evolutionary Theories of Aging; 1.3. Outline of Inflammation; 1.3.1. Innate Defense Mechanisms
1.3.2. Genetic Variations of Inflammatory Responses1.3.3. Inflammation and Energy; 1.3.4. Amyloids and Inflammation; 1.4. Bystander Damage and Dependent Variables in Senescence; 1.4.1. Free Radical Bystander Damage (Type 1); 1.4.2. Glyco-oxidation (Type 2); 1.4.3. Chronic Proliferation (Type 3); 1.4.4. Mechanical Bystander Effects (Type 4); Part II; 1.5. Arterial Aging and Atherosclerosis; 1.5.1. Overview and Ontogeny; 1.5.2. Hazards of Hypertension; 1.5.3. Mechanisms; 1.5.3.1. Inflammation; 1.5.3.2. Hemodynamics; 1.5.3.3. Aging; 1.5.3.4. Endothelial Progenitor Cells
1.5.4. Blood Risk Factors for Vascular Disease and Overlap with Acute Phase Responses1.6. Alzheimer Disease and Vascular-related Dementias; 1.6.1. Neuropathology of Alzheimer Disease; 1.6.2. Inflammation in Alzheimer Disease; 1.6.3. Prodromal Stages of Alzheimer Disease; 1.6.4. Overlap of Alzheimer and Cerebrovascular Changes; 1.6.5. Insulin and IGF-1 in Vascular Disease and Alzheimer Disease; 1.6.6. Blood Inflammatory Proteins: Markers for Disease or Aging, or Both?; 1.7. Inflammation in Obesity; 1.8. Processes of Normal Aging in the Absence of Specific Diseases; 1.8.1. Brain
1.8.2. Generalized Inflammatory Changes in Normal Tissue Aging1.9. Summary; Chapter 2 :Infections, Inflammogens, and Drugs; 2.1. Introduction; 2.2. Vascular Disease; 2.2.1. Historical Associations of Infections and Vascular Mortality; 2.2.2. Modern Serologic Associations; 2.3. Infections from the Central Tube: Metchnikoff Revisited; 2.3.1. Humans: Leakage from Periodontal Disease and Possibly the Lower Intestine; 2.3.2. Worms and Flies as Models for Human Intestinal Microbial Intrusion; 2.4. Aerosols and Dietary Inflammogens; 2.4.1. Aerosols; 2.4.2. Food
2.5. Infections, Inflammation, and Life Span2.5.1. Historical Human Populations; 2.5.2. Longer Rodent Life Spans with Improved Husbandry; 2.6. Are Infections a Cause of Obesity?; 2.7. Inflammation, Dementia, and Cognitive Decline; 2.7.1. Alzheimer Disease; 2.7.2. HIV, Dementia, and Amyloid; 2.7.3. Peripheral Amyloids; 2.7.4. Inflammation and Cognitive Decline During 'Usual' Aging; 2.8. Immunosenescence and Stem Cells; 2.8.1. Immunosenescence and Cumulative Exposure; 2.8.2. Immunosenescence and Telomere Loss; 2.8.3. Inflammation and Stem Cells; 2.9. Cancer, Infection, and Inflammation
2.9.1. Helicobacter Pylori and Hepatitis B Virus
Record Nr. UNINA-9910813754903321
Finch Caleb Ellicott  
Amsterdam ; ; Boston, : Academic Press, c2007
Materiale a stampa
Lo trovi qui: Univ. Federico II
Opac: Controlla la disponibilità qui