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Biosimilars for Cancer Treatment : A Promising Approach
Biosimilars for Cancer Treatment : A Promising Approach
Autore Bhatt Shvetank
Edizione [1st ed.]
Pubbl/distr/stampa Singapore : , : Springer, , 2024
Descrizione fisica 1 online resource (316 pages)
Altri autori (Persone) DurejaHarish
Gunvantbhai PatelSamir
Samir PatelArchita
DuaKamal
ISBN 981-9730-01-5
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Record Nr. UNINA-9910869176203321
Bhatt Shvetank  
Singapore : , : Springer, , 2024
Materiale a stampa
Lo trovi qui: Univ. Federico II
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Immunotherapy Against Lung Cancer [[electronic resource] ] : Emerging Opportunities and Challenges / / edited by Shvetank Bhatt, Rajaraman Eri Eri, Bey-Hing Goh, Keshav Raj Paudel, Terezinha de Jesus Andreoli Pinto, Kamal Dua
Immunotherapy Against Lung Cancer [[electronic resource] ] : Emerging Opportunities and Challenges / / edited by Shvetank Bhatt, Rajaraman Eri Eri, Bey-Hing Goh, Keshav Raj Paudel, Terezinha de Jesus Andreoli Pinto, Kamal Dua
Autore Bhatt Shvetank
Edizione [1st ed. 2023.]
Pubbl/distr/stampa Singapore : , : Springer Nature Singapore : , : Imprint : Springer, , 2023
Descrizione fisica 1 online resource (393 pages)
Disciplina 616.994
Altri autori (Persone) EriRajaraman Eri
GohBey-Hing
PaudelKeshav Raj
Andreoli PintoTerezinha de Jesus
DuaKamal
Soggetto topico Cancer - Treatment
Tumors - Immunological aspects
Cancer Therapy
Tumour Immunology
ISBN 981-9971-41-1
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Chapter 1_An introduction to Lung cancer -- Chapter 2_Immunobiology of Lung cancer -- Chapter 3_Evolution of Lung cancer treatment from classical chemotherapy to advanced immunotherapy -- Chapter 4_Recent advances in the immunotherapy of Lung cancer -- Chapter 5_PD-1/PD-L1 inhibitors for the treatment of Lung cancer -- Chapter 6_CTLA-4 inhibitors for the treatment of Lung cancer -- Chapter 7_Adoptive T cell therapy for the treatment of Lung cancer -- Chapter 8_LAG-3 inhibitors for the treatment of Lung cancer -- Chapter 9_IDO and TGF-β inhibitors for the treatment of Lung cancer -- Chapter 10_OX40 and CD40 agonists for the treatment of Lung cancer -- Chapter 11_ICOS and GITR agonists for the treatment of Lung cancer -- Chapter 12_Vaccines and oncolytic virus for the treatment of Lung cancer.-Chapter 13_Targeting Toll-like receptors for the treatment of Lung cancer -- Chapter 14_Current and future perspectives of combining chemotherapy and stereotactic body radiation therapy with immunotherapy -- Chapter 15_Synergistic potential of PD-1/PD-L1 inhibitors and CTLA-4 inhibitors for the treatment of Lung cancer -- Chapter 16_Synergistic potential of antigen-specific vaccines and immunomodulatory agents for the treatment of Lung cancer -- Chapter 17_Important biomarkers for better evaluation of checkpoint inhibitors and another immunotherapy in Lung cancer -- Chapter 18_Insight on the clinical trials of immunotherapy for the treatment of Lung cancer -- Chapter 19_Future perspectives of the Cancer Immunotherapy for the treatment of Lung cancer.
Record Nr. UNINA-9910845494503321
Bhatt Shvetank  
Singapore : , : Springer Nature Singapore : , : Imprint : Springer, , 2023
Materiale a stampa
Lo trovi qui: Univ. Federico II
Opac: Controlla la disponibilità qui
Immunotherapy Against Lung Cancer [[electronic resource] ] : Emerging Opportunities and Challenges / / edited by Shvetank Bhatt, Rajaraman Eri Eri, Bey-Hing Goh, Keshav Raj Paudel, Terezinha de Jesus Andreoli Pinto, Kamal Dua
Immunotherapy Against Lung Cancer [[electronic resource] ] : Emerging Opportunities and Challenges / / edited by Shvetank Bhatt, Rajaraman Eri Eri, Bey-Hing Goh, Keshav Raj Paudel, Terezinha de Jesus Andreoli Pinto, Kamal Dua
Autore Bhatt Shvetank
Edizione [1st ed. 2023.]
Pubbl/distr/stampa Springer Nature Singapore, 2023
Descrizione fisica 1 online resource (393 pages)
Disciplina 616.994
Altri autori (Persone) EriRajaraman Eri
GohBey-Hing
PaudelKeshav Raj
Andreoli PintoTerezinha de Jesus
DuaKamal
Soggetto topico Cancer - Treatment
Tumors - Immunological aspects
Cancer Therapy
Tumour Immunology
ISBN 981-9971-41-1
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Chapter 1_An introduction to Lung cancer -- Chapter 2_Immunobiology of Lung cancer -- Chapter 3_Evolution of Lung cancer treatment from classical chemotherapy to advanced immunotherapy -- Chapter 4_Recent advances in the immunotherapy of Lung cancer -- Chapter 5_PD-1/PD-L1 inhibitors for the treatment of Lung cancer -- Chapter 6_CTLA-4 inhibitors for the treatment of Lung cancer -- Chapter 7_Adoptive T cell therapy for the treatment of Lung cancer -- Chapter 8_LAG-3 inhibitors for the treatment of Lung cancer -- Chapter 9_IDO and TGF-β inhibitors for the treatment of Lung cancer -- Chapter 10_OX40 and CD40 agonists for the treatment of Lung cancer -- Chapter 11_ICOS and GITR agonists for the treatment of Lung cancer -- Chapter 12_Vaccines and oncolytic virus for the treatment of Lung cancer.-Chapter 13_Targeting Toll-like receptors for the treatment of Lung cancer -- Chapter 14_Current and future perspectives of combining chemotherapy and stereotactic body radiation therapy with immunotherapy -- Chapter 15_Synergistic potential of PD-1/PD-L1 inhibitors and CTLA-4 inhibitors for the treatment of Lung cancer -- Chapter 16_Synergistic potential of antigen-specific vaccines and immunomodulatory agents for the treatment of Lung cancer -- Chapter 17_Important biomarkers for better evaluation of checkpoint inhibitors and another immunotherapy in Lung cancer -- Chapter 18_Insight on the clinical trials of immunotherapy for the treatment of Lung cancer -- Chapter 19_Future perspectives of the Cancer Immunotherapy for the treatment of Lung cancer.
Record Nr. UNINA-9910853990503321
Bhatt Shvetank  
Springer Nature Singapore, 2023
Materiale a stampa
Lo trovi qui: Univ. Federico II
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Medicinal plants for lung diseases : a pharmacological and immunological perspective / / Kamal Dua [and five others], editors
Medicinal plants for lung diseases : a pharmacological and immunological perspective / / Kamal Dua [and five others], editors
Pubbl/distr/stampa Singapore : , : Springer, , [2021]
Descrizione fisica 1 online resource (625 pages)
Disciplina 616.248
Soggetto topico Emphysema, Pulmonary - Alternative treatment
Lungs - Diseases, Obstructive - Alternative treatmenT
Malalties del pulmó
Plantes medicinals
Medicina alternativa
Quimioteràpia
Soggetto genere / forma Llibres electrònics
ISBN 981-336-850-0
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Record Nr. UNINA-9910494558003321
Singapore : , : Springer, , [2021]
Materiale a stampa
Lo trovi qui: Univ. Federico II
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Role of Oxidative Stress in Pathophysiology of Diseases / / edited by Pawan K. Maurya, Kamal Dua
Role of Oxidative Stress in Pathophysiology of Diseases / / edited by Pawan K. Maurya, Kamal Dua
Edizione [1st ed. 2020.]
Pubbl/distr/stampa Singapore : , : Springer Singapore : , : Imprint : Springer, , 2020
Descrizione fisica 1 online resource (303 pages)
Disciplina 616.39
Soggetto topico Oxidative stress
Human physiology
Cancer research
Diabetes
Neurosciences
Nanotechnology
Oxidative Stress
Human Physiology
Cancer Research
Fisiologia patològica
Estrès oxidatiu
Soggetto genere / forma Llibres electrònics
ISBN 981-15-1568-9
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Chapter 1. Oxidative stress and oral diseases -- Chapter 2. Role of Oxidative stress in Chronic Liver Diseases -- Chapter 3. In-vitro and In-vivo Antioxidant activity of nanoparticles -- Chapter 4. Oxidative stress in neurology and neurodegenerative processes -- Chapter 5. The role of oxidative stress in respiratory diseases -- Chapter 6. Role of Synbiotics in alleviating oxidative stress in colorectal cancer -- Chapter 7. Oxidative Stress and Immunological Complexities in Multi-Drug Resistant Tuberculosis -- Chapter 8. Infection-induced Oxidative Stress in Chronic Respiratory Diseases -- Chapter 9. Oxidative stress in neuropsychiatric and neurodegenerative diseases -- Chapter 10. Oxidative stress monitoring in in vitro and in vivo models -- Chapter 11. Effects of Yoga on Aging.
Record Nr. UNINA-9910409682603321
Singapore : , : Springer Singapore : , : Imprint : Springer, , 2020
Materiale a stampa
Lo trovi qui: Univ. Federico II
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Targeting cellular signalling pathways in lung diseases / / Kamal Dua [and four others], editors
Targeting cellular signalling pathways in lung diseases / / Kamal Dua [and four others], editors
Pubbl/distr/stampa Gateway East, Singapore : , : Springer, , [2021]
Descrizione fisica 1 online resource (921 pages)
Disciplina 616.2407
Soggetto topico Lungs - Diseases - Molecular aspects
ISBN 981-336-827-6
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Intro -- Preface -- Acknowledgment -- Contents -- About the Editors -- 1: Introduction to Lung Diseases -- 1.1 Chronic Obstructive Pulmonary Disorder (COPD) -- 1.1.1 Definition -- 1.1.2 Epidemiology -- 1.1.3 Causes -- 1.1.4 Symptoms (Fig. 1.2) -- 1.1.5 Pathophysiology -- 1.1.6 Treatment -- 1.2 Asthma -- 1.2.1 Definition -- 1.2.2 Epidemiology -- 1.2.3 Causes (Fig. 1.3) -- 1.2.4 Symptoms (Fig. 1.4) -- 1.2.5 Pathophysiology -- 1.2.6 Types of Asthma [40] -- 1.2.7 Treatment -- 1.3 Lung Cancer -- 1.3.1 Classification -- 1.3.2 Epidemiology -- 1.3.3 Etiology and Risk Factors [45] (Fig. 1.6) -- 1.3.4 Sign and Symptoms -- 1.3.5 Diagnosis -- 1.3.6 Staging of Lung Cancer -- 1.3.7 Treatment [54] -- 1.4 Acute Respiratory Tract Infections -- 1.4.1 Upper Respiratory Tract Infections -- 1.4.1.1 Acute Pharyngitis -- 1.4.1.2 Acute Sinusitis -- 1.4.1.3 Acute Viral Laryngitis -- 1.4.2 Lower Respiratory Tract Infection -- 1.4.2.1 Pneumonia -- 1.4.2.2 Influenza -- 1.5 Idiopathic Pulmonary Fibrosis (IPF) -- 1.5.1 Epidemiology -- 1.5.2 Causes -- 1.5.3 Pathophysiology -- 1.5.4 Symptoms -- 1.5.5 Diagnosis -- 1.5.6 Treatment -- References -- 2: Targeting Molecular and Cellular Mechanisms in Asthma -- 2.1 Introduction -- 2.1.1 Rationale to the Study -- 2.1.2 Asthma: A Global Health Burden -- 2.1.3 Asthma: GINA Definition -- 2.2 Traditional Understanding and Parallel Non-specific Therapy -- 2.2.1 Major Types of Asthma -- 2.2.2 Allergens Are Major Inducers for Atopic Asthma -- 2.2.3 Pathophysiology -- 2.2.4 Symptomatic Therapy -- 2.3 Current Understanding and Steps Towards Pathway-Specific Therapy(Fig. 2.1) -- 2.3.1 Cells Involved in Asthma Pathogenesis and Respective Targets -- 2.3.1.1 Airway Epithelium -- 2.3.1.2 Dendritic Cells -- Dendritic Cell Modulator as a Target -- 2.3.1.3 Lymphocytes -- 2.3.1.4 T Helper Type 2 (Th2) Cells -- Th2 Cytokines as Target.
2.3.1.5 B Lymphocytes and IgE -- 2.3.1.6 Mast Cells -- 2.3.2 Th2-High Eosinophilic Asthma Versus Th2-Low Neutrophilic Asthma -- 2.3.2.1 Eosinophilic Asthma/Th2-High Asthma -- 2.3.2.2 Neutrophilic Asthma -- 2.3.3 Asthma: A Complex Syndrome with Multiple Endotypes -- 2.4 Nitro-Oxidative Stress in Asthma Pathogenesis -- 2.4.1 Oxidative Stress -- 2.4.2 Mitochondrial Dysfunction in Asthma -- 2.4.3 Nitric Oxide (NO) -- 2.5 Airway Remodelling in Asthma: The Knowledge that Will Shape the Future Treatment -- 2.6 Non-pharmacological Intervention for Asthma: Bronchial Thermoplasty -- 2.7 Tailoring the Patient Needs Via `Personalised Approach´ in Asthma -- 2.8 Conclusions and Future Perspectives -- References -- 3: Various Cellular and Molecular Axis Involved in the Pathogenesis of Asthma -- 3.1 Introduction -- 3.2 Epidemiology and Risk Factors -- 3.3 Granulocytes: As Predictors of Asthma -- 3.3.1 Basophils and Allergic Inflammation -- 3.3.1.1 Stimuli for Basophil Activation -- 3.3.1.2 Inflammatory Mediators of Basophils -- 3.3.1.3 Basophil Adhesion Molecules -- 3.3.2 Neutrophil Granule Contents -- 3.3.2.1 Mechanisms in Neutrophil Activation -- 3.3.2.2 Neutrophil in Airway Inflammation and its Signal Transduction Pathways -- 3.3.2.3 Neutrophil Apoptosis: The Resolution of Inflammation -- 3.3.3 Eosinophilic Asthma -- 3.3.3.1 Eosinophil-Derived Cytokines and Associated Bronchial Hyperresponsiveness -- 3.4 Fibroblasts and Myofibroblasts -- 3.4.1 Extracellular Matrix Production in the Airways -- 3.4.2 Fibroblast to Myofibroblast Transition -- 3.5 Pathological Signaling Pathways Involved in the Activation of Lung-Resident Macrophages and Dendritic Cells (DCs) -- 3.5.1 Macrophage Activation Pathways -- 3.5.1.1 IL-4/IL-13 Signaling Pathway -- 3.5.1.2 TNF-α Signaling Pathway -- 3.5.1.3 TGF-β Signaling Pathway -- 3.5.2 Reactive Oxygen and Nitrogen Species.
3.5.3 Antigen Presentation by Dendritic Cells and their Role in the Pathology of Asthma -- 3.6 Bronchial Epithelial Cells -- 3.6.1 Mechanisms of Endothelial Cell Activation -- 3.6.1.1 Cytokine Activation -- 3.6.1.2 Role of Epithelial Cells in Type 2-Driven and Non-type 2 Allergic Asthma -- 3.7 Airway Epithelial and Smooth Muscle Cells -- 3.7.1 Physiological Barrier -- 3.7.2 Airway Epithelial Cell-Derived Mediators -- 3.7.2.1 Nitric Oxide (NO) -- 3.7.2.2 Endothelin -- 3.7.2.3 Arachidonic Acid Metabolites -- 3.7.2.4 Inflammatory Cytokines -- 3.7.2.5 Cell Adhesion Molecules -- 3.7.2.6 Platelet-Activating Factor (PAF) -- 3.7.2.7 Tachykinin -- 3.7.2.8 Histamine -- 3.7.2.9 Adenosine -- 3.8 Role of Innate Immunity in Asthma -- 3.8.1 CD4+ and CD8+ T Cells in Asthma -- 3.8.2 Cellular Receptors of Innate Immunity in Asthma -- 3.8.3 Innate-like T Cells and Asthma -- 3.9 Conclusion -- References -- 4: Targeting Molecular and Cellular Mechanisms in Steroid-Resistant Asthma -- 4.1 Introduction -- 4.2 Inducers of Steroid Resistance in Asthma -- 4.3 Shedding Light on the Cellular Mechanisms Underlying the Glucocorticoid Receptor Signalling Pathway-Mediated Steroid Resis... -- 4.3.1 Neutrophils -- 4.3.2 Factors Inducing Airway Neutrophilia -- 4.3.3 Eosinophils -- 4.3.4 Group 2 Innate Lymphoid Cells (ILC-2) -- 4.3.5 T Helper Type 9 Cells (Th9 Cells) -- 4.3.6 CD8+ T Cells -- 4.4 Possible Molecular Mechanisms behind Steroid-Resistant (SR) Asthma (Fig. 4.1) -- 4.4.1 Bioavailability of Glucocorticoid Receptor (GR) and the Underlying Molecular Mechanism -- 4.4.2 IL-17A High and IFN-γ High Phenotypes -- 4.4.3 PI3K/HDAC Signalling Pathway -- 4.4.4 p38 MAPK -- 4.4.5 NLPR3 Inflammasome -- 4.4.6 Lack of Autophagy -- 4.4.7 Other Key Mechanisms -- 4.5 Tailoring Steroid-Refractory Asthma: The Possible Diagnosis and Therapeutics -- 4.5.1 Immunosuppressive Drugs.
4.5.2 Biologic Therapies -- 4.5.3 Highly Potent Glucocorticoids -- 4.5.4 Vitamin D -- 4.5.5 Sulforaphane -- 4.6 Conclusions and Future Outlook -- References -- 5: Targeting Molecular and Cellular Mechanisms in Chronic Obstructive Pulmonary Disease -- 5.1 Introduction -- 5.2 Pathophysiology of COPD -- 5.2.1 Inflammatory Mediators and Cells -- 5.2.1.1 Neutrophils (NPHs) -- 5.2.1.2 Macrophages (MPs) -- 5.2.1.3 T-Lymphocyte -- Eosinophils (Eos) -- 5.2.1.4 Dendritic Cells (DCs) -- 5.2.1.5 Epithelial Cells (EP) -- 5.2.2 Protease and Antiprotease Imbalance -- 5.2.3 Oxidative Stress (OS) -- 5.3 Mediators Involved in COPD -- 5.3.1 Lipid Mediators -- 5.3.1.1 Prostaglandin -- 5.3.1.2 Thromboxane -- 5.3.1.3 Leukotrienes (LT) -- 5.3.1.4 Platelet-Activating Factor (PAF) -- 5.3.2 Peptide Mediators -- 5.3.2.1 Endothelins (ET) -- 5.3.2.2 Bradykinin (BK) -- 5.3.2.3 Tachykinins (TK) -- 5.3.2.4 Chemokines -- 5.3.2.5 Interleukin-8 (IL-8) -- 5.4 Potential Targets and Therapies for COPD -- 5.4.1 PDE (Phosphodiesterase) Inhibitors -- 5.4.2 Hypersecretion of Mucus -- 5.4.3 Bronchodilators -- 5.4.4 Glucocorticoids (Inhaled Corticosteroids) -- 5.4.5 Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) -- 5.4.5.1 CXCR2 Inhibitors -- 5.4.6 Phosphoinositide-3 Kinase Delta (PI3K) Inhibitor -- 5.4.7 Targeting Inflammatory Markers -- 5.4.8 Other COPD Treatments -- 5.5 Conclusion -- References -- 6: Probing the Cellular and Molecular Mechanisms Underlying in the Pathogenesis of Chronic Obstructive Pulmonary Disease -- 6.1 Introduction -- 6.2 Subtypes of COPD -- 6.2.1 Chronic Bronchitis -- 6.2.2 Emphysema -- 6.2.3 Small Airway Disease -- 6.3 Inflammatory Cells Involved in COPD -- 6.3.1 Epithelial Cells -- 6.3.2 Macrophages -- 6.3.3 Neutrophils -- 6.3.4 Eosinophil -- 6.3.5 Dendritic Cells -- 6.3.6 T-Lymphocytes -- 6.4 Inflammatory Mediators Involved in COPD.
6.4.1 Lipid Mediators -- 6.4.2 Pro-inflammatory Cytokines -- 6.4.3 Inflammasomes -- 6.4.4 Role of Reactive Oxygen Species (ROS) and Oxidative Stress in COPD -- 6.5 Conclusion -- References -- 7: Chronic Obstructive Pulmonary Disease: Molecular Basis of Pathogenesis and Targeted Therapeutic Approaches -- 7.1 Introduction -- 7.2 Epidemiology -- 7.3 Aetiology of Disease Progression -- 7.3.1 Inflammation -- 7.3.1.1 Neutrophils -- 7.3.1.2 Macrophages -- 7.3.1.3 T Lymphocytes -- 7.3.1.4 Eosinophils -- 7.3.1.5 Dendritic Cells -- 7.3.1.6 Epithelial Cells -- 7.3.2 Oxidative Stress -- 7.3.3 Genetic Predisposition -- 7.3.4 Epigenetics -- 7.3.5 Activity of Proteases -- 7.3.5.1 Neutrophil Wlastase -- 7.3.5.2 Cysteine Proteases -- 7.3.5.3 Matrix Metalloproteinases -- 7.4 Current Treatments and Their Drawbacks -- 7.4.1 Quit Smoking -- 7.4.2 Vaccination -- 7.4.3 Physical Activity -- 7.4.4 Pharmacological Treatment -- 7.4.5 Emerging Antioxidants for an Alternative Therapeutic Approach -- 7.4.6 Interventional Treatments -- 7.4.7 Oxygen and Ventilator Support -- 7.4.8 Comorbidity Treatment -- 7.5 Conclusion -- References -- 8: Exploring the `Dormancy Activation Switch´ in the Tumour Microenvironment for Metastatic Lung Cancer: The Possible Role of ... -- 8.1 Lung Cancer -- 8.2 Metastatic Niche in Different Sites -- 8.3 Lymph Node Metastasis -- 8.3.1 MAPK Signalling in Lymph Node Metastasis of Lung Cancer -- 8.4 Pleura Metastasis -- 8.4.1 CXCR4/CXCL12 Signalling and Pleura Metastasis in Lung Cancer -- 8.5 Liver Metastasis -- 8.5.1 ALK and Liver Metastasis in Lung Cancer -- 8.6 Brain Metastasis -- 8.6.1 Rho/ROCK Signalling and Brain Metastasis in Lung Cancer -- 8.6.2 PI3K/AKT Signalling and Brain Metastasis in Lung Cancer -- 8.7 Bone Metastasis -- 8.7.1 RANK/RANKL Signalling and Bone Metastasis in Lung Cancer -- 8.8 Controversial Role miRNA in Lung Cancer Metastasis.
8.9 Concluding Remarks.
Record Nr. UNINA-9910488713203321
Gateway East, Singapore : , : Springer, , [2021]
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Lo trovi qui: Univ. Federico II
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Understanding Allergic Airway Diseases : Contemporary Treatment Paradigm / / edited by Parteek Prasher, Mousmee Sharma, Sachin Kumar Singh, Ronan MacLoughlin, Kavita Pabreja, Kamal Dua
Understanding Allergic Airway Diseases : Contemporary Treatment Paradigm / / edited by Parteek Prasher, Mousmee Sharma, Sachin Kumar Singh, Ronan MacLoughlin, Kavita Pabreja, Kamal Dua
Autore Prasher Parteek
Edizione [1st ed. 2024.]
Pubbl/distr/stampa Singapore : , : Springer Nature Singapore : , : Imprint : Springer, , 2024
Descrizione fisica 1 online resource (271 pages)
Disciplina 616.202
Altri autori (Persone) SharmaMousmee
SinghSachin Kumar
MacLoughlinRonan
PabrejaKavita
DuaKamal
Soggetto topico Pharmacology
Pharmaceutical chemistry
Pharmacovigilance
Immunology
Nanotechnology
Quantum dots
Pharmaceutics
Drug Safety and Pharmacovigilance
Quantum Dots
ISBN 9789819719532
9789819719525
Formato Materiale a stampa
Livello bibliografico Monografia
Lingua di pubblicazione eng
Nota di contenuto Chapter 1. Introduction to Allergic Airway Diseases -- Chapter 2. Pathophysiology of Allergic airway diseases -- Chapter 3. Immunology of Allergic Airway Diseases -- Chapter 4. Epidemiology of Allergic Airway Diseases -- Chapter 5. Nanotechnology in the Diagnosis of Allergic Airway Diseases -- Chapter 6. Metal and metal oxide Nanoparticles for the Management of Allergic Airway Diseases -- Chapter 7.-Nanomicelles for the Management of Allergic Airway Diseases -- Chapter 8 -- Carbon nanotubes (CNTs): A novle therapeutic approach in the management of allergic airway disease -- Chapter 9. Liposomes for the Management of Allergic Airway Diseases -- Chapter 10. Nanoparticles Solutions for Allergic Airway Disorders: Current Trends and Future Prospects -- Chapter 11. Solid lipid nanoparticles for the Management of Allergic Airway Diseases -- Chapter 12. Role of Dendrimers in Management of Allergic Airway Diseases -- Chapter 13. Nanofibers for the Management of Allergic Airway Diseases -- Chapter 14. Quantum dots for the Management of Allergic Airway Diseases -- Chapter 15. Future Directions in Allergic Airway Diseases.
Record Nr. UNINA-9910869168903321
Prasher Parteek  
Singapore : , : Springer Nature Singapore : , : Imprint : Springer, , 2024
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Lo trovi qui: Univ. Federico II
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