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[null,"1.","Record Nr.",null,"UNINA9910449711703321"]
[null,null,"Autore",null,"Cripps Thomas"]
[null,null,"Titolo",null,"Slow fade to black [[electronic resource] ] : the Negro in American film, 1900-1942 / / Thomas Cripps"]
[null,null,"Pubbl/distr/stampa",null,"New York, : Oxford University Press, 1993"]
[null,null,"ISBN",null,"1-280-43908-4"]
[{"$nulls":4},"0-19-972787-2"]
[{"$nulls":4},"1-60129-568-5"]
[null,null,"Descrizione fisica",null,"1 online resource (0 p. ) : 99 halftones"]
[null,null,"Disciplina",null,"791.43/028/0922"]
[null,null,"Soggetti",null,"African Americans in motion pictures"]
[{"$nulls":4},"African Americans in the motion picture industry"]
[{"$nulls":4},"Electronic books."]
[null,null,"Lingua di pubblicazione",null,"Inglese"]
[null,null,"Formato",null,"Materiale a stampa"]
[null,null,null,"Livello bibliografico","Monografia"]
[null,null,null,"Note generali","Cover title: The Negro in American film, 1900-1942."]
[null,null,null,"Nota di bibliografia","Includes bibliographical references and indexes."]
[null,null,null,"Sommario/riassunto","Slow Fade to Black is a history of US African-American accomplishment in film from the earliest movies through World War II. It explores the growth of discrimination as filmmakers became more and more intrigued with myths of the Old South."]
[null,"2.","Record Nr.",null,"UNINA9910161647903321"]
[null,null,"Autore",null,"Gilles J. Guillemin"]
[null,null,"Titolo",null,"Glial Cells: Managers of Neuro-immunity"]
[null,null,"Pubbl/distr/stampa",null,"Frontiers Media SA, 2016"]
[null,null,"Descrizione fisica",null,"1 online resource (224 p.)"]
[null,null,"Collana",null,"Frontiers Research Topics"]
[null,null,"Soggetti",null,"Neurosciences"]
[null,null,"Lingua di pubblicazione",null,"Inglese"]
[null,null,"Formato",null,"Materiale a stampa"]
[null,null,null,"Livello bibliografico","Monografia"]
[null,null,null,"Sommario/riassunto","Immune responses within the brain are still scarcely explored. Nerve tissue damage is accompanied by the activation of glial cells, primarily microglia and astroglia, and such activation is responsible for the release of cytokines and chemokines that maintain the local inflammatory response and actively recruit lymphocytes and monocytes to the damaged areas. Theoretically, these responses are designed to repair the brain damage. However, alterations, or a chronic perpetuation of these responses may underlie a number of neuro-pathologies. It is thought that each inflammatory scenario within the brain have a specific biochemical footprint characterized by the release of determined cytokines, chemokines and growing factors able to define particular immunological responses. Alongside, glial cells transform their cell body, become larger and develop higher number of branches adopting an active morphological phenotype. These changes are related with the search of interactions with other cells, such as bystander resident cells of the brain parenchyma, but also cells homing from the blood stream. In this process, microglia and astrocytes communicates with other cells by the formation of specific intercellular connections that are still poorly understood. These interactions are complex and entail the arrangement of cytoskeletal compounds, secretory and phagocytic domains. In this particular crosstalk there is a two-way communication in which glial cells and target cells come together establishing interfaces with specific information exchange. This way, glial cells orchestrate the particular response recruiting cellular subsets within the central nervous system and organizing the resolution of the brain damage. In this Frontiers Research Topic, we "]
[{"$nulls":4},"compile a selection of articles unfolding diverse aspects of glial-derived inflammation, focused on neurodegenerative diseases and other nervous system disorders, with special emphasis on microglia/macrophages as leading actors managing neuro-immunity."]