LEADER 01123cam0-22003611i-450- 001 990000034950403321 005 20051129164216.0 035 $a000003495 035 $aFED01000003495 035 $a(Aleph)000003495FED01 035 $a000003495 100 $a20020821d1950----km-y0itay50------ba 101 0 $aeng 102 $aUS 105 $ay-------001yy 200 1 $aHigh-speed computing devices$fby the staff of Engineering research associates$gsupervised By C. B. Tompkins, and J. H.Wakelin$gedited by W. W. Stifler 210 $aNew York$cMcGraw-Hill book company$d1950 215 $aXI, 451 p.$cill.$d24 cm 610 0 $aAerodinamica sperimentale 676 $a621.39 702 1$aStifler,$bW. W. JR. 702 1$aTompkins,$bCharles Brown 702 1$aWakelin,$bJames Henry 710 02$aEngineering Research Associates$012992 801 0$aIT$bUNINA$gRICA$2UNIMARC 901 $aBK 912 $a990000034950403321 952 $a13 B 64 26$b21654$fFINBC 952 $a09 004-008$b0597$fDINAE 959 $aFINBC 959 $aDINAE 996 $aHigh-speed computing devices$9105853 997 $aUNINA LEADER 05316nam 22006614a 450 001 9910830049703321 005 20230617021322.0 010 $a1-280-52038-8 010 $a9786610520381 010 $a3-527-60529-0 010 $a3-527-60145-7 035 $a(CKB)1000000000019317 035 $a(EBL)481642 035 $a(OCoLC)174056223 035 $a(SSID)ssj0000104500 035 $a(PQKBManifestationID)11138246 035 $a(PQKBTitleCode)TC0000104500 035 $a(PQKBWorkID)10079755 035 $a(PQKB)11381024 035 $a(MiAaPQ)EBC481642 035 $a(EXLCZ)991000000000019317 100 $a20030331d2003 uy 0 101 0 $aeng 135 $aur|n|---||||| 181 $ctxt 182 $cc 183 $acr 200 00$aApoptosis and autoimmunity$b[electronic resource] /$fedited by M. Hermann and J.R. Kalden 210 $aWeinheim $cWiley-VCH$dc2003 215 $a1 online resource (405 p.) 300 $a"From mechanism to treatments"--Cover. 311 $a3-527-30442-8 320 $aIncludes bibliographical references and index. 327 $aApoptosis and Autoimmunity; Preface; Contents; List of Contributors; Part 1 General Features of Apoptosis; 1 Apoptosis and Autoimmunity; 1.1 Introduction; 1.2 Autoimmune Diseases Associated with Targeted Cell Destruction; 1.2.1 What is the Mode of Cell Death?; 1.2.2 What Cells and What Effector Pathways are Responsible for Cell Death?; 1.3 Autoimmune Diseases Associated with Enhanced Cell Growth and Survival; 1.4 Autoimmune Diseases Associated with Abnormal Processing of Dying Cells; 1.5 Conclusions; 1.6 References; 2 Caspase Knockouts: Matters of Life and Death 327 $a2.1 Death, Development and Immune Function2.2 Apoptotic Pathways: from Nematode to Mammals; 2.3 Triggering a Killer: General Aspects of Caspase Activation; 2.4 Caspase-1 and -11: More than Mediators of Inflammatory Cytokines?; 2.5 Caspase-8 and the FAS Signaling Pathway; 2.6 Caspase-3: The Chief Executioner?; 2.7 Caspase-9: Mitochondrial Activation and the Apoptosome; 2.8 Caspase-2: A Duality of Function; 2.9 Caspase-12: Responding to Stress; 2.10 Compensatory Caspase Activation: A Caveat to Knockout Analysis; 2.11 Caspases: More than Simple Killers; 2.12 Concluding Remarks; 2.13 References 327 $aPart 2 Clearance of Apoptotic Cells3 Anti-inflammatory and Immunoregulatory Effects of Apoptotic Cells; 3.1 Introduction; 3.2 Anti-inflammatory Effects of Apoptotic Cells on Monocytes/Macrophages; 3.3 The Role of Anti-inflammatory Cytokines for the Inhibition of Pro-inflammatory Cytokine Production; 3.4 Monocyte/Macrophage Receptors receiving the Anti-inflammatory Signal from Apoptotic Cells; 3.5 Intracellular Signaling Events Causing the Anti-inflammatory State in Macrophages; 3.6 Apoptotic Cells Impair MHC Class II Surface Expression on Monocytes 327 $a3.7 Influence of Apoptotic Cells on DC Function in Allogeneic MLR3.8 The Presence of Apoptotic Cells can Shift the T(h) Cell Response towards T(h)2; 3.9 Apoptotic Cells Suppress Delayed-type Hypersensitivity (DTH) In Vivo; 3.10 Necrosis and Inflammation; 3.11 Implications of the Anti-inflammatory and Immunodulatory Effects of Apoptotic Cells for Health and Disease; 3.11.1 Apoptosis and Pregnancy; 3.11.2 Apoptosis and Irradiation; 3.11.3 Apoptosis and Cancer; 3.11.4 Apoptosis and Infections; 3.11.5 Apoptosis and Blood Transfusions; 3.12 References; 4 Complement and Apoptosis; 4.1 Introduction 327 $a4.2 Programmed Cell Death (PCD)4.3 Complement; 4.4 Complement and Apoptosis; 4.4.1 Role of Complement in the Execution Phase; 4.4.2 Complement Activation by Apoptotic Cells; 4.5 Apoptosis, Complement and Autoimmunity; 4.6 References; 5 Soluble Factors that Bind to Dying Cells Control the Outcome of Corpse Disposal: The Role of Pentraxins, Collectins and Autoantibodies; 5.1 Introduction; 5.2 Soluble Factors Involved in Apoptotic Cell Recognition and Internalization; 5.2.1 Corpse Clearance at Rest: Collectins; 5.2.2 Corpse Clearance at Rest: Cationic Factors and Other PS-binding Moieties 327 $a5.2.3 Corpse Clearance during Acute Inflammation: Pentraxins 330 $aThis is the first comprehensive book about the relationship between apoptosis and autoimmune diseases. It offers a unique up-to-date overview on research results on the defective execution of apoptosis and the incomplete clearance of apoptotic cells. The molecular and cellular mechanisms involved are described in detail. As a possible consequence of apoptotic dysfunction, the development of severe autoimmune diseases (e.g., rheumatoid arthritis, systemic lupus erythematosus) is discussed. An outlook on future research topics includes the evaluation of novel therapeutic strategies. 606 $aApoptosis 606 $aAutoimmunity 606 $aAutoimmune diseases 615 0$aApoptosis. 615 0$aAutoimmunity. 615 0$aAutoimmune diseases. 676 $a571.9/73 676 $a616.97/8 676 $a616.978 701 $aHerrmann$b M$g(Martin),$cDr.$01702778 701 $aKalden$b J. 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Introduction; 2. Economic History and Modern India: Redifining the Link; 3. Rural Labour and Colonialism; 4. Agricultural Labour: Lessons from Wage Data; 5. Was there an Industrial Decline in India in the Early Nineteenth Century?; 6. Labour-intensive Industrialization; 7. Women and Industrialization; 8. Women in the Crafts; 9. Labour and Power: A Critique of 'Subaltern Studies'; 10. Conclusion; Notes; References; Index 330 $aAs author of the hugely influential The Economic History of India 1857-1947, Tirthankar Roy has established himself as the leading contemporary economic historian of India. Here, Roy turns his attention to labour and livelihood and the nature of economic change in the Subcontinent. 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