LEADER 08570nam 2200601 a 450 001 9910828555303321 005 20240410172613.0 010 $a1-62100-388-4 035 $a(CKB)2550000001043321 035 $a(EBL)3021398 035 $a(SSID)ssj0000883502 035 $a(PQKBManifestationID)12369742 035 $a(PQKBTitleCode)TC0000883502 035 $a(PQKBWorkID)10925515 035 $a(PQKB)10953650 035 $a(MiAaPQ)EBC3021398 035 $a(Au-PeEL)EBL3021398 035 $a(CaPaEBR)ebr10683056 035 $a(OCoLC)839302704 035 $a(EXLCZ)992550000001043321 100 $a20110801d2012 uy 0 101 0 $aeng 135 $aur|n|---||||| 181 $ctxt 182 $cc 183 $acr 200 00$aCoagulation$b[electronic resource] $ekinetics, structure formation and disorders /$fAnett M. Taloyan and David S. Bankiewicz, editors 205 $a1st ed. 210 $aHauppauge, N.Y. $cNova Science Publishers$dc2012 215 $a1 online resource (221 p.) 225 1 $aRecent advances in hematology research 300 $aDescription based upon print version of record. 311 $a1-62100-331-0 320 $aIncludes bibliographical references and index. 327 $aIntro -- COAGULATION -- COAGULATION -- CONTENTS -- PREFACE -- A NEW APPROACH TO THE THEORY OF BROWNIAN COAGULATION AND DIFFUSION-LIMITED REACTIONS -- Abstract -- 1. Part 1. Brownian Coagulation Theory -- 1.1. Introduction -- 1.2. Diffusion Relaxation in Ensemble of Brownian Particles -- 1.3. Coagulation Rate Equation -- 1.3.1. Applicability of the Diffusion Approach to Particles Coagulation -- 1.3.2. Diffusion Mixing Condition -- 1.4. Kinetic Regime: High Collision Frequency (0 c) -- 1.5. Kinetic Regime: Low Collision Frequency (0 c) -- 1.5.1. Continuum Mode (Ra ) -- 1.5.2. Free Molecular Mode (Ra ) -- 1.5.3. Transition Mode (Ra ) -- 1.5.4. Interpolation Formulas -- 1.5.5. Applicability Range of the Kinetic Approach -- 1.6. Next Approximation of the Random Walk Theory -- 1.6.1. Brownian Particles Coagulation -- 1.6.2. Heavy Vapor Molecules Condensation -- 1.7. Discussion -- CONCLUSION -- 2. Part 2. Diffusion-Limited Reaction Rate Theory -- 2.1. Introduction -- 2.2. Rate Equations -- 2.2.1. Applicability of the Diffusion Approach to Particles Collisions -- 2.2.2. Diffusion Mixing Condition -- 2.2.3. Applicability of the Reaction Rate Equation -- 2.3. Reaction Rate in 3-D Case -- 2.3.1. Continuum Mode (BAABBArrRaa,, ) -- 2.3.2. Free Molecular Mode (BAABaaR, ) -- 2.4. Reaction Rate in 2-D Case -- 2.5. Reaction Rate on 3-D Discrete Lattice -- 2.6. Reaction Rate on 2-D Discrete Lattice -- CONCLUSION -- APPENDIX -- ACKNOWLEDGMENTS -- REFERENCES -- DEREGULATION OF COAGULATION DURING SEPSIS-INDUCED DISSEMINATED INTRAVASCULAR COAGULATION -- Abstract -- INTRODUCTION -- 1. Physiological Coagulation and Fibrinolysis -- 2. Overview of Sepsis -- 3. Overview of Disseminated Intravvascular Coagulation -- 4. Deregulation of Coagulation by Bacteria -- 5. Deregulation of Fibrinolysis by Bacteria -- 6. Coagulation Factor and Inhibitor Therapies. 327 $aCONCLUSION -- Authors' CONTRIBUTIONS -- ACKNOWLEDGEMENTS -- REFERENCES -- COAGULATION: KINETIC, STRUCTURE, FORMATION AND DISORDERS -- Abstract -- 1. Introduction -- 2. Kinetics of Coagulation Systems and Clot Formation -- 2.1. Primary Hemostasis -- 2.2. Coagulation Is Constituted by Interacting Elements -- 2.3. The Extrinsic Route of Coagulation -- 2.4. The Intrinsic Route of Coagulation -- 2.5. The Cellular Model of Coagulation -- 2.6. Activation of the Coagulation System -- 3. Kinetics -- 3.1. Coagulation and Inflammatory Components -- 3.2. Coagulation and Interaction with Endothelial Cells and Leukocytes -- 3.3. Cross-Talk between Clotting and Complement System (C) -- 4. Disorders -- 4.1. Thrombophilia -- 4.1.1. Acquired Factors Associated to Thrombosis -- 4.1.1.1. Procoagulant States and Their Association with Humoral Immunity -- 4.1.1.2. Concepts of Antibodies in Thrombophilia -- 4.2. Genetic Factors Associated to Thrombophilia -- 4.2.1. Deficiency of Antithrombin III -- 4.2.2. Deficiency of Protein C -- 4.2.3. Deficiency of Protein S -- 4.2.4. Mutation of Factor V -- 4.2.5. Mutation G20210A of Prothrombin -- CONCLUSION -- REFERENCES -- FLOC CHARACTERISTICS AND THE INFLUENCING FACTORS -- ABSTRACT -- 1. INTRODUCTION -- 2. MATERIALS AND METHODS -- 2.1. Coagulant Preparation and Characteristics -- 2.2. Water Samples -- 2.3. Jar Tests and Floc On-Line Monitor -- 3. CHARACTERIZATION OF FLOCS FORMED BY DIFFERENT AL-BASED COAGULANTS -- 3.1. Floc Formation, Breakage and Re-Growth -- 3.2. Effect of Shear Rate on Floc Size -- 3.3. Floc Fractal Structure Analysis -- 4. EFFECT OF PH ON FLOC PROPERTIES -- 4.1. Effect of PH on Coagulation Efficiency -- 4.2. Effect of PH on Floc Formation, Breakage and Re-Growth -- 4.3. Effect of PH on Floc Fractal Structures -- CONCLUSION -- REFERENCES. 327 $aSUBSTRATE INDUCED COAGULATION (SIC) IN AQUEOUS AND NON-AQUEOUS MEDIA FOR THE PREPARATION OF ADVANCED BATTERY MATERIALS -- Abstract -- 1.SubstrateInducedCoagulation(SIC)inAqueousandNon-aqueousMedia -- 1.1.DLVOTheory -- 1.2.SurfaceCharging -- 1.3.Zeta-Potential -- 2.TheStabilityofAqueousandNon-aqueousDispersions -- 2.1.StabilityofNon-aqueousDispersions -- 2.2.TraceWaterinNon-aqueousDispersions -- 2.3.CarbonBlackDispersions -- 2.3.1.CarbonBlackDispersionsinPolarMedia -- 2.3.2.CarbonBlackDispersionsinNon-polarMedia -- 2.4.TitaniaandAluminaDispersions -- 2.4.1.TitaniaandAluminaDispersionsinPolarMedia -- 2.4.2.TitaniaandAluminaDispersionsinNon-polarMedia -- 3.AdvancedBatteryMaterials -- 3.1.TheCathodeMaterialLithiumCobaltOxide -- 3.2.HighlyConductiveCompositeElectrodes -- 3.3.Core-ShellCathodeMaterials -- Acknowledgements -- References -- THE LABORATORY DIAGNOSIS OF THE PRE-PHASE OF PATHOLOGIC INTRAVASCULAR COAGULATION -- Abstract -- Introduction -- Hitherto Routine Hemostasis Tests for PIC Diagnosis -- New Routine Hemostasis Tests for Diagnosis of Early PIC -- IIa -Test -- Ultra-specific IIa Generation Tests -- Fibrinogen Function+Antigen+Ratio -- Undiluted Antithrombin III Activity (AT3) -- Active Endotoxin = Endotoxin Reactivity -- Special Antigentic Parameters for PIC Diagnosis -- CONCLUSION -- REFERENCES -- NEONATAL COAGULATION PROBLEM -- Introduction to Coagulation Defect in Neonatal -- Platelet Defect in the Neonate -- Vascular Defect in the Neonate -- Coagulation Defect in the Neonate -- Thrombohemostatic Defect in the Neonate -- REFERENCES -- COAGULATION AND WALL SHEAR STRESS IN LIVING DONOR LIVER TRANSPLANTATION -- 1. Introduction -- 2. Shear Stress Theory and Liver Regeneration -- Following Phx -- 3. Concept of Immune System and Role of Shear Stress in Liver Regeneration Following Phx. 327 $a4. The Experimental and Clinical Data of Wall Shear Stress in the Liver -- Experimental Data -- A Comparison of the Phenotype of Cells Between the Liver and the Irrigation Solution. -- Abundance of NKT Cells in the Parenchymal Space of the Liver -- Clinical Data in LDLT -- Changes of Thymus-Derived Cells in the Graft Liver by the Perfusion of HTK -- Solution in LDLT -- Changes of NKT Cells Among CD3+T Cells in the Grafts Liver by the Perfusion of HTK Solusion in LDLT -- 5. Shear Stress and PAI-1 During Liver Regeneration Following Phx -- Flow-Induced Changes in Expression of the PAI-1 Gene in Hepatocytes -- Flow-Induced Changes in the Release of PAI-1 by Hepatocytes -- Shear Stress Dependency of Flow-Induced PAI-1 Expression -- 6. Blood Coagulation and Fibrinolytic Systems During Liver Regeneration in LDLT -- Patients and Methods -- Results -- 7. Heme Oxygenase-1 and Bilirubin Metabolism in Clinical LDLT -- Materials and Methods -- RESULTS -- Changes in Serum Total Bilirubin Following Adult LDLT -- Changes in D/T Ratio Following Adult LDLT -- Correlation Between Total Bilirubin and COHB Following Adult LDLT -- Augmentation of Heme Oxygenase-1 Expression in the Graft Immediately After Implantation in Adult LDLT -- CONCLUSION -- REFERENCES -- INDEX. 410 0$aRecent advances in hematology research series. 606 $aBlood$xCoagulation 606 $aBlood coagulation disorders 615 0$aBlood$xCoagulation. 615 0$aBlood coagulation disorders. 676 $a616.1/57 701 $aTaloyan$b Anett M$01719433 701 $aBankiewicz$b David S$01719434 801 0$bMiAaPQ 801 1$bMiAaPQ 801 2$bMiAaPQ 906 $aBOOK 912 $a9910828555303321 996 $aCoagulation$94117255 997 $aUNINA