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100   $a20111213d2011      uy         0
101 0 $aeng
135   $aur|n|---|||||
181   $ctxt
182   $cc
183   $acr
200 04$aThe molecular basis for the link between maternal health and the origin of fetal congenital abnormalities $ean overview of association with oxidative stress /$feditor, Bashir M. Matata ; co-editor, Maqsood M. Elahi
205   $a1st ed.
210   $a[Sharjah, United Arab Emirates $cBentham eBooks$d2011]
215   $a1 online resource (136 p.)
300   $aDescription based upon print version of record.
311 1 $a1-60805-536-1 
320   $aIncludes bibliographical references and index.
327   $a01 Title.pdf; 02 Cover page; 03 REVISED eBooks End User License Agreement-Website; 04 TABLE OF CONTENTS; 05 Forward Matata and Elahi; 06 Preface of the book; 07 CONTRIBUTORS; 08 CHAPTER 1- Elahi and Matata -Jun  2011; 09 CHAPTER 2-Dupont et al [R1]; 10 CHAPTER 3_Rev_final; 11 CHAPTER 4- Nagai [R1]; 12 CHAPTER 5- Elahi and Matata June 2011; 13 CHAPTER 6- Aly Rev; 14 CHAPTER 7- Mohn et al; 15 CHAPTER 8- Gonzalez et al Corrected Proof _May 24 11_; 16 CHAPTER_9-_Simeoni_et_al[1]; 17 Index- Matata et al Book
330   $aThis e-book discusses the molecular relationship between biological systems and risk factors for in-utero oxidative insults, maternal health and the impact on fetus at different critical developmental time windows (prenatal or even postnatal). Plausibly, prenatal and early postnatal periods are the most critical "windows" to oxidative stress programming insults. Pro-oxidative stress is a common feature of many conditions associated with adverse fetal growth and/or preterm birth, such as pre-eclampsia, diabetes, smoking, malnutrition or excessive nutrition, infection or inflammation.
606   $aFetal heart$xBlood-vessels$xAbnormalities
606   $aOxidative stress
615  0$aFetal heart$xBlood-vessels$xAbnormalities.
615  0$aOxidative stress.
676   $a600
702   $aMatata$b Bashir M.
702   $aElahi$b Maqsood M.
801  0$bMiAaPQ
801  1$bMiAaPQ
801  2$bMiAaPQ
906   $aBOOK
912   $a9910808521503321
996   $aThe molecular basis for the link between maternal health and the origin of fetal congenital abnormalities$94008174
997   $aUNINA