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Yuzhalin 205 $a1st ed. 2013. 210 1$aNew York, NY :$cSpringer New York :$cImprint: Springer,$d2013. 215 $a1 online resource (74 p.) 225 1 $aSpringerBriefs in Immunology,$x2194-2773 ;$v4 300 $aBibliographic Level Mode of Issuance: Monograph 311 $a1-4614-8862-1 320 $aIncludes bibliographical references and index. 327 $aThe Hypothesis of Virus-Driven Atherosclerosis -- The Role of Herpes Simplex Virus-1 and Herpes Simplex Virus-2 in Atherosclerosis -- How may HSV promote atherosclerosis? -- Evidence from epidemiological studies -- Evidence from cardiovascular events -- The Role of Epstein-Barr virus in Atherosclerosis -- EBV and atherosclerosis: the published data from basic and epidemiological investigations -- The Role of Enteroviruses, Parvovirus B19, Respiratory Syncytial Virus and Measles Virus in Atherosclerosis -- Enteroviruses -- Parvovirus B19 -- Respiratory Syncytial Virus (RSV) -- Measles Virus -- The Role of Hepatitis A, B, and C in Atherosclerosis -- Hepatitis A virus (HAV), atherosclerosis, and related diseases -- Hepatitis B virus (HBV), atherosclerosis, and related diseases -- Hepatitis C virus (HCV), atherosclerosis, and related diseases -- HCV and changes in immunological and atherotic profile -- HCV and subclinical atherosclerosis -- HCV and CAD -- HCV and myocardial infarction -- HCV and stroke -- Conclusions -- Summaries: Are We There Yet?. 330 $aWill address an important, yet underrepresented, topic. The correlation between viruses and atherosclerosis has been a focal point of the authors? work, for a number of years. This volume will explore the relationship between different viral strains and   atherosclerosis. It will begin by describing the hypothesis and denoting the mechanisms of virus-driven atherosclerosis, then expanding on the subject by focusing on different virus strains?from Herpes, to Epstein-Barr, to the triad of Hepatitis viruses, et al?on a chapter-by-chapter basis. 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