LEADER 02416nam 2200577Ia 450 001 9910782892503321 005 20230721005021.0 010 $a1-74223-073-3 035 $a(CKB)1000000000716502 035 $a(EBL)421340 035 $a(OCoLC)476256131 035 $a(SSID)ssj0000367761 035 $a(PQKBManifestationID)11265674 035 $a(PQKBTitleCode)TC0000367761 035 $a(PQKBWorkID)10343576 035 $a(PQKB)11232020 035 $a(MiAaPQ)EBC421340 035 $a(Au-PeEL)EBL421340 035 $a(CaPaEBR)ebr10355591 035 $a(EXLCZ)991000000000716502 100 $a20090219d2008 uy 0 101 0 $aeng 135 $aur|n|---||||| 181 $ctxt 182 $cc 183 $acr 200 10$aWorth fighting for$b[electronic resource] $einside the Your Rights at Work campaign /$fKathie Muir 210 $aSydney, NSW $cUNSW Press$d2008 215 $a1 online resource (255 p.) 300 $aDescription based upon print version of record. 311 $a1-921410-77-9 320 $aIncludes bibliographical references (p. 231-233) and index. 327 $aContents; preface and acknowledgments; abbreviations; prologue; The context of the challenge to the unions; The rise of media-driven politics; Meeting the communication challenge; Mobilising to win; Campaigning on the other side: selling Work Choices; Staying on message despite the differences; Doing politics differently; epilogue; notes; bibliography; index 330 $aThis book tells the story of the ACTU's 'Your Rights at Work' campaign against Work Choices, the largest, most expensive and most sophisticated political campaign ever mounted in Australia, and one with a decisive impact on the 2007 federal election. 606 $aIndustrial relations$zAustralia$y21st century 606 $aLabor laws and legislation$zAustralia$y21st century 606 $aPolitical campaigns$zAustralia$xPsychological aspects 606 $aEmotions$xPolitical aspects 615 0$aIndustrial relations 615 0$aLabor laws and legislation 615 0$aPolitical campaigns$xPsychological aspects. 615 0$aEmotions$xPolitical aspects. 676 $a331.0994 700 $aMuir$b Kathie$01537511 801 0$bMiAaPQ 801 1$bMiAaPQ 801 2$bMiAaPQ 906 $aBOOK 912 $a9910782892503321 996 $aWorth fighting for$93786847 997 $aUNINA LEADER 03965nam 22005775 450 001 9910409683603321 005 20200704063541.0 010 $a981-15-1580-8 024 7 $a10.1007/978-981-15-1580-4 035 $a(CKB)5300000000003643 035 $a(DE-He213)978-981-15-1580-4 035 $a(MiAaPQ)EBC6130754 035 $a(PPN)243229488 035 $a(EXLCZ)995300000000003643 100 $a20200309d2020 u| 0 101 0 $aeng 135 $aurnn|008mamaa 181 $ctxt$2rdacontent 182 $cc$2rdamedia 183 $acr$2rdacarrier 200 10$aLectin in Host Defense Against Microbial Infections /$fedited by Shie-Liang Hsieh 205 $a1st ed. 2020. 210 1$aSingapore :$cSpringer Singapore :$cImprint: Springer,$d2020. 215 $a1 online resource (XIV, 230 p. 25 illus., 21 illus. in color.) 225 1 $aAdvances in Experimental Medicine and Biology,$x0065-2598 ;$v1204 311 $a981-15-1579-4 320 $aIncludes bibliographical references. 327 $aC-type Lectin Receptors in Antifungal Immunity -- Immune recognition of pathogen-derived glycolipids through Mincle -- CLEC5A: A promiscuous pattern recognition receptor to microbes and beyond -- Collectins: Innate immune pattern recognition molecules -- Insect C-type lectins in microbial infections -- Galectins in Host Defense against Microbial Infections -- Galectins in Host-Pathogen Interactions: Structural, Functional and Evolutionary Aspects -- Siglecs at the Host-Pathogen Interface -- Siglecs that associate with DAP12. 330 $aThis book systemically presents the latest research on lectins, covering all the major topics in the field, including the heterocomplex of lectins and Toll-like receptors, protective versus pathogenic functions in connection with microbial infections, and novel strategies for enhancing host immunity against infectious diseases caused by viruses, bacteria, and fungi. Lectins are a large group of glycan-binding proteins that recognize diverse glycan and non-glycan structures expressed on prokaryotic and eukaryotic cells, and are vital to cell-cell interactions, the attachment of microbes to host cells, and the recognition and activation of immune responses to exogenous and endogenous danger signals. The composition and structure of microbes are complex and include numerous ?pathogen-associated molecular patterns? or ?damage-associated molecular patterns?. As such, microbes? interactions with immune cells activate multiple innate immunity receptors and produce distinct inflammatory reactions, which can be protective to contain microbial invasion, or pathogenic to cause tissue damage and shock syndrome in the host. 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