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100   $a20230630d2018      uy             0
101 0 $aeng
135   $aur|||||||||||
181   $ctxt$2rdacontent
182   $cc$2rdamedia
183   $acr$2rdacarrier
200 00$aViral Infection and Apoptosis /$fedited by Marc Kvansakul
210  1$aBasel, Switzerland :$cMDPI - Multidisciplinary Digital Publishing Institute,$d2018.
215   $a1 online resource (270 pages) $cillustrations
320   $aIncludes bibliographical references.
330   $aAnnotation Apoptosis is a form of programmed cell death that enables the removal of damaged, infected, or otherwise unwanted cells in a controlled manner. Apoptosis can be initiated by multiple independent pathways that ultimately converge at a point where proteolytic enzymes belonging to the caspase family are activated, which dismantle the apoptotic cell. Multicellular organism have employed apoptotic mechanisms during host defence in response to viral infection to limit or prevent viral spread and replication. Consequently, viruses have evolved sophisticated molecular countermeasures to disarm host apoptotic defences, and this series of reviews and primary research articles in this Special Issue explores the intricate molecular interplay between viruses and their hosts when they battle for control of host apoptotic check-points.
606   $aApoptosis
615  0$aApoptosis.
676   $a571.936
702   $aKvansakul$b Marc
801  0$bNjHacI
801  1$bNjHacl
906   $aBOOK
912   $a9910688427803321
996   $aViral Infection and Apoptosis$92929118
997   $aUNINA