LEADER 01443nam--2200397---450- 001 990000318980203316 005 20051111165934.0 010 $a2-911702-03-4 035 $a0031898 035 $aUSA010031898 035 $a(ALEPH)000031898USA01 035 $a0031898 100 $a20010105d1998----km-y0itay0103----ba 101 $afre 102 $aFR 105 $a||||||||001yy 200 1 $aHomo homini lupus ou l'homme détrompé el despeñadero de la vida$eune référence de Jacques Lacan dans ses Écrits au Criticòn de Baltasar Gracián$fpréface de Mercedes Blanco$gPostface de Débora Rabinovich 210 $aParis$cE.C.F.-A.C.F.$d1998 215 $a57p.$d22 cm 225 2 $aLes documents de la bibliothèque de l'Ecole de la Cause freudienne$v3 410 0$12001$aLes documents de la bibliothèque de l'École de la Cause freudienne 461 1$1001-------$12001 604 $aGraciàn, Baltasar. El Criticon 676 $a863.3 702 1$aLACAN,$bJacques 801 0$aIT$bsalbc$gISBD 912 $a990000318980203316 951 $aVI.5.A. 674(VI ps B 907)$b155478 L.M.$cVI ps B$d00008273 959 $aBK 969 $aUMA 979 $aTAMI$b40$c20010105$lUSA01$h1223 979 $c20020403$lUSA01$h1639 979 $aPATRY$b90$c20040406$lUSA01$h1622 979 $aCOPAT2$b90$c20051111$lUSA01$h1659 996 $aHomo homini lupus ou l'homme détrompé el despeñadero de la vida$9879199 997 $aUNISA LEADER 03719 am 2200673 n 450 001 9910313024703321 005 20180215 010 $a2-35596-000-3 024 7 $a10.4000/books.irasec.723 035 $a(CKB)4960000000012701 035 $a(FrMaCLE)OB-irasec-723 035 $a(oapen)https://directory.doabooks.org/handle/20.500.12854/45871 035 $a(PPN)229999735 035 $a(EXLCZ)994960000000012701 100 $a20180703j|||||||| ||| 0 101 0 $aeng 135 $auu||||||m|||| 181 $ctxt$2rdacontent 182 $cc$2rdamedia 183 $acr$2rdacarrier 200 10$aEducation, Economy and Identity $eTen years of Educational Reform in Thailand /$fSupat Chupradit, Audrey Baron-Gutty 210 $aBangkok $cInstitut de recherche sur l?Asie du Sud-Est contemporaine$d2018 215 $a1 online resource (118 p.) 311 $a616-90282-0-3 330 $aModern education in Thailand started at the end of the nineteenth century under the impulse of King Chulalongkorn. Many scholars tracing back the evolution from traditional education to a modern education system emphasized the feeling of necessity that motivated this transformation. Wyatt (1969), Mead (2004) and Watson (1982) underlined the need for a modern administration, to handle the Siamese nation-state ?as? the Western states, and in that respect, the key role played by education to structure the new Siam and to appear to the eyes of the world as civilized (Peleggi 2002). The shaping of a new education took place amidst strong political struggles. Siam needed to stand firm within the regional arena, swept by the winds of Western colonialism. Internally, King Chulalongkorn had to legitimize his power and to unify the kingdom by integrating satellite kingdoms into a wider space, the Siamese nation state. Education was vital for this mission as it would contribute not only to bringing state power into the provinces through state-paid teachers and government officials, but also to transmitting a whole nation-related imagery to the young generations. Giving rise to Thai-ness among the populations located at the margins of the kingdom was a tremendous ordeal. In the Southern part of the kingdom, population was mainly Muslim, spoke Malay and felt culturally closer to the Malay state (Dulyakasem 1991). In the Northern part, incorporating the Lanna kingdom and hill tribe populations into Siam proved not to be easy. Ideological, social and national values were introduced into education delivered to students, and with the implementation of the Compulsory Education Act of 1921, school attendance tied children and parents to the nation state and made them liable to it. 606 $aAsian Studies 606 $aéducation 606 $asavoirs 606 $aréformes 606 $aThaïlande 606 $aknowledge 606 $areform 606 $alocal 606 $aThailand 610 $areform 610 $aThailand 610 $alocal 610 $aknowledge 615 4$aAsian Studies 615 4$aéducation 615 4$asavoirs 615 4$aréformes 615 4$aThaïlande 615 4$aknowledge 615 4$areform 615 4$alocal 615 4$aThailand 700 $aBaron-Gutty$b Audrey$01314733 701 $aBuadaeng$b Kwanchewan$01314734 701 $aBurapharat$b Chitrlada$01314735 701 $aChupradit$b Supat$01314736 701 $aLeepreecha$b Prasit$01314737 701 $aChupradit$b Supat$01314736 701 $aBaron-Gutty$b Audrey$01314733 801 0$bFR-FrMaCLE 906 $aBOOK 912 $a9910313024703321 996 $aEducation, Economy and Identity$93031913 997 $aUNINA LEADER 07655nam 2202437z- 450 001 9910557787303321 005 20210501 035 $a(CKB)5400000000045515 035 $a(oapen)https://directory.doabooks.org/handle/20.500.12854/69055 035 $a(oapen)doab69055 035 $a(EXLCZ)995400000000045515 100 $a20202105d2020 |y 0 101 0 $aeng 135 $aurmn|---annan 181 $ctxt$2rdacontent 182 $cc$2rdamedia 183 $acr$2rdacarrier 200 00$aRecent Advances and Clinical Outcomes of Kidney Transplantation$eVolume 2 210 $aBasel, Switzerland$cMDPI - Multidisciplinary Digital Publishing Institute$d2020 215 $a1 online resource (270 p.) 311 08$a3-03936-407-3 311 08$a3-03936-408-1 330 $aAdvances in immunosuppression and kidney transplant techniques have led to significant improvements in the short-term survival of the renal allograft. Long-term graft survival, however, has relatively lagged behind and has now become one of the main problems in kidney transplantation. In this Special Issue, we discuss the recent research developments in kidney transplants that may affect long-term allografts, as well as the survival of the patient. The latest developments in living kidney donation are also explored. 517 $aRecent Advances and Clinical Outcomes of Kidney Transplantation 606 $aMedicine$2bicssc 610 $aacute humoral rejection 610 $aacute rejection 610 $aadaptive immune system 610 $aADPKD 610 $aage 610 $aall-cause mortality 610 $aallograft survival 610 $aAlport syndrome 610 $aantibiotic resistance 610 $aantibody-mediated rejection (AMR) 610 $aapoptosis 610 $aarsenic 610 $aatypical hemolytic uremic syndrome 610 $aautosomal dominant polycystic kidney disease 610 $aBelatacept 610 $abeliefs 610 $abiomarker 610 $aBK virus 610 $aBK virus nephropathy 610 $aBKV 610 $aBKVAN 610 $abranched chain amino acids 610 $aC/D ratio 610 $aC/D-ratio 610 $aC3d-binding assay 610 $acalcineurin inhibitor nephrotoxcity 610 $acalcineurin inhibitors 610 $acancer 610 $acancer mortality 610 $acardiovascular high risk 610 $acardiovascular mortality 610 $aCD45RC 610 $achildren 610 $acreatinine 610 $adelayed graft function 610 $adiet 610 $adietary intake 610 $adonor-specific antibodies (DSA) 610 $aeculizumab 610 $aendothelial dysfunction 610 $aepidemiology 610 $aepidermal growth factor 610 $aethnicity 610 $afast tacrolimus metabolizers 610 $afirst-line therapy 610 $afish consumption 610 $aFK506 610 $afluid management 610 $agenetic relationship donor/recipient 610 $agenomics 610 $aGFR 610 $aglomerulus 610 $aglucose intolerance 610 $agoal-directed fluid therapy 610 $agraft failure 610 $agraft survival 610 $agrowth hormone 610 $ahealthy subject 610 $ahospitalization 610 $ahyperchloremia 610 $ahyperoxaluria 610 $ahypomagnesaemia 610 $ahypothermic 610 $ahypoxic inducible factor 610 $aimmunosuppression 610 $ainequity 610 $ainfectious mortality 610 $ainnate immune system 610 $ainsulin resistance 610 $ainsulin secretion 610 $ainsulin-like growth factor 1 610 $airon 610 $airon deficiency 610 $aischemia reperfusion injury 610 $akidney 610 $akidney allograft 610 $akidney function 610 $akidney graft dysfunction 610 $akidney transplant 610 $aKidney transplant 610 $akidney transplant (KT) 610 $akidney transplant recipients 610 $akidney transplantation 610 $aliving donor 610 $aliving donor kidney transplantation 610 $aliving kidney donation 610 $aliving-donor kidney transplantation 610 $alymphocyte 610 $amachine perfusion 610 $amagnesium 610 $amalignancy 610 $amalondialdehyde 610 $ameta-analysis 610 $amethanogenesis 610 $aMethanosphaera stadtmanae 610 $amortality 610 $amucins 610 $amuscle mass 610 $amycophenolic acid 610 $an/a 610 $aN1-methyl-2-pyridone-5-carboxamide 610 $aN1-methylnicotinamide 610 $anative nephrectomy 610 $anecrosis 610 $anephropathy 610 $anephrotoxicity 610 $anew onset diabetes after renal transplantation 610 $anew-onset diabetes 610 $aniacin status 610 $anight-time renal transplantation 610 $anormothermic 610 $aobesity 610 $aopportunistic infection 610 $aoral glucose tolerance test 610 $aorgan preservation 610 $aoutcome 610 $aoutcomes 610 $aoxalate 610 $aoxidative stress 610 $aoxidative stress. 610 $apatient outcome 610 $apatient survival 610 $apatient survival/outcome 610 $aperioperative complications 610 $apharmacokinetic 610 $apharmacokinetics 610 $aphylogeny 610 $aphysical activity 610 $aplasmapheresis 610 $apolyomavirus 610 $apost-transplant diabetes mellitus 610 $apost-transplant malignancy 610 $apost-transplantation diabetes mellitus 610 $aposttransplant diarrhea 610 $aproteasome inhibitor 610 $aproton pump inhibitor 610 $aproton-pump inhibitors 610 $arenal cell carcinoma 610 $arenal transplant 610 $arenal transplant recipients 610 $arenal transplantation 610 $arenal transplantation. 610 $asingle nucleotide polymorphisms 610 $asubarachnoid hemorrhage 610 $asulfate-reducing bacteria 610 $asurgical complications 610 $asurvival 610 $asystematic reviews 610 $atacrolimus 610 $atacrolimus metabolism 610 $atemperature 610 $atiming of removal 610 $atransplant 610 $atransplant recipients 610 $atransplant rejection 610 $atransplantation 610 $atryptophan 610 $aUPEC 610 $aureteric stent 610 $aurinary biomarkers 610 $aurinary excretion 610 $aurinary excretion of N1-methylnicotinamide 610 $aurinary tract infection 610 $aurological complications 610 $aUropathogenic E. coli 610 $avirulence traits 610 $avitamin B3 610 $avitamin C 610 $a?-GST 610 $a?-GST 615 7$aMedicine 700 $aCheungpasitporn$b Wisit$4edt$01277893 702 $aThongprayoon$b Charat$4edt 702 $aLeeaphorn$b Napat$4edt 702 $aCheungpasitporn$b Wisit$4oth 702 $aThongprayoon$b Charat$4oth 702 $aLeeaphorn$b Napat$4oth 906 $aBOOK 912 $a9910557787303321 996 $aRecent Advances and Clinical Outcomes of Kidney Transplantation$93012222 997 $aUNINA LEADER 04515nam 2200481z- 450 001 9910557662003321 005 20211118 035 $a(CKB)5400000000044886 035 $a(oapen)https://directory.doabooks.org/handle/20.500.12854/73704 035 $a(oapen)doab73704 035 $a(EXLCZ)995400000000044886 100 $a20202111d2020 |y 0 101 0 $aeng 135 $aurmn|---annan 181 $ctxt$2rdacontent 182 $cc$2rdamedia 183 $acr$2rdacarrier 200 00$aShaping the Brain by Neuronal Cytoskeleton: From Development to Disease and Regeneration 210 $cFrontiers Media SA$d2020 215 $a1 online resource (185 p.) 311 08$a2-88963-552-X 330 $aThe coordinated action of the different cytoskeletal polymers--microtubules, actin filaments and neurofilaments-- is essential for the establishment, remodeling and maintenance of neuronal architecture throughout the neuron lifetime. Neurons are among the most polarized cells, with a long thin axon and multiple thicker and shorter dendrites. Achieving this complex morphology, and the precise and accurate formation of an intricate network of synaptic contacts is critical for the proper transmission and reception of signals in the brain. Neuronal polarization precedes axon outgrowth and the subsequent differentiation of short neurites into dendrites, as part of the neuronal differentiation program that involves both intrinsic and extrinsic signals that converge at the cytoskeletal level. Growth cones, which are sensory and locomotor structures located at the tip of growing axons, are key elements in the transduction of extracellular cues into cytoskeletal changes, guiding axons to their right destinations. Neuronal migration, another crucial process during brain development, occurs in close coordination with neuronal differentiation. Migration involves as well an extensive rearrangement of neuronal cell shape that relies on cytoskeleton reorganization. Further processes, such as dendritic spine formation and growth, establishment of synaptic contacts or synaptic plasticity in mature neurons also depend on cytoskeletal dynamics. Fine-tune regulation of neuronal cytoskeleton is therefore crucial for the maintenance of neuronal integrity and functionality. Mutations in genes that code for cytoskeletal proteins often have deleterious effects in neurons, such as abnormal migration or differentiation, deficient axonal transport of organelles and synaptic vesicles, or impaired synaptic signaling. Several human Nervous System disorders, including neurodevelopmental, psychiatric, and neurodegenerative diseases, have been linked to cytoskeletal dysfunction. Cytoskeletal reorganization is also crucial to regulate nerve cell repair following Nervous System injury. Many of the pathways that control cell-intrinsic axon regeneration lead to axon cytoskeletal remodeling. Moreover, most extracellular cues that inhibit regeneration of damaged axons in Central Nervous System following traumatic injury or neurodegeneration, are known to modulate cytoskeletal dynamics and organization. Based on these findings, regulators of cytoskeleton dynamics have emerged as promising therapeutic targets in several brain disorders and in the context of regeneration of injured axons. Hence, remodeling of neuronal cytoskeleton underlies all the dramatic morphological changes that occur in developing and adult neurons. Understanding the specific molecular mechanisms that control cytoskeleton rearrangements in neurons is far from complete. This Frontiers Research Topic gathers a selection of articles focused on the diverse and key roles of cytoskeleton in neuronal biology. 517 $aShaping the Brain by Neuronal Cytoskeleton 606 $aNeurosciences$2bicssc 606 $aScience: general issues$2bicssc 610 $aactin cytoskeleton 610 $aastrocyte cytoskeleton 610 $amicrotubules (MTs) 610 $aneuron 610 $aneuronal cytoskeleton 610 $atau 615 7$aNeurosciences 615 7$aScience: general issues 700 $aLaura Sayas$b C$4edt$01302165 702 $aMendes Sousa$b Monica$4edt 702 $aAvila$b Jesus$4edt 702 $aLaura Sayas$b C$4oth 702 $aMendes Sousa$b Monica$4oth 702 $aAvila$b Jesus$4oth 906 $aBOOK 912 $a9910557662003321 996 $aShaping the Brain by Neuronal Cytoskeleton: From Development to Disease and Regeneration$93026187 997 $aUNINA