LEADER 00977nam0-2200325---450- 001 990009101140403321 005 20100719095156.0 010 $a0803980582 035 $a000910114 035 $aFED01000910114 035 $a(Aleph)000910114FED01 035 $a000910114 100 $a20091009d1988----km-y0itay50------ba 101 0 $aeng 102 $aGB 105 $a--------001cy 200 1 $aJournalist at war$ethe dynamic of news reporting during the Falkland conflict$fby David E. Morrison and Howard Tumber 210 $aOxford$cSage Publication$d1988 215 $a370 p.$ctab.$d22 cm 610 0 $aGiornalismo$aStati Uniti 676 $a071.3$v22$zita 700 1$aMorrison,$bDavid E.$g$0320631 701 1$aTumber,$bHoward$f<1951- >$0320629 801 0$aIT$bUNINA$gRICA$2UNIMARC 901 $aBK 912 $a990009101140403321 952 $a071.3 MOR 1$b1899$fBFS 959 $aBFS 996 $aJournalist at war$9781868 997 $aUNINA LEADER 11170nam 2200565 450 001 9910502621703321 005 20220921165657.0 010 $a3-030-83078-0 035 $a(CKB)4100000012038117 035 $a(MiAaPQ)EBC6738522 035 $a(Au-PeEL)EBL6738522 035 $a(OCoLC)1273075595 035 $a(PPN)258058358 035 $a(EXLCZ)994100000012038117 100 $a20220627d2021 uy 0 101 0 $aeng 135 $aurcnu|||||||| 181 $ctxt$2rdacontent 182 $cc$2rdamedia 183 $acr$2rdacarrier 200 10$aFood addiction, obesity, and disorders of overeating $ean evidence-based assessment and clinical guide /$fClaire E. Wilcox 210 1$aCham, Switzerland :$cSpringer,$d[2021] 210 4$d©2021 215 $a1 online resource (224 pages) 311 $a3-030-83077-2 320 $aIncludes bibliographical references and index. 327 $aIntro -- Introduction: Obesity, Eating Disorders and Food Addiction: Towards a Synthesis -- References -- Contents -- Contributors -- Part I: Standard Approaches to Clinical Assessment and Treatment of Obesity and Binge Eating Disorder (BED) -- 1: Determinants of Body Weight: Metabolism and the Homeostatic System -- 1.1 Energy Balance -- 1.2 Metabolism -- 1.3 Why Weight Gain Is So Easy and Weight Loss So Hard -- 1.4 Homeostatic Feeding Mechanisms -- 1.4.1 The Hypothalamus -- 1.4.2 Key Neuropeptides -- 1.4.3 Other Factors -- 1.5 Neuropeptides, Obesity, and Disordered Eating -- 1.6 Conclusion -- References -- 2: Obesity -- 2.1 Definition of Obesity -- 2.2 Epidemiology -- 2.3 Assessment -- 2.4 Causes, Contributors, and Risk Factors -- 2.5 Genetics -- 2.6 Management -- 2.6.1 Overview of Treatment -- 2.6.2 Comprehensive Models and Behavioral Weight Loss Therapy -- 2.6.3 Dietary Component -- 2.6.4 Physical Activity Component -- 2.6.5 Behavioral Modification Component -- 2.6.6 Pharmacotherapy -- 2.6.7 Dietary Supplements and Procedures to Avoid -- 2.6.8 Bariatric Surgery -- 2.7 Conclusion -- References -- 3: Binge-Related Eating Disorders (Binge Eating Disorder and Bulimia Nervosa) -- 3.1 Epidemiology -- 3.2 Diagnosis and Assessment of BED -- 3.3 Epidemiology of BN -- 3.4 Diagnosis and Assessment of BN -- 3.5 Etiology and Mechanisms of BED and BN -- 3.6 Treatment of BED: General Considerations -- 3.7 Psychotherapies for BED -- 3.8 Pharmacotherapy for BED -- 3.9 Nutritional Recommendations for BED -- 3.10 Best Practices and Guidelines for BED Treatment -- 3.11 Obesity and BED Treatment -- 3.12 Treatment of BN -- 3.13 Treatments for both BED and BN -- 3.14 Conclusion -- References -- Part II: Can the Food Addiction Concept Improve Treatment? -- 4: Problems with Current Approaches to Treating Disorders of Overeating. 327 $a4.1 Weight Loss with Available Treatments Is Modest -- 4.2 Maintenance of Weight Loss Is Difficult -- 4.3 The Biology Behind the Difficulty of Weight Loss and Maintenance -- 4.4 Eating Disorder (ED) Treatment Success Rates -- 4.5 ED and Obesity Treatments Give Conflicting Messages -- 4.6 Side Effects of Diets -- 4.7 Limitations and Side Effects of Medications -- 4.8 Side Effects of Bariatric Surgery -- 4.9 Difficulty of Exercising When Obese -- 4.10 Conclusion -- References -- 5: The Food Addiction Concept: History, Controversy, Potential Pitfalls, and Promises -- 5.1 History -- 5.2 Is the FA Concept Valid? -- 5.2.1 Can Obesity Be Explained by FA? -- 5.2.2 Is FA Distinct from BED and Bulimia? -- 5.2.3 Do DSM Criteria for SUD Present in Relation to Food in Humans and Do Symptoms Cluster Together? -- 5.2.4 Is It Valid to Claim Certain Foods Are "Addictive," and Might It Be More Accurate to Consider FA a Behavioral Disorder? -- 5.3 Is the FA Model Useful, and Do Benefits Outweigh Harms? -- 5.3.1 Abstinence-Based Food Plans -- 5.3.2 Self-Efficacy -- 5.3.3 Public Health -- 5.3.4 Stigma -- 5.4 Potential Promises for the Future -- 5.5 Increasing Community Acceptance -- 5.6 Conclusion -- References -- Part III: Clinical Evidence for Food Addiction -- 6: Clinical Evidence for the Validity of Food Addiction -- 6.1 Shared DSM Criteria -- 6.2 Yale Food Addiction Scale Development -- 6.3 SUD and Disordered Eating Co-occur -- 6.4 Sweet Preference, Addiction Transfer, and Cross-Sensitization -- 6.5 Overlapping Neuropsychological, Emotional, and Personality Traits, Psychiatric Diagnoses, and Predisposing Conditions (Trauma and Stress) -- 6.6 Conclusion -- References -- Part IV: Basic Biology of Food Addiction, and Its Overlap with Substance Use Disorders -- 7: Neurobiology and Cognitive Neuroscience of Substance Use Disorders. 327 $a7.1 Overview of Substance Use Disorders -- 7.2 Core Brain Regions -- 7.3 Reward -- 7.4 Conditioning: Positive Reinforcement -- 7.5 Motivation: Positive Reinforcement -- 7.6 Tolerance: Downregulation of Dopamine and Opioid System -- 7.7 Withdrawal and Hyperkatifeia -- 7.8 Conditioning and Motivation: Negative Reinforcement -- 7.9 Impulsivity and Executive Function Deficits -- 7.10 Benefits of Understanding the Neurobiology -- 7.11 Conclusion -- References -- 8: Neurobiology and Cognitive Neuroscience of Hedonic Eating -- 8.1 Reward and Hedonic Liking -- 8.2 Conditioning: Positive Reinforcement -- 8.3 Motivation: Positive Reinforcement -- 8.4 Food Reward, Conditioning, and Reward Motivation: Additional Factors -- 8.5 Tolerance and Downregulation of DA and Opioid Systems -- 8.6 Withdrawal -- 8.7 Conditioning and Motivation: Negative Reinforcement -- 8.8 Impulse Control and Executive Function Deficits -- 8.9 Conclusion -- References -- 9: Additional Biological Mechanisms of Hedonic Eating -- 9.1 Interactions Between the Homeostatic System and Hedonic System -- 9.1.1 Anatomy -- 9.1.2 Appetite-Regulating Neuropeptides Modulate Hedonic Eating -- 9.1.3 Appetite-Regulating Neuropeptides Moderate Drug and Alcohol Use -- 9.2 Stress, Hedonic Eating, and the Reward System -- 9.2.1 The Anatomy of the Stress Response -- 9.2.2 Acute and Chronic Stress Promote Hedonic Eating -- 9.3 Genetics -- 9.4 In Utero Exposure -- 9.5 Neuroinflammation -- 9.6 Oxidative Stress -- 9.7 Gut Microbiome and Gut-Brain Axis -- 9.8 Adrenergic System -- 9.9 Sleep and Circadian Rhythm -- 9.10 Serotonin System -- 9.11 Endocannabinoid System -- 9.12 Functional Connectivity -- 9.13 Conclusions -- References -- 10: Treatment-Related Evidence that Food Addiction Is a Valid Construct -- 10.1 Pharmacotherapy-Related Evidence -- 10.1.1 Stimulants. 327 $a10.1.2 Opioid Antagonists -- 10.1.3 Topiramate and Zonisamide -- 10.1.4 GLP-1 Agonists -- 10.1.5 Other Medications to Note -- 10.2 Bariatric Surgery -- 10.3 Conclusion -- References -- 11: Highly Palatable Foods Are Addictive -- 11.1 Problematic and "Addictive" Foods -- 11.2 Association Between HP Food Intake and Addiction in Animal Models -- 11.3 Association Between HP Food Intake and Weight Gain/Disordered Eating in Humans -- 11.4 Why Are HP Foods More Associated with Addictive Eating Patterns? -- 11.4.1 Innate Preferences -- 11.4.2 Conditioning from Rapid Post-oral Glucose Rise -- 11.4.3 Stimulation of Reward System by HP Foods -- 11.4.4 Effects on Inflammatory Processes, Oxidative Stress and Gut Microbiome -- 11.4.5 Cessation Leads to Withdrawal -- 11.4.6 Adverse Effects on Mood and Anxiety -- 11.4.7 Reduction in Executive Function -- 11.4.8 Reduction in Satiety Due to Changes in Homeostatic Feeding -- 11.4.9 Individual Variability -- 11.4.10 Feeding Patterns Influence Food Addiction -- 11.5 State Effects of Hunger/Food Restriction on Reward Circuitry and Brain Function -- 11.6 Artificial Sweeteners and Sugar Substitutes -- 11.7 What Should Be Considered Addictive Food? -- 11.8 Conclusion -- References -- Part V: Assessment and Treatment of Food Addiction -- 12: Evaluation of Food Addiction: Importance, Epidemiology, Diagnosis, and Assessment -- 12.1 Importance of Assessing for Food Addiction -- 12.2 Epidemiology of FA -- 12.3 YFAS: Scoring and Interpretation -- 12.4 Other Important Assessment Considerations and Common Comorbidities -- 12.4.1 SUD -- 12.4.2 ED History -- 12.4.3 Obesity History and Related Health Concerns -- 12.4.4 Psychiatric Comorbidity -- 12.5 Conclusion -- References -- 13: How to Treat Food Addiction from a Nutritional Perspective: Consideration of Diet and Abstinence. 327 $a13.1 Nutritional Approaches and Consideration of Abstinence -- 13.2 Related Tips -- 13.2.1 Increase Satiety and Brain Health-Promoting Foods -- 13.2.2 Do Not Over-restrict Calorie Intake -- 13.2.3 Realize that Craving Will Diminish with Time in Recovery -- 13.2.4 Abstinence Is Not Absolute: Avoid All-or-Nothing Thinking -- 13.2.5 Is It Better to Start More Extreme or Use a Graded Approach During Initiation? -- 13.2.6 Track Progress -- 13.3 How to Incorporate FA Treatment into ED Treatment Programming -- 13.4 What to Do with "Normal Weight" FA Patients? -- 13.5 Conclusion -- References -- 14: Clinical Applications of the Food Addiction Concept -- 14.1 Treatment Overview -- 14.2 Supplemental Programmatic Elements Which Might Be Useful for Treatment of FA -- 14.2.1 Psychoeducation: FA Is a Brain-Based Disorder -- 14.2.2 Psychosocial Interventions -- 14.2.2.1 CBT -- 14.2.2.2 Behavioral Weight Management -- 14.2.2.3 Motivational Interviewing (MI) -- 14.2.2.4 Psychotherapy to Reduce Negative Affect States, Improve Emotion Regulation, and Address Alexithymia -- 14.2.2.5 Mindfulness-Based (MB) Approaches -- 14.2.2.6 Body Image Work -- 14.2.3 Importance of Sleep -- 14.2.4 Importance of Exercise -- 14.2.5 Importance of Getting Psychiatric and Psychological Care -- 14.2.6 Neuromodulation Techniques -- 14.2.7 12-Step Programs and Other Support -- 14.2.8 Medications -- 14.2.9 Bariatric Surgery -- 14.3 Subtyping and FA Treatment Matching -- 14.3.1 Within-FA Treatment Matching -- 14.3.2 Using FA as a Treatment Matching Variable for Patients with Obesity and BE -- 14.4 Conclusion -- References -- Part VI: Research Possibilities -- 15: Emerging Treatments and Areas for Future Research -- 15.1 Emerging Treatments for Disordered Eating -- 15.1.1 Neurostimulation -- 15.1.2 Real-Time fMRI (Rt-fMRI) Neurofeedback Training. 327 $a15.1.3 Cognitive Training. 606 $aCompulsive eating 606 $aTrastorns de la conducta alimentària$2thub 606 $aTrastorns de la gana$2thub 606 $aObesitat$2thub 608 $aLlibres electrònics$2thub 615 0$aCompulsive eating. 615 7$aTrastorns de la conducta alimentària 615 7$aTrastorns de la gana 615 7$aObesitat 676 $a616.8526 700 $aWilcox$b Claire E.$01072709 801 0$bMiAaPQ 801 1$bMiAaPQ 801 2$bMiAaPQ 906 $aBOOK 912 $a9910502621703321 996 $aFood Addiction, Obesity, and Disorders of Overeating$92568950 997 $aUNINA LEADER 01243nam a2200241 i 4500 001 991001466699707536 008 s2005 it 000 0 ita d 020 $a8871376862 035 $ab13376937-39ule_inst 040 $aDip. SSC$bita 082 0 $a344.0224026 100 1 $aBertozzi, Paride$0231955 245 12$a I diritti dei genitori lavoratori :$bla tutela della maternità e della paternità : testo unico delle disposizioni legislative in materia di tutela e sostegno della maternità e della paternità : guida ragionata e commentata alla lettura del decreto legislativo 26 marzo 2001, n. 151 riveduta ed aggiornata con le novità introdotte dal D.Lgs. 23 aprile 2003, n. 115, le più recenti pronunce giurisprudenziali e le più importanti circolari ministeriali /$cParide Bertozzi 250 $a2. ed. 260 $aRoma :$bCieRre,$c2005 300 $a214 p. ;$c21 cm 650 4$aDiritto del lavoro 907 $a.b13376937$b02-04-14$c07-02-06 912 $a991001466699707536 945 $aLE021 DI9D81$g1$i2021000121043$lle021$nBiblioteca$o-$pE0.00$q-$rl$s- $t0$u3$v1$w3$x0$y.i14189707$z07-02-06 996 $aDiritti dei genitori lavoratori$91094389 997 $aUNISALENTO 998 $ale021$b07-02-06$cm$d- $e-$fita$git $h2$i0 LEADER 04058 am 2200577 n 450 001 9910256647003321 005 20171130 010 $a2-87854-900-7 035 $a(CKB)4100000001948665 035 $a(FrMaCLE)OB-psn-5338 035 $a(oapen)https://directory.doabooks.org/handle/20.500.12854/44769 035 $a(PPN)225228149 035 $a(EXLCZ)994100000001948665 100 $a20180202j|||||||| ||| 0 101 0 $afre 135 $auu||||||m|||| 181 $ctxt$2rdacontent 182 $cc$2rdamedia 183 $acr$2rdacarrier 200 13$aLa déréglementation des économies anglo-saxonnes $eBilan et perspectives /$fMartine Azuelos 210 $aParis $cPresses Sorbonne Nouvelle$d2017 215 $a1 online resource (160 p.) 311 $a2-87854-076-X 330 $aLes économies anglo-saxonnes ont vécu, entre le milieu des années 1970 et la fin des années 1980, sous le signe de la déréglementation, et le mouvement qu'elles impulsèrent ne tarda pas à se propager à l'ensemble du monde, transformant ainsi de façon radicale les règles du jeu auxquelles sont soumis les agents économiques. Souvent dû à des facteurs techniques, ce processus fut également encouragé par les pouvoirs publics qui, face à la crise, ne voyaient d'autre salut que la mise en cause de pratiques qui avaient fait l'objet d'un assez large consensus pendant les années de croissance consécutives à la seconde guerre mondiale. Longtemps conçu comme un ensemble de garde-fous destiné à limiter les abus et à protéger les agents, le cadre réglementaire n'apparaissait plus guère que comme un carcan entravant le libre jeu des forces du marché, nuisant à l'efficacité économique et expliquant donc le ralentissement de la croissance. Cet ouvrage, qui rassemble les actes d'un colloque organisé en juin 1993 à l'Université de la Sorbonne Nouvelle par le Centre d'études et de recherches sur la vie économique des pays anglo-saxons (CERVEPAS), ne prétend pas épuiser le sujet. Il enrichit toutefois notre réflexion en proposant une étude comparée des évolutions qui se sont produites en Grande-Bretagne et aux États-Unis, sur lesquelles universitaires français et acteurs ou observateurs privilégiés venus d'outre-Manche ou d'outre-Atlantique portent des regards croisés. Situant la déréglementation dans la perspective de l'histoire des faits et de la pensée économiques, il analyse aussi les mesures mises en ?uvre dans des secteurs comme la défense et l'audiovisuel, ou encore dans le domaine bancaire et financier. Se trouve ainsi mise en lumière la très grande variété des mécanismes en jeu. dont ne saurait rendre compte un chapelet d'analyses monocausales. À l'épaisseur des faits se devait en effet de répondre la diversité des angles d'approche. 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