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010   $a3-319-58106-6
024 7 $a10.1007/978-3-319-58106-4
035   $a(CKB)3710000001631524
035   $a(MiAaPQ)EBC4938536
035   $a(DE-He213)978-3-319-58106-4
035   $a(PPN)203852176
035   $a(EXLCZ)993710000001631524
100   $a20170803d2017      u|             0
101 0 $aeng
135   $aurcnu||||||||
181   $2rdacontent
182   $2rdamedia
183   $2rdacarrier
200 10$aMolecules, Systems and Signaling in Liver Injury$b[electronic resource] /$fedited by Wen-Xing Ding, Xiao-Ming Yin
205   $a1st ed. 2017.
210  1$aCham :$cSpringer International Publishing :$cImprint: Springer,$d2017.
215   $a1 online resource (290 pages) $cillustrations (some color)
225 1 $aCell Death in Biology and Diseases,$x2625-2902
311   $a3-319-58105-8 
320   $aIncludes bibliographical references at the end of each chapters and index.
330   $aThis essential volume presents comprehensive information on cell death and autophagy in liver diseases, including the role and molecular signaling pathways of cell death in alcohol and non-alcoholic fatty liver disease, bile acids, hepatitis C virus and drug-induced liver injury. The book starts with a discussion of lipotoxicity in non-parenchymal cells, followed by a discussion of cell death and autophagy in cholangiocytes, hepatic stellate cells and Kupffer cells in hepatic biliary diseases, fibrosis and liver inflammation. The book also covers Bcl-2 family proteins, beta-catenin and HMGB1 signaling in regulating cell death in the liver as well as mitochondria, ER stress and gut microbiota on liver injury. The Cell Death in Biology and Diseases series has recruited world experts ranging from basic scientists to clinicians on cell death in liver diseases. Likewise the contributors of this volume are leaders in their fields with worldwide expertise and perspective. Molecules, Systems and Signaling in Liver Injury is an essential companion to Hepatocytes and Non-Parenchymal Cells and Diseases. It is beneficial for both clinicians and basic scientists and is relevant to those working on drug discovery for preventing and treating liver diseases by targeting cell death and autophagy pathways.
410  0$aCell Death in Biology and Diseases,$x2625-2902
606   $aCell cycle
606   $aApoptosis
606   $aCell physiology
606   $aHepatology
606   $aCell membranes 
606   $aCell Cycle Analysis$3https://scigraph.springernature.com/ontologies/product-market-codes/L16030
606   $aApoptosis$3https://scigraph.springernature.com/ontologies/product-market-codes/L16060
606   $aCell Physiology$3https://scigraph.springernature.com/ontologies/product-market-codes/L33010
606   $aHepatology$3https://scigraph.springernature.com/ontologies/product-market-codes/H33088
606   $aMembrane Biology$3https://scigraph.springernature.com/ontologies/product-market-codes/L16050
615  0$aCell cycle.
615  0$aApoptosis.
615  0$aCell physiology.
615  0$aHepatology.
615  0$aCell membranes .
615 14$aCell Cycle Analysis.
615 24$aApoptosis.
615 24$aCell Physiology.
615 24$aHepatology.
615 24$aMembrane Biology.
676   $a617.1
702   $aDing$b Wen-Xing$4edt$4http://id.loc.gov/vocabulary/relators/edt
702   $aYin$b Xiao-Ming$4edt$4http://id.loc.gov/vocabulary/relators/edt
906   $aBOOK
912   $a9910253941703321
996   $aMolecules, Systems and Signaling in Liver Injury$92019295
997   $aUNINA