LEADER 02958nam 2200517 450 001 9910164016103321 005 20170223114314.0 010 $a1-284-09071-X 035 $a(CKB)3710000001056378 035 $a(MiAaPQ)EBC4441423 035 $a(CaSebORM)9781284090703 035 $a(EXLCZ)993710000001056378 100 $a20170403h20162016 uy 0 101 0 $aeng 135 $aurcnu|||||||| 181 $2rdacontent 182 $2rdamedia 183 $2rdacarrier 200 10$aAuditing IT infrastructures for compliance /$fMarty M. Weiss and Michael G. Solomon 205 $aSecond edition. 210 1$aBurlington, Massachusetts :$cJones & Bartlett Learning,$d2016. 210 4$dİ2016 215 $a1 online resource (458 pages) $cillustrations 225 1 $aJones & Bartlett Learning Information Systems Security & Assurance Series 311 $a1-284-09070-1 320 $aIncludes bibliographical references and index. 327 $aThe need for information systems security compliance -- Overview of U.S. compliance laws -- What is the scope of an IT compliance audit? -- Auditing standards and frameworks -- Planning an IT infrastructure audit for compliance -- Conducting an IT infrastructure audit for compliance -- Writing the IT infrastructure audit report -- Compliance within the user domain -- Compliance within the workstation domain -- Compliance within the LAN domain -- Compliance within the LAN-to-WAN domain -- Compliance within the WAn domain -- Compliance within the remote access domain -- Compliance within the system/application domain -- Ethics, education, and certification for IT auditors. 330 $aThe Second Edition of Auditing IT Infrastructures for Compliance provides a unique, in-depth look at recent U.S. based Information systems and IT infrastructures compliance laws in both the public and private sector. Written by industry experts, this book provides a comprehensive explanation of how to audit IT infrastructures for compliance based on the laws and the need to protect and secure business and consumer privacy data. Using examples and exercises, this book incorporates hands-on activities to prepare readers to skillfully complete IT compliance auditing. 410 0$aJones & Bartlett Learning information systems security & assurance series. 517 3 $aAuditing Information Technology infrastructures for compliance 606 $aComputer security 606 $aComputer networks$xSecurity measures 606 $aCompliance auditing 615 0$aComputer security. 615 0$aComputer networks$xSecurity measures. 615 0$aCompliance auditing. 676 $a005.8 700 $aWeiss$b Martin$g(Martin M.),$01245617 702 $aSolomon$b Michael G. 801 0$bMiAaPQ 801 1$bMiAaPQ 801 2$bMiAaPQ 906 $aBOOK 912 $a9910164016103321 996 $aAuditing IT infrastructures for compliance$92888811 997 $aUNINA LEADER 04218nam 2200469z- 450 001 9910227350203321 005 20210211 035 $a(CKB)4100000000883831 035 $a(oapen)https://directory.doabooks.org/handle/20.500.12854/53604 035 $a(oapen)doab53604 035 $a(EXLCZ)994100000000883831 100 $a20202102d2017 |y 0 101 0 $aeng 135 $aurmn|---annan 181 $ctxt$2rdacontent 182 $cc$2rdamedia 183 $acr$2rdacarrier 200 00$aMitochondria: Hubs of Cellular Signaling, Energetics and Redox Balance 210 $cFrontiers Media SA$d2017 215 $a1 online resource (228 p.) 225 1 $aFrontiers Research Topics 311 08$a2-88945-239-5 330 $aPoised at the convergence of most catabolic and anabolic pathways, mitochondria are the center of heterotrophic aerobic life, representing a hub in the overall metabolic network of cells. The energetic functions performed by mitochondria face the unavoidable redox hurdle of handling huge amounts of oxygen while keeping its own as well as the cellular redox environment under control. Reactive oxygen species (ROS) are produced in the respiratory chain as a result of the energy supplying function of mitochondria. Originally considered an unavoidable by-product of oxidative phosphorylation, ROS have become crucial signaling molecules when their levels are kept within physiological range. This occurs when their production and scavenging are balanced within mitochondria and cells. Mitochondria-generated hydrogen peroxide can act as a signaling molecule within mitochondria or in the cytoplasm, affecting multiple networks that control, for example, cell cycle, stress response, cell migration and adhesion, energy metabolism, redox balance, cell contraction, and ion channels. However, under pathophysiological conditions, excessive ROS levels can happen due to either overproduction, overwhelming of antioxidant defenses, or both. Under oxidative stress, detrimental effects of ROS include oxidation of protein, lipids, and nucleic acids; mitochondrial depolarization and calcium overload; and cell-wide oscillations mediated by ROS-induced ROS release mechanisms. Mitochondrial dysfunction is central in the pathogenesis of numerous human maladies including cardiomyopathies and neurodegeneration. Diseases characterized by altered nutrient metabolism, such as diabetes and cancer, exhibit elevated ROS levels. These may contribute to pathogenesis by increasing DNA mutation, affecting regulatory signaling and transcription, and promoting inflammation. Under metabolic stress, several ionic channels present in the inner and outer mitochondrial membranes can have pro-life and -death effects. In the present E-book, based on the Frontiers Research Topic entitled: "Mitochondria: Hubs of cellular signaling, energetics and redox balance", we address one of the fundamental questions that the field of ROS biology faces today: how do mitochondria accomplish a reliable energy provision and at the same time keep ROS levels within physiological, non-harming, limits but crucial for cellular signaling function? Additionally, and within the perspective of mitochondria as signaling-energetic hubs in the extensive cellular metabolic network, we ask how can their collective dynamics scale from the subcellular to the cellular, tissue and organ levels to affect function in health and disease. 517 $aMitochondria 606 $aPhysiology$2bicssc 610 $ahypertrophic and diabetic cardiomyopathies 610 $aketone bodies 610 $alight- and anesthetics-induced cardioprotection 610 $alipid catabolism 610 $anecroptosis 610 $apost-translational modifications 610 $aredox aging 610 $aredox and energetic compartmentation 610 $aredox metabolism and signaling 610 $askeletal-cardiac muscle and brain protection 615 7$aPhysiology 700 $aAmadou K. S. Camara$4auth$01304390 702 $aMiguel A. Aon$4auth 906 $aBOOK 912 $a9910227350203321 996 $aMitochondria: Hubs of Cellular Signaling, Energetics and Redox Balance$93027354 997 $aUNINA