LEADER 03039nam 22005295 450 001 9910300303503321 005 20200702164606.0 010 $a4-431-56541-8 024 7 $a10.1007/978-4-431-56541-3 035 $a(CKB)4100000002485514 035 $a(MiAaPQ)EBC5311245 035 $a(DE-He213)978-4-431-56541-3 035 $a(PPN)224636928 035 $a(EXLCZ)994100000002485514 100 $a20180223d2018 u| 0 101 0 $aeng 135 $aurcnu|||||||| 181 $2rdacontent 182 $2rdamedia 183 $2rdacarrier 200 10$aPathogenesis of Idiopathic Scoliosis /$fedited by Masafumi Machida, Stuart L. Weinstein, Jean Dubousset 205 $a1st ed. 2018. 210 1$aTokyo :$cSpringer Japan :$cImprint: Springer,$d2018. 215 $a1 online resource (188 pages) $cillustrations (some color), photographs, tables 311 $a4-431-56539-6 320 $aIncludes bibliographical references at the end of each chapters. 330 $aThis book provides comprehensive coverage of current topics in idiopathic scoliosis (IS). A three-dimensional deformity of the spine, the condition is characterized by lateral curvature combined with vertebral rotation. The primary lesion, however, lies in the median sagittal plane, taking the form of a lordosis. Although the clinical manifestations of scoliosis have been well documented, its cause and pathogenesis have not yet been determined. Research into what causes IS has focused on the structural elements of the spine, spinal musculature, collagenous structures, the endocrine system, the central nervous system, and genetics. Results of these studies have brought about a new perception of IS epiphenomena, but the main cause of IS remains unknown. Recently, several investigators have produced new hypotheses regarding the cause of IS using the developing techniques of genetics, biochemistry, and neurology. 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