03958nam 2200961z- 450 991056646570332120220506(CKB)5680000000037723(oapen)https://directory.doabooks.org/handle/20.500.12854/81019(oapen)doab81019(EXLCZ)99568000000003772320202205d2022 |y 0engurmn|---annantxtrdacontentcrdamediacrrdacarrierRedox Imbalance and Mitochondrial Abnormalities in Kidney DiseaseBaselMDPI - Multidisciplinary Digital Publishing Institute20221 online resource (200 p.)3-0365-3757-0 3-0365-3758-9 The kidney performs important functions in the human body and can inflict either acute kidney injury (AKI) or chronic kidney disease (CKD). AKI can be induced by kidney ischemia, drugs such as cisplatin, and heavy metals such as cadmium and arsenic. CKD can be induced by drugs, heavy metals, hypertension, and diabetes, as well as cancer. Importantly, nearly all kidney disorders have been shown to involve redox imbalance, reductive stress, oxidative stress, and mitochondrial abnormalities such as impaired mitochondrial homeostasis, including disrupted mitophagy and deranged mitochondrial unfolded protein responses. Understanding how these redox-related dysregulated pathways operate may give us new insights into how to design novel approaches to fighting kidney disease. This Special Issue of Biomolecules entitled "Redox imbalance and mitochondrial abnormalities in kidney disease" covers a variety of topics focusing on oxidative stress, mitochondrial dysfunction, and antioxidation enhancement implicated in kidney disease or kidney transplantation.Medicine and NursingbicsscPharmacologybicsscacute kidney injury (AKI)antioxidant defensebiogenesiscadmiumcaloric restrictioncell deathchronic kidney diseasechronic kidney disease (CKD)controlled oxygenated rewarmingdiabetesdiabetic kidney diseasefatty acid (FA) β-oxidationischemiakidneykidney allograftkidney diseaseskidney injurykidney rejectionmajor adverse cardiovascular outcomesmitochondriamitochondrial dynamicsmitochondrial dysfunctionmitochondrial homeostasismitochondrial metabolismmitochondrial oxidative stressmitochondrial proteinsmitochondrial reactive oxygen speciesmitochondrial redox signalingmitochondrial uncouplingmitophagyn/aNADH/NAD+Oryza sativaoxidative damageoxidative phosphorylation (OXPHOS)oxidative stresspodocytopathiesproximal tubulereactive oxygen species (ROS)redoxredox imbalancerenal toxicityrenalaserewarming injuryrice huskSGLT2TCA cycle metabolitestemperature paradoxtricarboxylic acid (TCA) cycleWarburg effectMedicine and NursingPharmacologyYan Liang-Junedt1314102Yan Liang-JunothBOOK9910566465703321Redox Imbalance and Mitochondrial Abnormalities in Kidney Disease3031708UNINA01664nam0 22003613i 450 CFI019003220251003044131.0881316886120071031d1990 ||||0itac50 baitaitz01i xxxe z01nz01ncRDAcarrierContributo alla teoria della funzione di produzioneD. Cantarelli, G. Marangoni, L. CupelloPadovaCEDAM1990XV, 236 p.24 Ricerca finanziata con il contributo del M.P.I. e del C.N.R.ProduzioneModelli matematiciFIRSBLC028996I338.0015118Produzione. Modelli matematici20Cantarelli, DavideCFIV088739070103463Marangoni, GiandemetrioCFIV008039070120442Cupello, LauraCFIV104263070374917Marangoni, G. D.CFIV158665Marangoni, GiandemetrioMarangoni, G. D.CFIV158665Marangoni, GiandemetrioITIT-00000020071031IT-BN0095 NAP 01POZZO LIB.Vi sono collocati fondi di economia, periodici di ingegneria e scienze, periodici di economia e statistica e altri fondi comprendenti documenti di economia pervenuti in dono. CFI0190032Biblioteca Centralizzata di Ateneo1 v.1 v. 01POZZO LIB.ECON MON 8929 0101 0000089815E VMA 1 v. (Precedente collocazione: GUERRAZZI B 448)B 2007103120071031 01Contributo alla teoria della funzione di produzione62924UNISANNIO