00917nam a2200265 i 4500991002361819707536070418r20041993it a 001 0 mul 8802047529b13515639-39ule_instDip.to Studi GiuridiciitaitalatTerentius Afer, Publius439355Commedie /di Publio Terenzio Afro ; a cura di Orazio BiancoRist.Torino :UTET,c2004906 p. ;[8] c. di tav. :ill. ;24 cm.Classici latiniTesto latino a fronteBianco, Orazio.b1351563919-04-0719-04-07991002361819707536LE027 R-XXVI/UTET Terentius12027000128860le027pE54.60-no 00000.i1442860x19-04-07Commedie101247UNISALENTOle02718-04-07ma -mulit 0001913nam 2200361 450 991068846160332120230628094726.0(CKB)5400000000043316(NjHacI)995400000000043316(EXLCZ)99540000000004331620230628d2020 uy 0engur|||||||||||txtrdacontentcrdamediacrrdacarrierMitochondria and Brain Disorders /edited by Stavros J. BaloyannisLondon, England :IntechOpen,2020.1 online resource (124 pages)1-78985-653-1 The mitochondrion is a unique and ubiquitous organelle that contains its own genome, encoding essential proteins that are major components of the respiratory chain and energy production system. Mitochondria play a dominant role in the life and function of eukaryotic cells including neurons and glia, as their survival and activity depend upon mitochondrial energy production and supply. Besides energy production, mitochondria also play a vital role in calcium homeostasis and may induce apoptosis by excitotoxicity. Mitochondrial dysfunction is related to common neurological diseases, such as Parkinson's disease, Alzheimer's disease, Friedreich's ataxia, Huntington's disease, and Multiple Sclerosis. An efficient treatment of mitochondrial dysfunction would open new horizons in the therapeutic perspectives of a substantial number of inflammatory and degenerative neurological disorders.MitochondriaBrainDiseasesEtiologyMitochondria.BrainDiseasesEtiology.571.657Baloyannis Stavros J.NjHacINjHaclBOOK9910688461603321Mitochondria and Brain Disorders1944353UNINA