01913nam 2200361 450 991068842780332120230630051647.03-03842-654-710.3390/books978-3-03842-654-7(CKB)5400000000000658(NjHacI)995400000000000658(EXLCZ)99540000000000065820230630d2018 uy 0engur|||||||||||txtrdacontentcrdamediacrrdacarrierViral Infection and Apoptosis /edited by Marc KvansakulBasel, Switzerland :MDPI - Multidisciplinary Digital Publishing Institute,2018.1 online resource (270 pages) illustrationsIncludes bibliographical references.Annotation Apoptosis is a form of programmed cell death that enables the removal of damaged, infected, or otherwise unwanted cells in a controlled manner. Apoptosis can be initiated by multiple independent pathways that ultimately converge at a point where proteolytic enzymes belonging to the caspase family are activated, which dismantle the apoptotic cell. Multicellular organism have employed apoptotic mechanisms during host defence in response to viral infection to limit or prevent viral spread and replication. Consequently, viruses have evolved sophisticated molecular countermeasures to disarm host apoptotic defences, and this series of reviews and primary research articles in this Special Issue explores the intricate molecular interplay between viruses and their hosts when they battle for control of host apoptotic check-points.ApoptosisApoptosis.571.936Kvansakul MarcNjHacINjHaclBOOK9910688427803321Viral Infection and Apoptosis2929118UNINA