01900nam 2200409 450 991068338420332120230627052404.03-0365-6713-510.3390/books978-3-0365-6713-6(CKB)5700000000354388(NjHacI)995700000000354388(EXLCZ)99570000000035438820230627d2023 uy 0engur|||||||||||txtrdacontentcrdamediacrrdacarrierAmyloid-β Structure, Function, and Pathophysiological Significance in Neurodegenerative Diseases /edited by Masashi Tanaka, Kenjiro Ono, Satoshi SaitoBasel, Switzerland :MDPI - Multidisciplinary Digital Publishing Institute,2023.1 online resource (276 pages)3-0365-6712-7 This reprint includes 16 original articles published in a Special Issue that focuses on the pathological significance of amyloid-β in neurodegenerative diseases, such as Alzheimer's disease, the main cause of dementia. Collections in this reprint provide novel insights into the underlying mechanisms and therapeutic potentials of repositioning drugs and new agents for Alzheimer's disease and related dementias. These findings are expected to contribute to the development of innovative strategies for the prevention and treatment of such diseases.Alzheimer's diseasePatientsRehabilitationAlzheimer's diseasePatientsCareAlzheimer's diseasePatientsRehabilitation.Alzheimer's diseasePatientsCare.616.831Tanaka MasashiOno KenjiroSaito SatoshiNjHacINjHaclBOOK9910683384203321Amyloid-β3393001UNINA