00868nam0-22003011i-450-99000496874040332119990530000496874FED01000496874(Aleph)000496874FED0100049687419990530g19599999km-y0itay50------baitay-------001yyObras escogidasSan Juan de La Cruzedición y prologo de Ignacio B. Anzoàtegui4.a ed.MadridEspasa-Calpe1959.148 p.18 cmColección austral326Juan de la Cruz<santo ;1542-1591>200144Anzoategui,Ignazio B.ITUNINARICAUNIMARCBK990004968740403321YP 3 326Fil. Mod. 7097FLFBCFLFBCObras escogidas526003UNINA06054nam 2201537z- 450 991055771670332120220111(CKB)5400000000046158(oapen)https://directory.doabooks.org/handle/20.500.12854/76898(oapen)doab76898(EXLCZ)99540000000004615820202201d2021 |y 0engurmn|---annantxtrdacontentcrdamediacrrdacarrierNeuroprotection: Rescue from Neuronal Death in the BrainBasel, SwitzerlandMDPI - Multidisciplinary Digital Publishing Institute20211 online resource (408 p.)3-0365-1994-7 3-0365-1995-5 Dear Colleagues, The brain is vulnerable to injury. Following injury in the brain, apoptosis or necrosis may occur easily, leading to various functional disabilities. Neuronal death is associated with a number of neurological disorders including hypoxic ischemia, epileptic seizures, and neurodegenerative diseases. The brain subjected to injury is regarded to be responsible for the alterations in neurotransmission processes, resulting in functional changes. Oxidative stress produced by reactive oxygen species has been shown to be related to the death of neurons in traumatic injury, stroke, and neurodegenerative diseases. Therefore, scavenging or decreasing free radicals may be crucial for preventing neural tissues from harmful adversities in the brain. Neurotrophic factors, bioactive compounds, dietary nutrients, or cell engineering may ameliorate the pathological processes related to neuronal death or neurodegeneration and appear beneficial for improving neuroprotection. As a result of neuronal death or neuroprotection, the brain undergoes activity-dependent long-lasting changes in synaptic transmission, which is also known as functional plasticity. Neuroprotection implying the rescue from neuronal death is now becoming one of global health concerns. This Special Issue attempts to explore the recent advances in neuroprotection related to the brain. This Special Issue welcomes original research or review papers demonstrating the mechanisms of neuroprotection against brain injury using in vivo or in vitro models of animals as well as in clinical settings. The issues in a paper should be supported by sufficient data or evidence. Prof. Bae Hwan Lee Guest EditorNeuroprotectionResearch & information: generalbicsscacid-sensing ion channelsadenoviral vectoradiponectinagingAlzheimer's diseaseamilorideamyloid-betaamyotrophic lateral sclerosisantioxidantantioxidantsapoptosisascorbic acidastrocytesastrocytic networksaxotomyBDNFbean phosphatidylserine (Bean-PS)blood brain barrierbrainbrain targetingbrain-derived neurotrophic factorcalcium activitycatecholaminescation-chloride cotransporterscell deathcholine acetyltransferase (ChAT)connexin 43cortisolCRISPR/cas9depressionelectroneutral transportendotheline-1excitotoxicityGDNFgerbilgliaglioblastomaglobal cerebral ischemiaGPR4 receptorhippocampushomocysteinehuman umbilical cord blood mononuclear cellsinflammationinsulinischemiaischemic strokeKCCslactatelipid emulsionliver growth factorMEK/ERKmetabolic diseasemicrogliamiddle cerebral artery occlusionmiRNAsmoderate hypoxiamotoneuronsMPP+N-acetyl-l-cysteinen/ananoparticle-based drug deliveryNCAMneural injuryneurodegenerationneurodegenerative diseaseneurodegenerative disorderneurogenesisneuronal deathneuroprotectionnimodipinenitriteNKCCsoculomotor systemorganotypic hippocampal slice cultureoxidative stressoxygen-glucose deprivationParkinson's diseasePGC-1αphysical exercisePI3K/AKTpreventive gene therapypsychomotor functionpyridoxine deficiencyreaction timereperfusion injurysodium-hydrogen exchanger-1status epilepticusstrokesubarachnoid haemorrhagesynaptic plasticityTg2576 transgenic micetransient receptor potential melastatin 2trimethyltin (TMT)trophic factorsvascular endothelial growth factorvascular endothelial growth factor receptor 2VEGFWNK-SPAK/OSR1zincResearch & information: generalLee Bae Hwanedt1311353Lee Bae HwanothBOOK9910557716703321Neuroprotection: Rescue from Neuronal Death in the Brain3030280UNINA