02732nam 2200505 450 991013771550332120221008084036.0953-51-6831-2(CKB)3230000000076644(NjHacI)993230000000076644(oapen)https://directory.doabooks.org/handle/20.500.12854/65670(OCoLC)727068009(EXLCZ)99323000000007664420221008d2012 uy 0engur|||||||||||txtrdacontentcrdamediacrrdacarrierPancreatitis treatment and complications /edited by Luis RodrigoIntechOpen2012[Place of publication not identified] :InTech,[2012]©20121 online resource (226 pages) illustrationsNIH publication ;no. 08-1596Title from PDF caption title screen (nih.gov, viewed May 23, 2011)."July 2008"--[8] p.953-51-0109-9 Includes bibliographical references.Pancreatitis may be acute or chronic. Although they can be caused by similar aetiologies, they tend to follow distinct natural histories. Around 80% of acute pancreatitis (AP) diagnoses occur as secondary to gallstone disease and alcohol misuse. This disease is commonly associated with the sudden onset of upper abdominal that is usually severe enough to warrant the patient seeking urgent medical attention. Overall, 10 to 25% of AP episodes are classified as severe, leading to an associated mortality rate of 7 to 30%. Treatment is conservative and consists of general medical support performed by experienced teams, sometimes in ICUs. Although most cases of acute pancreatitis are uncomplicated and resolve spontaneously, the presence of complications has significant prognostic importance. Necrosis, hemorrhage, and infection convey rates of up to 25%, 50%, and 80% mortality, respectively. Other complications such as pseudocyst formation, pseudoaneurysm formation, or venous thrombosis increase morbidity and mortality to a lesser degree. The presence of pancreatic infection must be avoided.Pancreatitis PancreatitisHepatologyPancreatitis.616.37Rodrigo Saez Luisedt1394523Rodrigo LuisNational Digestive Diseases Information Clearinghouse (U.S.)National Institute of Diabetes and Digestive and Kidney Diseases (U.S.)NjHacINjHaclBOOK9910137715503321Pancreatitis3452045UNINA