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1. |
Record Nr. |
UNISA996385225403316 |
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Autore |
Potter Christopher <1591-1646.> |
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Titolo |
VVant of charitie iustly charged, on all such Romanists, as dare (without truth or modesty) affirme, that Protestancie destroyeth salvation [[electronic resource] ] : In answer to a late popish pamphlet intituled Charity mistaken &c. By Christopher Potter D.D. chaplaine to his Maty in ordinarie, and provost of Queenes Colledge in Oxford |
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Pubbl/distr/stampa |
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Oxford, : Printed by the printers to the University, and are to be sold by Iohn Clarke under S. Peters Church in Corne-hill [, London], 1633 |
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Descrizione fisica |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Note generali |
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A reply to: Knott, Edward. Charity mistaken, with the want whereof, Catholickes are unjustly charged. |
The title page may be a cancel. |
Running title reads: Answer to Charity mistaken. |
"Charity mistaken. Chap. 6." (caption title) begins new pagination and register. |
Identified as STC 20135a on UMI microfilm. |
Reproduction of the original in the Folger Shakespeare Library. |
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2. |
Record Nr. |
UNINA9910367564903321 |
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Autore |
Brosh Robert M |
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Titolo |
DNA Replication Stress / Robert M. Brosh |
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Pubbl/distr/stampa |
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MDPI - Multidisciplinary Digital Publishing Institute, 2019 |
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Basel, Switzerland : , : MDPI, , 2019 |
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ISBN |
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Descrizione fisica |
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1 electronic resource (368 p.) |
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Soggetti |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Sommario/riassunto |
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This Special Issue of International Journal of Molecular Sciences (IJMS) is dedicated to the mechanisms mediated at the molecular and cellular levels in response to adverse genomic perturbations and DNA replication stress. The relevant proteins and processes play paramount roles in nucleic acid transactions to maintain genomic stability and cellular homeostasis. A total of 18 articles are presented which encompass a broad range of highly relevant topics in genome biology. These include replication fork dynamics, DNA repair processes, DNA damage signaling and cell cycle control, cancer biology, epigenetics, cellular senescence, neurodegeneration, and aging. As Guest Editor for this IJMS Special Issue, I am very pleased to offer this collection of riveting articles centered on the theme of DNA replication stress. The blend of articles builds upon a theme that DNA damage has profound consequences for genomic stability and cellular homeostasis that affect tissue function, disease, cancer, and aging at multiple levels and through unique mechanisms. I thank the authors for their excellent contributions, which provide new insight into this fascinating and highly relevant area of genome biology. |
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