Horizons in neuroscience research . Volume 1 / / Andres Costa and Eugenio Villalba, editors

• Record Nr.: UNINA9910808293803321
• Pubbl/distr/stampa: Hauppauge, NY, : Nova Science Publishers, c2010
• ISBN: 1-61324-996-9
• Edizione: [1st ed.]
• Descrizione fisica: 1 online resource (452 p.)
• Collana: Horizons in neuroscience research ; ; v. 1
• Altri autori (Persone): CostaAndres; VillalbaEugenio
• Disciplina: 612.8
• Soggetti: Neurosciences; Nervous system
• Lingua di pubblicazione: Inglese
• Formato: Materiale a stampa
• Livello bibliografico: Monografia
• Note generali: Description based upon print version of record.
• Nota di bibliografia: Includes bibliographical references and index.
• Nota di contenuto: ""HORIZONS IN NEUROSCIENCE RESEARCH, VOLUME 1""; ""CONTENTS""; ""PREFACE""; ""NERVOUS, IMMUNE, ENDOCRINE REGULATORY SYSTEMS AND DISEASES ASSOCIATED WITH NERVE GROWTH FACTOR CO-SECRETION""; ""SUMMARY""; ""ABBREVIATIONS""; ""INTRODUCTION""; ""1. NERVE GROWTH FACTOR, ITS RECEPTORS AND THE SIGNALING MECHANISMS""; ""1.1. The Characteristics of Nerve Growth Factor""; ""1.2. Nerve Growth Factor Receptors""; ""1.2.1. Tyrosin Kinase (TrkA) Receptor""; ""1.2.2. P75ntr Receptor""; ""1.3. Nerve Growth Factor Receptor Signaling Mechanisms""; ""1.3.1. Tyrosine Kinase Receptor A Signaling""; ""1.3.2. Cell Signaling Via P75ntr Receptor""""1.3.3. Transactivation Between the Nerve Growth Factor-Bound Tyrosine Kinase A and the G Protein-Coupled Adenosine or Adrenergic Receptor Signalings""; ""1.3.3.1. Transactivation Between the Tyrosine Kinase A and Adrenergic Receptors""; ""1.3.3.2. Transactivation Between the TrkA and Adenosine A2 Receptors""; ""REFERENCES""; ""2. CELLS SECRETING NERVE GROWTH FACTOR AND THEIR TISSUE LOCALIZATION""; ""2.1. Neuronal Tissues""; ""2.2. Nonneuronal Tissues""; ""2.2.1. Bone Marrow""; ""2.2.2. Lung Tissues""; ""2.2.3. Adipose Tissues""; ""2.2.4. Orbital Tissues""""2.2.5 Endocrine Tissues""; ""2.2.5.1. Adrenal Glands""; ""2.2.5.2Pituitary Gland""; ""2.2.5.3.Thyroid Glands""; ""REFERENCES""; ""3. NERVE GROWTH FACTOR IN INFLAMMATORY, ; ALLERGIC AND AUTOIMMUNE PROCESSES""; ""3.1. Nerve Growth Factor Modulates the Inflammatory Responses Via Prostaglandins""; ""3.1.1. Nerve Growth Factor Associates with the Inflammatory Pain""; ""3.2. Nerve Growth Factor in Allergic Diseases""; ""3.2.1. Characterization of the Allergic Responses""; ""3.2.2. The Sensory Neuronal Innervation and Nerve Growth Factor in Allergy""; ""3.2.3. Nerve Growth Factor is Involved in the Allergic Inflammation Associated with Fibrosis""""3.3. Relationship Between Nerve Growth Factor and Apoptosis""; ""3.4. Nerve Growth Factor in the Autoimmune Diseases""; ""3.4.1. Suppressor Regulatory T Cells in the Autoimmune Diseases""; ""3.4.2. Neurogenic Inflammation""; ""3.4.2.1. Nerve Growth Factor Regulatory Role in the Autoimmune Diseases""; ""3.5. Nerve Growth Factor Involvement in the Stress""; ""3.5.1. Stress Induced Endocrine Alterations""; ""3.5.2. The Relationship Between the Stress Induced Endocrine and Cytokine Processes""; ""3.5.3. Nerve Growth Factor During Stress Links to the Endocrine and Immune Networks""""3.5.4. The Network Between the Stress Induced Inflammation and the Actions of the Nerve Growth Factor. The Neurogenic Pain""; ""3.6 Nerve Growth Factor Exerts an Effect Towards the Direction of T Helper 2 Dominance""; ""3.6.1 The Effects of Stress, Glucocorticoids, Norepinephrines and CRH on the Direction Towards Th2 Dominance""; ""3.6.2 The Immunocompetent Cells Express Glucocorticoid and Catecholamine Receptors""; ""3.6.3 The Th1 Apoptosis-Susceptibility and the Th2 Apoptosis-Resistance Mediated by NGF""