1.

Record Nr.

UNINA9910782359603321

Titolo

Angiogenesis in Inflammation: Mechanisms and Clinical Correlates [[electronic resource] /] / edited by Michael Seed, David A. Walsh

Pubbl/distr/stampa

Basel : , : Birkhäuser Basel : , : Imprint : Birkhäuser, , 2008

ISBN

1-281-87657-7

9786611876579

3-7643-7650-3

Edizione

[1st ed. 2008.]

Descrizione fisica

1 online resource (186 p.)

Collana

Progress in Inflammation Research, , 2296-4525

Altri autori (Persone)

SeedMichael P

WalshDavid A (David Andrew)

Disciplina

612.1/3

616.0473

Soggetti

Immunology

Cancer

Internal medicine

Blood-vessels - Diseases

Cytology

Cancer Biology

Internal Medicine

Angiology

Cell Biology

Lingua di pubblicazione

Inglese

Formato

Materiale a stampa

Livello bibliografico

Monografia

Note generali

Description based upon print version of record.

Nota di bibliografia

Includes bibliographical references and index.

Nota di contenuto

Neurogenic angiogenesis and inflammation -- The angiogenic drive in chronic inflammation: Hypoxia and the cytokine milieu -- Dendritic cells and angiogenesis -- The lymphocyte in inflammatory angiogenesis -- The fibroblast and myofibroblast in inflammatory angiogenesis -- Chemokines and cytokines in inflammatory angiogenesis -- Modelling angiogenesis in inflammation -- Angiogenesis in the inflammation of arthritis.

Sommario/riassunto

Angiogenesis is an essential component of inflammation and its resolution. This volume provides up-to-date information on the latest developments in the pathology, mechanisms and therapy of



angiogenesis dependent inflammatory disease. Recent years have seen large advances in angiogenesis research, especially in oncology. Traditionally mechanisms in inflammation angiogenesis were inferred from tumour angiogenesis, however recent research has matured highlighting the similarities and dissimilarities between these processes. This volume relates the lessons learned from tumour biology applied to inflammation. This issue of Angiogenesis in Inflammation: Mechanisms and Clinical Correlates develops current knowledge on the mechanisms at the molecular and cellular levels as they relate to inflammation, including acute and chronic inflammation, neurogenic initiation, and the role of the multiple cellular components that comprise inflammation: granulocytes, macrophages, fibroblasts, dendritic cells and lymphocytes. This is related to inflammatory disease: not only the familiar angiogenesis dependent diseases of rheumatoid arthritis and psoriasis, but also loci such as the lung, gastric ulcers, the eye with uveitis, wound healing and periodontal disease and their therapy, how this knowledge may be used in the discovery of novel therapeutics. The volume brings together experts in each of these fields to link the molecular and cellular processes in angiogenesis to those of inflammation and disease, culminating in a discourse on areas for future therapies.