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1. |
Record Nr. |
UNINA990007464780403321 |
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Titolo |
Industrializzazione senza fratture / a cura di Giorgio Fuà e Carlo Zacchia |
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Pubbl/distr/stampa |
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ISBN |
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Descrizione fisica |
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Collana |
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Disciplina |
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Locazione |
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DTE |
FLFBC |
FGBC |
SES |
FSPBC |
ILFGE |
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Collocazione |
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XV R 516 |
Collez. 272(158) |
XV R 62 |
F/1.4111 FUA/83 |
COLLEZ. 103 (158) |
A-IT 0065 |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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2. |
Record Nr. |
UNINA9910700821103321 |
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Titolo |
An overview of issues in testing intrusion detection systems [[electronic resource] /] / Peter Mell ... [and others] |
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Pubbl/distr/stampa |
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[Gaithersburg, Md.] : , : [U.S. Dept. of Commerce, National Institute of Standards and Technology], , [2003?] |
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Descrizione fisica |
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1 online resource (21 pages) |
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Collana |
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Altri autori (Persone) |
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Soggetti |
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Intrusion detection systems (Computer security) |
Intrusion detection systems (Computer security) - Government policy - United States |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Note generali |
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Title from PDF caption title screen (NIST, viewed Aug. 15, 2011). |
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Nota di bibliografia |
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Includes bibliographical references. |
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3. |
Record Nr. |
UNINA9910557303803321 |
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Autore |
Johansson Anders |
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Titolo |
Periodontitis : From Dysbiotic Microbial Immune Response to Systemic Inflammation |
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Pubbl/distr/stampa |
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Basel, Switzerland, : MDPI - Multidisciplinary Digital Publishing Institute, 2020 |
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Descrizione fisica |
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1 online resource (122 p.) |
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Soggetti |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Sommario/riassunto |
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Periodontitis is an infection-induced inflammatory disease accounting for huge healthcare costs and socio-economic impacts. Bacteria from the indigenous oral flora colonize the interspace between the tooth and the connective tissue, which induces an inflammatory response. If the bacteria proliferate and release virulence factors, they cause an imbalance in the host inflammatory response that induces degenerative processes in the surrounding tissues. This process is often slow, and the disease affects mainly older people, but the process could be rapid and affect young individuals if certain pathogens colonize the area. The two most studied periodontal pathogens, Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans, express virulence factors, including proteases and exotoxins. Periodontal bacteria and their products can be translocated to the peripheral circulation and are therefore linked to the risk pattern of several systemic diseases. However, it is not known if the increased risk for systemic disease associated with periodontitis is an effect of the invading bacteria and/or their released products, the release of components from the local inflammatory response, or a common host susceptibility pattern. The most studied periodontitis-associated systemic diseases are cardiovascular diseases and rheumatoid arthritis. Here, we want to shed light on mechanisms behind the associations of periodontal infections with systemic inflammation. |
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