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Record Nr. |
UNINA9910502621703321 |
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Autore |
Wilcox Claire E. |
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Titolo |
Food addiction, obesity, and disorders of overeating : an evidence-based assessment and clinical guide / / Claire E. Wilcox |
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Pubbl/distr/stampa |
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Cham, Switzerland : , : Springer, , [2021] |
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©2021 |
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ISBN |
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Descrizione fisica |
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1 online resource (224 pages) |
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Disciplina |
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Soggetti |
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Compulsive eating |
Trastorns de la conducta alimentària |
Trastorns de la gana |
Obesitat |
Llibres electrònics |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Nota di bibliografia |
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Includes bibliographical references and index. |
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Nota di contenuto |
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Intro -- Introduction: Obesity, Eating Disorders and Food Addiction: Towards a Synthesis -- References -- Contents -- Contributors -- Part I: Standard Approaches to Clinical Assessment and Treatment of Obesity and Binge Eating Disorder (BED) -- 1: Determinants of Body Weight: Metabolism and the Homeostatic System -- 1.1 Energy Balance -- 1.2 Metabolism -- 1.3 Why Weight Gain Is So Easy and Weight Loss So Hard -- 1.4 Homeostatic Feeding Mechanisms -- 1.4.1 The Hypothalamus -- 1.4.2 Key Neuropeptides -- 1.4.3 Other Factors -- 1.5 Neuropeptides, Obesity, and Disordered Eating -- 1.6 Conclusion -- References -- 2: Obesity -- 2.1 Definition of Obesity -- 2.2 Epidemiology -- 2.3 Assessment -- 2.4 Causes, Contributors, and Risk Factors -- 2.5 Genetics -- 2.6 Management -- 2.6.1 Overview of Treatment -- 2.6.2 Comprehensive Models and Behavioral Weight Loss Therapy -- 2.6.3 Dietary Component -- 2.6.4 Physical Activity Component -- 2.6.5 Behavioral Modification Component -- 2.6.6 Pharmacotherapy -- 2.6.7 Dietary Supplements and Procedures to Avoid -- 2.6.8 Bariatric Surgery -- 2.7 Conclusion -- References -- 3: Binge-Related Eating Disorders (Binge Eating Disorder and Bulimia |
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Nervosa) -- 3.1 Epidemiology -- 3.2 Diagnosis and Assessment of BED -- 3.3 Epidemiology of BN -- 3.4 Diagnosis and Assessment of BN -- 3.5 Etiology and Mechanisms of BED and BN -- 3.6 Treatment of BED: General Considerations -- 3.7 Psychotherapies for BED -- 3.8 Pharmacotherapy for BED -- 3.9 Nutritional Recommendations for BED -- 3.10 Best Practices and Guidelines for BED Treatment -- 3.11 Obesity and BED Treatment -- 3.12 Treatment of BN -- 3.13 Treatments for both BED and BN -- 3.14 Conclusion -- References -- Part II: Can the Food Addiction Concept Improve Treatment? -- 4: Problems with Current Approaches to Treating Disorders of Overeating. |
4.1 Weight Loss with Available Treatments Is Modest -- 4.2 Maintenance of Weight Loss Is Difficult -- 4.3 The Biology Behind the Difficulty of Weight Loss and Maintenance -- 4.4 Eating Disorder (ED) Treatment Success Rates -- 4.5 ED and Obesity Treatments Give Conflicting Messages -- 4.6 Side Effects of Diets -- 4.7 Limitations and Side Effects of Medications -- 4.8 Side Effects of Bariatric Surgery -- 4.9 Difficulty of Exercising When Obese -- 4.10 Conclusion -- References -- 5: The Food Addiction Concept: History, Controversy, Potential Pitfalls, and Promises -- 5.1 History -- 5.2 Is the FA Concept Valid? -- 5.2.1 Can Obesity Be Explained by FA? -- 5.2.2 Is FA Distinct from BED and Bulimia? -- 5.2.3 Do DSM Criteria for SUD Present in Relation to Food in Humans and Do Symptoms Cluster Together? -- 5.2.4 Is It Valid to Claim Certain Foods Are "Addictive," and Might It Be More Accurate to Consider FA a Behavioral Disorder? -- 5.3 Is the FA Model Useful, and Do Benefits Outweigh Harms? -- 5.3.1 Abstinence-Based Food Plans -- 5.3.2 Self-Efficacy -- 5.3.3 Public Health -- 5.3.4 Stigma -- 5.4 Potential Promises for the Future -- 5.5 Increasing Community Acceptance -- 5.6 Conclusion -- References -- Part III: Clinical Evidence for Food Addiction -- 6: Clinical Evidence for the Validity of Food Addiction -- 6.1 Shared DSM Criteria -- 6.2 Yale Food Addiction Scale Development -- 6.3 SUD and Disordered Eating Co-occur -- 6.4 Sweet Preference, Addiction Transfer, and Cross-Sensitization -- 6.5 Overlapping Neuropsychological, Emotional, and Personality Traits, Psychiatric Diagnoses, and Predisposing Conditions (Trauma and Stress) -- 6.6 Conclusion -- References -- Part IV: Basic Biology of Food Addiction, and Its Overlap with Substance Use Disorders -- 7: Neurobiology and Cognitive Neuroscience of Substance Use Disorders. |
7.1 Overview of Substance Use Disorders -- 7.2 Core Brain Regions -- 7.3 Reward -- 7.4 Conditioning: Positive Reinforcement -- 7.5 Motivation: Positive Reinforcement -- 7.6 Tolerance: Downregulation of Dopamine and Opioid System -- 7.7 Withdrawal and Hyperkatifeia -- 7.8 Conditioning and Motivation: Negative Reinforcement -- 7.9 Impulsivity and Executive Function Deficits -- 7.10 Benefits of Understanding the Neurobiology -- 7.11 Conclusion -- References -- 8: Neurobiology and Cognitive Neuroscience of Hedonic Eating -- 8.1 Reward and Hedonic Liking -- 8.2 Conditioning: Positive Reinforcement -- 8.3 Motivation: Positive Reinforcement -- 8.4 Food Reward, Conditioning, and Reward Motivation: Additional Factors -- 8.5 Tolerance and Downregulation of DA and Opioid Systems -- 8.6 Withdrawal -- 8.7 Conditioning and Motivation: Negative Reinforcement -- 8.8 Impulse Control and Executive Function Deficits -- 8.9 Conclusion -- References -- 9: Additional Biological Mechanisms of Hedonic Eating -- 9.1 Interactions Between the Homeostatic System and Hedonic System -- 9.1.1 Anatomy -- 9.1.2 Appetite-Regulating Neuropeptides Modulate Hedonic Eating -- 9.1.3 Appetite-Regulating Neuropeptides Moderate Drug and Alcohol |
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Use -- 9.2 Stress, Hedonic Eating, and the Reward System -- 9.2.1 The Anatomy of the Stress Response -- 9.2.2 Acute and Chronic Stress Promote Hedonic Eating -- 9.3 Genetics -- 9.4 In Utero Exposure -- 9.5 Neuroinflammation -- 9.6 Oxidative Stress -- 9.7 Gut Microbiome and Gut-Brain Axis -- 9.8 Adrenergic System -- 9.9 Sleep and Circadian Rhythm -- 9.10 Serotonin System -- 9.11 Endocannabinoid System -- 9.12 Functional Connectivity -- 9.13 Conclusions -- References -- 10: Treatment-Related Evidence that Food Addiction Is a Valid Construct -- 10.1 Pharmacotherapy-Related Evidence -- 10.1.1 Stimulants. |
10.1.2 Opioid Antagonists -- 10.1.3 Topiramate and Zonisamide -- 10.1.4 GLP-1 Agonists -- 10.1.5 Other Medications to Note -- 10.2 Bariatric Surgery -- 10.3 Conclusion -- References -- 11: Highly Palatable Foods Are Addictive -- 11.1 Problematic and "Addictive" Foods -- 11.2 Association Between HP Food Intake and Addiction in Animal Models -- 11.3 Association Between HP Food Intake and Weight Gain/Disordered Eating in Humans -- 11.4 Why Are HP Foods More Associated with Addictive Eating Patterns? -- 11.4.1 Innate Preferences -- 11.4.2 Conditioning from Rapid Post-oral Glucose Rise -- 11.4.3 Stimulation of Reward System by HP Foods -- 11.4.4 Effects on Inflammatory Processes, Oxidative Stress and Gut Microbiome -- 11.4.5 Cessation Leads to Withdrawal -- 11.4.6 Adverse Effects on Mood and Anxiety -- 11.4.7 Reduction in Executive Function -- 11.4.8 Reduction in Satiety Due to Changes in Homeostatic Feeding -- 11.4.9 Individual Variability -- 11.4.10 Feeding Patterns Influence Food Addiction -- 11.5 State Effects of Hunger/Food Restriction on Reward Circuitry and Brain Function -- 11.6 Artificial Sweeteners and Sugar Substitutes -- 11.7 What Should Be Considered Addictive Food? -- 11.8 Conclusion -- References -- Part V: Assessment and Treatment of Food Addiction -- 12: Evaluation of Food Addiction: Importance, Epidemiology, Diagnosis, and Assessment -- 12.1 Importance of Assessing for Food Addiction -- 12.2 Epidemiology of FA -- 12.3 YFAS: Scoring and Interpretation -- 12.4 Other Important Assessment Considerations and Common Comorbidities -- 12.4.1 SUD -- 12.4.2 ED History -- 12.4.3 Obesity History and Related Health Concerns -- 12.4.4 Psychiatric Comorbidity -- 12.5 Conclusion -- References -- 13: How to Treat Food Addiction from a Nutritional Perspective: Consideration of Diet and Abstinence. |
13.1 Nutritional Approaches and Consideration of Abstinence -- 13.2 Related Tips -- 13.2.1 Increase Satiety and Brain Health-Promoting Foods -- 13.2.2 Do Not Over-restrict Calorie Intake -- 13.2.3 Realize that Craving Will Diminish with Time in Recovery -- 13.2.4 Abstinence Is Not Absolute: Avoid All-or-Nothing Thinking -- 13.2.5 Is It Better to Start More Extreme or Use a Graded Approach During Initiation? -- 13.2.6 Track Progress -- 13.3 How to Incorporate FA Treatment into ED Treatment Programming -- 13.4 What to Do with "Normal Weight" FA Patients? -- 13.5 Conclusion -- References -- 14: Clinical Applications of the Food Addiction Concept -- 14.1 Treatment Overview -- 14.2 Supplemental Programmatic Elements Which Might Be Useful for Treatment of FA -- 14.2.1 Psychoeducation: FA Is a Brain-Based Disorder -- 14.2.2 Psychosocial Interventions -- 14.2.2.1 CBT -- 14.2.2.2 Behavioral Weight Management -- 14.2.2.3 Motivational Interviewing (MI) -- 14.2.2.4 Psychotherapy to Reduce Negative Affect States, Improve Emotion Regulation, and Address Alexithymia -- 14.2.2.5 Mindfulness-Based (MB) Approaches -- 14.2.2.6 Body Image Work -- 14.2.3 Importance of Sleep -- 14.2.4 Importance of Exercise -- 14.2.5 Importance of Getting Psychiatric and Psychological Care -- 14.2.6 Neuromodulation Techniques -- |
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14.2.7 12-Step Programs and Other Support -- 14.2.8 Medications -- 14.2.9 Bariatric Surgery -- 14.3 Subtyping and FA Treatment Matching -- 14.3.1 Within-FA Treatment Matching -- 14.3.2 Using FA as a Treatment Matching Variable for Patients with Obesity and BE -- 14.4 Conclusion -- References -- Part VI: Research Possibilities -- 15: Emerging Treatments and Areas for Future Research -- 15.1 Emerging Treatments for Disordered Eating -- 15.1.1 Neurostimulation -- 15.1.2 Real-Time fMRI (Rt-fMRI) Neurofeedback Training. |
15.1.3 Cognitive Training. |
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