1.

Record Nr.

UNINA9910458653103321

Titolo

The immune synapse as a novel target for therapy [[electronic resource] /] / Luis Graca, editor

Pubbl/distr/stampa

Basel, : Birkhäuser, c2008

ISBN

1-281-14091-0

9786611140915

3-7643-8296-1

Edizione

[1st ed. 2008.]

Descrizione fisica

1 online resource (203 p.)

Collana

PIR (Series)

Altri autori (Persone)

GracaLuis, MD.

Disciplina

616.079

Soggetti

Immune response - Molecular aspects

Immune response - Regulation

Synapses

Electronic books.

Lingua di pubblicazione

Inglese

Formato

Materiale a stampa

Livello bibliografico

Monografia

Note generali

"Progress in inflammation research".

Nota di bibliografia

Includes bibliographical references and index.

Nota di contenuto

The immune synapse and T cell activation: regulation by chemokines -- The induction of regulatory T cells by targeting the immune synapse -- Infiltrating the immunological synapse: prospects for the use of altered peptide ligands for the treatment of immune pathology -- Targeting CD4 for the induction of dominant tolerance -- Anti-CD3: from T cell depletion to tolerance induction -- Immune modulation by CD40L blockade -- CTLA-4-immunoglobulin and indoleamine 2,3-dioxygenase in dominant tolerance -- Adhesion molecules as therapeutic targets -- E3 ubiquitin ligases and immune tolerance: Targeting the immune synapse from within? -- FOXP3 biochemistry will lead to novel drug approaches for vaccines and diseases that lack suppressor T cells -- Transforming growth factor-?: From its effect in T cell activation to a role in dominant tolerance -- From mice to men: the challenges of developing tolerance-inducing biological drugs for the clinic.

Sommario/riassunto

The immune synapse can be compared to a molecular machine that controls T cell activation when getting in contact with an antigen-presenting cell (APC). The immune synapse is involved in the transfer of



information across the T cell–APC junction. It plays an essential role in the control and nature of the immune response. In recent years several approaches have been developed to reprogram the immune response by targeting molecules involved in the immune synapse. Monoclonal antibodies, such as those targeting the lymphocyte co-receptor, costimulatory and adhesion molecules (CD3, CD4, CD40L, CTLA4-Ig, LFA-1), or altered peptide ligands have been shown capable of inducing immune tolerance in transplantation, autoimmunity and allergy. This volume discusses the progress in the field, from basic science to clinical trials, and the major mechanisms involved. It is of interest to clinicians and researchers working in this area.

2.

Record Nr.

UNINA9910846984503321

Autore

Siller Georg

Titolo

Unsicheres Mitleid : Eine Begriffssuche im Ausgang von Wittgenstein / Georg Siller

Pubbl/distr/stampa

Bielefeld, : transcript Verlag, 2018

ISBN

9783839443224

3839443229

Edizione

[1st ed.]

Descrizione fisica

1 online resource (262 pages)

Collana

Edition Moderne Postmoderne

Classificazione

CI 5013

Disciplina

100

Soggetti

Mitleid; Emotion; Respekt; Philosophie der Emotionen; Wittgenstein; Aspektwahrnehmung; Aristoteles; Friedrich Nietzsche; Bertolt Brecht; Hannah Arendt; Sozialität; Ethik; Philosophiegeschichte; Analytische Philosophie; Sozialphilosophie; Philosophie; Compassion; Feeling; Respect; Philosophy of Emotions; Aspect-Perception; Aristotele; Social Relations; Ethics; History of Philosophy; Analytical Philosophy; Social Philosophy; Philosophy

Lingua di pubblicazione

Tedesco

Formato

Materiale a stampa

Livello bibliografico

Monografia

Nota di bibliografia

Includes bibliographical references.

Nota di contenuto

Frontmatter    1 Inhalt    5 Einleitung    9 1. Die Familienähnlichkeit von »Mitleid«    17 2. Varianten der Zuschreibung    83 3. Das Mitleid in der Neuropsychologie    159 4. Irritiertes Mitleid    183 Resümee    239 Danksagung    249 Literatur    251



Sommario/riassunto

Wie kaum ein anderes Gefühl ist Mitleid von Unsicherheiten geprägt: Der Begriff »Mitleid« wird widersprüchlich verwendet - aber auch das Gefühl selbst kann schwanken. Dies wirft Fragen der Angemessenheit auf. Georg Sillers genaue Lektüre Ludwig Wittgensteins zeigt, dass solche Unbestimmtheiten jedoch nicht als Defizite gesehen werden müssen: Erstens sind psychologische Begriffe in ihrer Bedeutungsvielfalt Teil unserer Lebensform und damit mehr als die Bezeichnung von Zuständen, zweitens stellt schwankendes Mitleid eine ganz eigene Haltung dar - und zwar häufig die des Respekts. Diese Perspektive ermöglicht neue Zugriffe auf Aristoteles, Nietzsche, Brecht, Arendt sowie die Neuropsychologie - und nicht zuletzt auf Wittgenstein selbst.