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1. |
Record Nr. |
UNINA9910782009503321 |
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Autore |
Lucas Scott |
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Titolo |
The betrayal of dissent [[electronic resource] ] : beyond Orwell, Hitchens and the new American century / / Scott Lucas |
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Pubbl/distr/stampa |
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London ; ; Sterling, Va., : Pluto, 2004 |
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ISBN |
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1-84964-476-4 |
1-281-75032-8 |
9786611750329 |
1-4356-6202-4 |
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Descrizione fisica |
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1 online resource (336 p.) |
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Disciplina |
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Soggetti |
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Dissenters - United States |
Government, Resistance to - United States |
Social control - United States |
War on Terrorism, 2001-2009 |
Iraq War, 2003-2011 |
United States Politics and government 2001-2009 |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Note generali |
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Bibliographic Level Mode of Issuance: Monograph |
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Nota di bibliografia |
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Includes bibliographical references (p. 233-316) and index. |
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Sommario/riassunto |
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Since his death in 1950, George Orwell has been canonised as England's foremost political writer, and the standard-bearer of honesty and decency for the honourable 'Left'. In this controversial polemic, Scott Lucas argues that the exaltation of Orwell, far from upholding dissent against the State, has sought to quash such opposition. Indeed, Orwell has become the icon of those who, in the pose of the contrarian, try to silence public opposition to US and U K foreign policy in the 'War on Terror'.Lucas's lively and readable critique of public intellectuals including Christopher Hitchens, Michael Walzer, David Aaronovitch, and Johann Hari - who have all invoked Orwellian honesty and decency to shut down dissent - will appeal to anyone disillusioned with the wars in Afghanistan and Iraq. Lucas contends that these leading journalists and commentators have used Orwell to justify their own political transition |
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from radicals to upholders of the establishment. All of them play influential roles in supporting the UK and US governments' charge that opponents of war -- and those who question the motives behind American foreign policy and its implementation -- should be condemned as 'appeasers of mass murder'.This controversial book shows how Orwell has been used since 9/11 to justify, in the guise of independent thought, the suppression of dissent. We must rescue ourselves from Orwell and from those who take on his guise so, as Lucas puts it, our 'silencing is... vital to a "manufacture of consent" for the wars which are supposedly being fought in our name and for our good'. |
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2. |
Record Nr. |
UNINA9910373918403321 |
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Titolo |
Modulation of Oxidative Stress in Heart Disease / / edited by Sajal Chakraborti, Naranjan S. Dhalla, Madhu Dikshit, Nirmal K. Ganguly |
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Pubbl/distr/stampa |
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Singapore : , : Springer Singapore : , : Imprint : Springer, , 2019 |
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ISBN |
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Edizione |
[1st ed. 2019.] |
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Descrizione fisica |
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1 online resource (XIII, 686 p. 114 illus., 100 illus. in color.) |
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Disciplina |
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Soggetti |
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Oxidative stress |
Cardiovascular system |
Cardiology |
Cell physiology |
Biochemistry |
Biomedical engineering |
Oxidative Stress |
Cardiovascular Biology |
Cell Physiology |
Biochemistry, general |
Biomedical Engineering/Biotechnology |
Estrès (Psicologia) |
Malalties del cor |
Llibres electrònics |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Sommario/riassunto |
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This book highlights the multifaceted roles of Reactive Oxygen Species (ROS) in modulating normal cellular and molecular mechanisms during the development of different types of heart disease. Each chapter in the book deals with the role that altered redox homeostasis plays in the pathophysiology of heart disease. In addition, the book explains how reactive oxidant species interact with their targets and provides novel strategies for attenuating oxidative stress-induced types of heart disease. The book not only covers ROS-induced response in heart disease at the cellular level, but also demonstrates that an imbalance of redox states has its roots in our genes, and explains the ways gene expression is regulated. In turn, it reviews potential sources of ROS, their pathological effects on the heart, and potential sites for therapeutic interventions. |
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