1.

Record Nr.

UNINA9910299487803321

Autore

Wong Cheng-Chi

Titolo

Turbo decoder architecture for beyond-4G applications / / Cheng-Chi Wong, Hsie-Chia Chang

Pubbl/distr/stampa

New York : , : Springer, , 2014

ISBN

1-4614-8310-7

Edizione

[1st ed. 2014.]

Descrizione fisica

1 online resource (viii, 100 pages) : illustrations

Collana

Gale eBooks

Disciplina

004.1

620

621.3815

621.382

Soggetti

Wireless communication systems

Signal processing - Digital techniques

Long-Term Evolution (Telecommunications)

Lingua di pubblicazione

Inglese

Formato

Materiale a stampa

Livello bibliografico

Monografia

Note generali

Description based upon print version of record.

Nota di bibliografia

Includes bibliographical references.

Nota di contenuto

Introduction -- Conventional Architecture of Turbo Decoder -- Turbo Decoder with Parallel Processing -- Low-Complexity Solution for Highly Parallel Architecture -- High Efficiency Solution for Highly Parallel Architecture.

Sommario/riassunto

This book describes the most recent techniques for turbo decoder implementation, especially for 4G and beyond 4G applications.  The authors reveal techniques for the design of high-throughput decoders for future telecommunication systems, enabling designers to reduce hardware cost and shorten processing time.  Coverage includes an explanation of VLSI implementation of the turbo decoder, from basic functional units to advanced parallel architecture. Several state-of-the-art techniques that improve complexity and/or throughput are introduced.  The authors discuss both hardware architecture techniques and experimental results, showing the variations in area/throughput/performance with respect to several techniques. This book also illustrates turbo decoders for 3GPP-LTE/LTE-A and IEEE 802.16e/m standards, which provide a low-complexity but high-flexibility circuit structure to support these standards and enables



designs that reconfigure block size and parallelism.  Case studies include the discussions of both throughput and performance of each mode (block size/parallelism/iteration).  This book not only highlights the critical design issues that restrict the speedup of parallel architecture, but it also provides the solutions to overcome these limitations by modifying slightly the turbo codec of modern standards.     ·         Offers readers a complete introduction to practical turbo decoder design; ·         Describes different design methodologies and explains the trade-offs between performance improvement and overhead; ·         Explains modern techniques for state-of-the-art designs; ·         Includes simulation and implementation results with respect to various decoder circuit designs; ·         Reveals novel approaches to higher operating efficiency of turbo decoders for beyond 4G applications.

2.

Record Nr.

UNINA9910557898903321

Autore

Holzmann Klaus

Titolo

Fibroblast Growth Factor Receptor (FGFR) Signaling Pathway in Tumor

Pubbl/distr/stampa

Basel, Switzerland, : MDPI - Multidisciplinary Digital Publishing Institute, 2020

Descrizione fisica

1 online resource (276 p.)

Soggetti

Medicine

Lingua di pubblicazione

Inglese

Formato

Materiale a stampa

Livello bibliografico

Monografia

Sommario/riassunto

Fibroblast growth factor (FGF) signal transmission has an essential function in embryonic development and tissue repair, and is dysregulated in the vast majority of malignancies studied. The FGF signaling in the tumor cells is usually increased by autocrine and paracrine mechanisms and gives them a high growth potential, resistance to apoptosis, neoangiogenesis and metastasis, all essential parameters relevant for tumor progression. This makes FGFs, and their



tyrosine kinase receptors FGFRs, valuable targets for therapeutic interventions. This book is a collection of 15 recent articles-both original work and reviews-that summarize the current research state effectively. The content covers FGF signaling aspects in gastric, skin, liver, esophageal cancer, melanoma, mesothelioma and glioblastoma, including one article that addresses the role of FGF in the tumor-microenvironment cross-talk. Several reports describe the development of compounds targeting FGFRs, their structure and interaction with the receptor molecules, and their effectivity in preclinical and clinical testing. In summary, the papers demonstrate the complexity of the topic, with various FGF ligands and receptors involved and the need for further research. They also present results that fuel hope that targeting cancer with dysfunctional FGF signaling can become a realistic treatment option.