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1. |
Record Nr. |
UNINA9910141043803321 |
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Autore |
Henderson Timothy J |
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Titolo |
Beyond borders : a history of Mexican migration to the United States / / Timothy J. Henderson |
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Pubbl/distr/stampa |
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Chichester, West Sussex ; ; Malden, MA, : Wiley-Blackwell, 2011 |
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ISBN |
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1-4443-9495-9 |
1-282-99020-9 |
9786612990205 |
1-4443-9496-7 |
1-4443-9494-0 |
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Descrizione fisica |
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1 online resource (196 p.) |
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Collana |
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A National Bureau of Economic Research conference report Mexican immigration to the United States |
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Disciplina |
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Soggetti |
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Mexicans - United States - History |
Electronic books. |
Mexico Emigration and immigration History |
Mexico Relations United States |
United States Emigration and immigration History |
United States Relations Mexico |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Note generali |
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Description based upon print version of record. |
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Nota di bibliografia |
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Includes bibliographical references and index. |
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Nota di contenuto |
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Beyond Borders: A History of Mexican Migration to the United States; Contents; List of Figures; Series Editor's Preface; Acknowledgments; Introduction; 1: Beginnings 1848 - 1920; 2: Restriction, Depression, and Deportation; 3: The Bracero Era 1942 - 1964; 4: Illegal Immigration and Response 1964 - 1990; 5: Free Trade and Homeland Security 1990 - Present; Epilogue and Conclusion; Notes; Further Reading; Index |
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Sommario/riassunto |
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Beyond Borders: A History of Mexican Migration to the United States details the origins and evolution of the movement of people from Mexico into the United States from the first significant flow across the border at the turn of the twentieth century up to the present day. Considers the issues from the perspectives of both the United States and MexicoOffers a reasoned assessment of the factors that drive Mexican immigration, explains why so many of the policies enacted in |
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Washington have only worsened the problem, and suggests what policy options might prove more effective<li |
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2. |
Record Nr. |
UNINA9910261135903321 |
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Autore |
Anna M. Lavezzi |
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Titolo |
New Approaches to the Pathogenesis of Sudden Intrauterine Unexplained Death and Sudden Infant Death Syndrome |
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Pubbl/distr/stampa |
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Descrizione fisica |
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1 electronic resource (104 p.) |
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Collana |
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Frontiers Research Topics |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Sommario/riassunto |
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Sudden Infant Death Syndrome (SIDS) is the leading cause of death among infants in the first year of age. The more known definition of SIDS is the sudden unexpected death of an infant less than 1 year of age, with onset of the fatal episode apparently occurring during sleep, that remains unexplained after a thorough investigation, including performance of a complete autopsy and review of the circumstances of death and the clinical history. Despite the success of the “Back to Sleep” campaigns to reduce the risks introduced worldwide, the frequency of SIDS (striking one infant every 750-1,000 live births) has not significantly declined in the last years. Sudden Intrauterine Unexplained Death Syndrome (SIUDS), referring to fetuses that die unexpectedly, particularly in the last weeks of gestation, without any cause even after a complete autopsy, including examination of the placental disk, umbilical cord and fetal membranes, has a six-eightfold greater incidence than that of SIDS. Even if the pathogenetic mechanism of these deaths has not yet been determined, the neuropathology seems to be a consistent substrate in both SIUDS and SIDS. Subtle common developmental abnormalities of brainstem nuclei checking the vital functions have been highlighted, frequently related to environmental |
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risk factors, such as cigarette smoke, air and water pollution, pesticides, food contamination, etc. Exogenous toxic factors can in fact interact in complex ways with the genetic constitution of the infant leading to polymorphisms and/or mutations of specific genes (as polymorphisms of the serotonin transporter gene 5-HTT, the regulator of the synaptic serotonin concentration, and of the PHOX2B, the key gene in the Congenital Central Hypoventilation Syndrome). These interactions can directly injure the development of the autonomic nervous system, frequently resulting in hypoplasia of the vital brainstem centers, and consequently in sudden death. It is very important to continue studying these syndromes and in particular identify all possible congenital alterations and their correlation with the exposure to environmental risk factors, in order to reduce their incidence and mitigate the surrounding social concern. The goal of this research topic is to propose new approaches to explain the pathogenesis of both SIUDS and SIDS and consequently new prevention strategies to decrease the incidence of these unexpected and very devastating events for families. Expert authors in the Topic field are encouraged to submit original research articles aimed to widen the current knowledge on the pathological substrates of these deaths, also considering the correlations with possible risk factors. Submissions of hypotheses, opinions and commentaries are also welcome. This Research Topic would lead to development of targeted risk-lowering strategies to reduce the incidence of both SIUDS and SIDS. Furthermore, the adoption of appropriate preventive measures could also lead to improve the quality of life in adults, promoting active and healthy aging. |
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