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Record Nr. |
UNINA9910130829703321 |
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Titolo |
2012 IEEE 18th International Conference on Embedded and Real-Time Computing Systems and Applications |
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Pubbl/distr/stampa |
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[Place of publication not identified], : IEEE, 2012 |
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ISBN |
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Descrizione fisica |
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1 online resource : illustrations |
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Disciplina |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Note generali |
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Bibliographic Level Mode of Issuance: Monograph |
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Sommario/riassunto |
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This paper describes a low-overhead and prolonged Linux process logging mechanism that is separated into networks for embedded systems with small memory. It is useful to log the behavior of Linux processes in Android in order to debug, test, and learn about the embedded systems. However, the logging system interferes with process scheduling and requires huge storage to capture the process behavior. We propose a separated Linux process logging mechanism to solve these problems. Two features are utilized to develop the mechanism.The first is an improved logging performance. When acquiring context switch log data from an operating system, some data may be lost because of limited memory. To solve this problem, we propose a low-overhead data generating, low-latency data gathering and sending mechanism. This has a lightweight binary and string data format that uses a context-dependent log compression algorithm.The second feature is the construction of a log data management system that is available over a network. The logging mechanism is separated into two parts - a Logging Environment (LE) and a Logging Monitoring Environment (LME) - to reduce the effect of the target device and increase the volume of log data stored area. The LE consists of a log generation part in the target embedded device and a log collection part on the server. The LME consists of various monitoring and debugging tools, such as a log analyzer or log visualizer. In order to utilize the tools on the LME, the generated log data is exported to the log |
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collection server via the network by an embedded device. The collected log data can be converted to an application-friendly format, such as Common Trace Format (CTF) or JavaScript Object Notation (JSON), on the log collection server to reduce the overhead of the embedded device in the LE and be easily available to the LME tools. We utilize an HTTP-based communication protocol between the LE and LME.As a result of an evaluation, the latency of data gathering and the overhead of data generation are decreased by 50%. The scheduling interference and the volume of log data also decrease. Our method is also comfortable transferring data across the network as well as CPU loading. |
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2. |
Record Nr. |
UNINA9910959970903321 |
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Titolo |
Osteoclasts : morphology, functions and clinical implications / / Alexander J. Brown and Johanna S. Walker, editors |
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Pubbl/distr/stampa |
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New York, : Nova Science Publishers, Inc., 2012 |
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ISBN |
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Edizione |
[1st ed.] |
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Descrizione fisica |
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1 online resource (182 p.) |
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Collana |
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Cell biology research progress |
Human anatomy and physiology |
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Altri autori (Persone) |
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BrownAlexander J |
WalkerJohanna S |
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Disciplina |
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Soggetti |
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Lingua di pubblicazione |
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Formato |
Materiale a stampa |
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Livello bibliografico |
Monografia |
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Note generali |
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Description based upon print version of record. |
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Nota di bibliografia |
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Includes bibliographical references and index. |
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Nota di contenuto |
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Intro -- OSTEOCLASTS -- OSTEOCLASTS -- Contents -- Preface -- Modulation of Osteoclast Differentiation and Function in Rheumatoid Arthritis -- Introduction -- Osteoclasts in Rheumatoid Arthritis -- Growth Factors -- Macrophage Colony Stimulating Factor -- Granulocyte Macrophage- Colony Stimulating Factor -- Transforming Growth Factor-Beta -- Tumor Necrosis Factor Super Family -- Receptor Activator of NF-Kappa B Ligand -- Tumor Necrosis Factor-Alpha -- Osteoprotegerin -- Interferon Family -- Interleukin Family -- Interleukin 1 -- Interleukin 6 -- Interleukin 10 -- Conclusion -- |
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Refrences -- Osteoclast Formation and Function and Its role in Skeletal Bone Disease -- Abstract -- Introduction -- Multinucleation -- Sealing Zone -- Ruffled Border -- Basolateral Membrane -- OCs and Clinical Implications -- Osteopetrosis -- Pycnodysostosis -- Paget's Disease -- Osteoporosis -- Cancer -- Inflammatory Arthritis -- Conclusion -- Acknowledgments -- References -- The High Osteoclastogenic Potential of Human Osteosarcoma Cells: Reciprocal Interaction between MG63 Osteoblast-Like Cells and Osteoclast Precursors -- Abstract -- 1. Introduction -- 2. Cell Culture Models -- 2.1. MG63 Cell Line -- 2.2. Peripheral Blood Mononuclear Cells (PBMC) -- 3. Osteoclastogenic Potential of the MG63 -- Cell Line -- 3.1. Paracrine-Mediated Mechanisms -- 3.2. Cell-to-Cell Contact Mechanisms -- 4. Modulation of the MG63 Cell Behavior by Osteoclastic Cells -- Conclusion -- References -- Osteoclast Biology in Paget's Disease of Bone -- Abstract -- Osteoclast Phenotype in PDB -- Viral and Genetic Factors in Paget's Disease of Bone -- Role of p62 in Osteoclast Signaling -- p62 as a Shuttling Factor -- Role of p62 Mutations in Paget's Disease of Bone -- Conclusion -- References -- Osteoclasts: The Major Actors in Bone Resorption -- Abstract -- Introduction. |
Osteoclast Differentiation and Activation -- Osteoclasts and Immune System Talk -- Osteoclasts in Bone Metastases -- Osteoclasts in Rheumatoid Arthritis -- Osteoclasts in Periprosthetic Osteolysis -- Osteoclasts in Pedriatic Disease -- Osteoclasts in Osteoporosis -- Conclusion -- References -- Role of the Immuno-Skeletal Interface in Physiological and Pathological Osteoclast Regulation -- Abstract -- Introduction -- 1. Physiological Osteoclast Formation and the Role of the RANK/RANKL/OPG Axis -- 2. Role of the Immuno-skeletal Interface in Physiological Osteoclast Renewal -- a) T Cells and B Cells, the Centerpiece of Cell-mediated Immunity -- b) T Cells and B Cells, Critical Regulators of Basal Osteoclastogenesis -- c) Disruption of the Immuno-skeletal Interface and Bone Loss Associated with HIV-Infection -- d) The immuno-skeletal Interface and Osteoclastic Bone Loss Associated with inflammatory Disease -- e) Role of the Immuno-skeletal interface in Estrogen Deficiency-induced Bone Loss -- Conclusion -- Acknowledgments -- References -- Multiple Functions of Osteoclasts and Potential Usefulness of Phosphatidylserine-Containing Liposomes on Bone Diseases -- Abstract -- Introduction -- Immune Fuctions of OCs -- Angiogenic Functions of OCs -- HSC Regulation Function of OCs -- Effects of PSLs on OCs -- Effects of PSLs on Macrophages/Miroglia -- Effects of PSLs on DCs -- Conclusion -- References -- Bone Formation and Osteoclastic Resorption after Implantation of beta-Tricalcium Phosphate (beta-TCP) -- Abstract -- Introduction -- Materials and Methods -- Experiment 1 -- Experiment 2 -- Results -- Experiment 1 -- Experiment 2 -- Discussion -- Conclusion -- References -- Osteoclasts of Patients with Neurofibromatosis 1 (NF1) -- Abstract -- Short Communication -- References -- Index. |
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Sommario/riassunto |
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In this book, the authors present current research in the study of the morphology, functions and clinical implications of osteoclasts. Topics include osteoclast formation and function and its risk in skeletal bone disease. |
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